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Trying to lose weight? Here’s why your genetics could be just as important as your exercise regime

<div class="theconversation-article-body"> <p><em><a href="https://theconversation.com/profiles/henry-chung-1279176">Henry Chung</a>, <a href="https://theconversation.com/institutions/university-of-essex-1291">University of Essex</a>; <a href="https://theconversation.com/profiles/chris-mcmanus-2226445">Chris McManus</a>, <a href="https://theconversation.com/institutions/university-of-essex-1291">University of Essex</a>, and <a href="https://theconversation.com/profiles/sally-waterworth-2226444">Sally Waterworth</a>, <a href="https://theconversation.com/institutions/university-of-essex-1291">University of Essex</a></em></p> <p>Weight loss is a complicated process. There are so many factors involved including your diet, how much sleep you get each night and the kind of exercise you do. Our recent study shows that your <a href="https://www.tandfonline.com/doi/full/10.1080/02701367.2024.2404981">specific genetic profile</a> may also have a dominant effect on how well you lose weight through exercise. This might explain why two people who do an identical workout will see very different results.</p> <p>We identified 14 genes that appeared to significantly contribute to how much weight a person lost through running. This suggests that some of us have a natural talent when it comes to burning fat and losing weight through exercise.</p> <p>To conduct our study, we recruited 38 men and women born in the UK aged between 20 and 40. None of the participants regularly exercised at the start of the study. The group was randomly divided, with one half following a strict eight-week endurance programme that consisted of three weekly runs of 20-30 minutes.</p> <p>The other group acted as a <a href="https://www.britannica.com/science/control-group">control</a>. They were instructed to refrain from exercise and continue their daily routines as normal over this study period, including diet and lifestyle habits.</p> <p>All participants conducted a running test to see how far they could run in 12 minutes, and were weighed before and after the study period. This was to gauge their initial fitness level and see how much they changed over the duration of the study. <a href="https://www.nhs.uk/conditions/obesity/">Body mass index</a> (BMI) was also calculated.</p> <p>Additionally, a saliva sample was collected from each person with a <a href="https://muhdo.com/?gclid=Cj0KCQjwiIOmBhDjARIsAP6YhSUB3WI81JP4Q_snYLhh-SBVNeCJNy2m63C8bKJFvO-nJ5UsHuCCdqMaAhTeEALw_wcB">DNA test kit</a> at the end of the study to assess their unique genetic profile.</p> <p>It’s important to note that everyone who participated in the study had a similar body weight, BMI and aerobic fitness level at the start of the study. This is beneficial for <a href="https://casp-uk.net/news/homogeneity-in-research/">multiple reasons</a>. It meant everyone was at the same starting point, and some <a href="https://www.sciencedirect.com/topics/nursing-and-health-professions/confounding-variable">confounding variables</a> were already controlled for such as <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10128125/">fitness level</a>. This ultimately improves accuracy in interpreting the results.</p> <h2>Exercise genes</h2> <p>Everyone in the exercise group managed to lose weight – around 2kg on average. The control group, on the other hand, put on a little bit of weight.</p> <p>While a 2kg weight loss may not sound like a lot, it’s significant considering the exercise regime only lasted eight weeks and participants made no <a href="https://www.intechopen.com/chapters/87186">changes to their diet</a>.</p> <p>More significant, however, was the large variation in results among those that exercised – with an up to 10kg difference in weight loss between some of the participants. In fact, everyone within the exercise group improved at different rates.</p> <p>Since we controlled for factors such as the <a href="https://pubmed.ncbi.nlm.nih.gov/3529283/">intensity, duration and frequency</a> of the exercises and used participants who’d had a similar body weight and fitness level at the start of the study, this suggests that some people naturally benefited more than others from endurance training.</p> <p>When we looked at the genetic profiles of our participants, we found that differences in each person’s response to the exercise was strongly associated with their specific genetics.</p> <p>We showed there was a strong linear correlation between the amount of weight participants lost and 14 genes that have previously been shown to be associated with body weight, metabolism or <a href="https://www.nature.com/articles/s41380-018-0017-5">psychological conditions</a> that affect BMI. The greater number of these genes a participant had, the more weight they lost. Our results also revealed that around 63% of the variance in weight lost among participants were explained by the genes identified.</p> <p>For example, research has shown the <a href="https://www.ncbi.nlm.nih.gov/gene/10891">PPARGC1A gene</a> plays a role in metabolism and the <a href="https://link.springer.com/article/10.1007/S11033-020-05801-Z">use of fats for energy</a> while exercising. Our study found that all participants who lost more than 1.5kg from exercise had this gene. Those who lost less than this did not have this gene.</p> <p>Our findings align with what <a href="https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0249501&amp;s2=P875440273_1683331208925004155">previous studies</a> have shown. But while previous papers have only looked at the link between individual genes and weight loss, ours is the first to show that 14 different genes appear to work in combination to affect whether a person loses weight from endurance exercise.</p> <h2>Piece of the puzzle</h2> <p>Our study also suggests that while some people possess genes that make it easier for them to get fit and lose weight, people with these favourable genetics can only flourish if they actually exercise. In fact, our control group also had a number of these listed genes, but without exercise these genes could not activate, and so the participants did not lose any weight.</p> <p>While our study provides compelling findings, it’s not without limitations. Since we only looked at endurance-based exercise, it will be important for future studies to investigate whether there are similar links between weight loss, genetics and combinations of different types of training (such as a mixture of endurance and strength sessions into a training plan).</p> <p>It’s also worth mentioning that exercise is only <a href="https://www.who.int/activities/controlling-the-global-obesity-epidemic">one piece of the puzzle</a> when it comes to weight loss. So even if you have all 14 of these genes, you won’t lose any weight or get fit if you don’t exercise and maintain a healthy diet and sleep pattern.</p> <p>On the flip side, someone that only has a few of these favourable genes can still benefit if they exercise and are mindful of other aspects of their lifestyle.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/240506/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em><a href="https://theconversation.com/profiles/henry-chung-1279176">Henry Chung</a>, Lecturer in Sport and Exercise Science, <a href="https://theconversation.com/institutions/university-of-essex-1291">University of Essex</a>; <a href="https://theconversation.com/profiles/chris-mcmanus-2226445">Chris McManus</a>, Lecturer, School of Sport, Rehabilitation and Exercise Sciences, <a href="https://theconversation.com/institutions/university-of-essex-1291">University of Essex</a>, and <a href="https://theconversation.com/profiles/sally-waterworth-2226444">Sally Waterworth</a>, Lecturer, School of Sport, Rehabilitation and Exercise Sciences, <a href="https://theconversation.com/institutions/university-of-essex-1291">University of Essex</a></em></p> <p><em>Image credits: Shutterstock </em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/trying-to-lose-weight-heres-why-your-genetics-could-be-just-as-important-as-your-exercise-regime-240506">original article</a>.</em></p> </div>

Body

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Breaking the silence on prostate cancer: man’s family legacy highlights importance of early detection

<p>James Murray, 55, an architect from Melbourne, always knew he was living with a genetic "time bomb." A fifth-generation prostate cancer sufferer, his family’s battle with the disease spans over 170 years. From his great-great-grandfather, who chronicled his symptoms in 1847, to his uncles and father who also faced the disease, prostate cancer has been a grim constant in his family.</p> <p>“My family has what's been called a ‘spectacular history’ with prostate cancer,” James reflects. “But that’s not the kind of thing you want to hear. My great-great-grandfather had it, though they couldn’t test for it back then. My grandfather died from it, my father got it, and both of his brothers had it as well. It’s been a constant in our family for generations.”</p> <p>James' great-great-grandfather, Robert William Felton Lathrop Murray, a soldier and the founder of the <em>Hobart Town Gazette</em>, documented his battle with the disease long before modern medicine could provide a diagnosis. Since then, generation after generation of Murrays has grappled with the same fate. </p> <p>For James, however, early detection was key. Diagnosed in February 2022, he underwent surgery just a few months later in May, removing what doctors described as an aggressive form of the disease. But his story, unlike his ancestors, had a different ending.</p> <p>“In a way, we’ve been cursed by prostate cancer," explains James. "But it’s also been a blessing because it made us all hyper-aware of the importance of early detection. My dad was incredibly diligent, and that saved his life. He made sure I was on top of my PSA testing from my mid-40s, and that’s how we caught it early for me.” </p> <p>Prostate-Specific Antigen (PSA) testing and early detection are now recognised as crucial, particularly for men with a family history of prostate cancer. Yet, dangerous myths surrounding prostate cancer testing continue to cost lives, warns Associate Professor Weranja Ranasinghe, Deputy Leader of the Urological Society of Australia and New Zealand’s (USANZ) GU-Oncology Special Advisory Group.</p> <p>“Many men believe that they need to have urinary symptoms to be concerned about prostate cancer,” explains Ranasinghe. “The reality is that most prostate cancers develop without any symptoms at all. Urinary symptoms are not reliable – most of these symptoms are caused by non-cancerous enlargement of the prostate but advanced cancer can also give you urinary symptoms. So it is important to get checked.”</p> <p>An even greater barrier, however, is the outdated belief that prostate cancer screening requires an invasive rectal exam. Ranasinghe stresses that this is no longer the case: “A simple PSA blood test, combined with advanced imaging like MRI, is now the standard approach. Many men are avoiding tests due to this outdated fear, but in the GP setting, a rectal exam is currently not required for detecting prostate cancer."</p> <p>With over 26,000 Australian men expected to be diagnosed with prostate cancer this year, the Urological Society of Australia and New Zealand (USANZ) is raising awareness during Prostate Cancer Awareness Month about the importance of early testing and debunking these misconceptions.</p> <p>Ranasinghe also points out that men with a strong family history should be particularly vigilant. “Men with female relatives who have had breast or ovarian cancer are also at an increased risk of prostate cancer due to the same genetic mutations. So we recommend that men with a strong family history get a PSA test at the age of 40, which is earlier than the recommended age.”</p> <p>For James Murray, the decision to undergo surgery quickly after his diagnosis proved life-saving. “When my PSA levels started rising in February 2022, I wasn’t shocked," he recalls. "I had always known this day would come. It wasn’t something I feared, but more something I was prepared for. I told myself, ‘Okay, it’s here, let’s deal with it,’ and I just focused on getting through the surgery as quickly as possible.”</p> <p>James feels fortunate compared to his uncles, whose diagnoses came much later in life, leading to more severe outcomes. His surgery went well, and he's had a quick recovery, returning to work and feeling grateful that they caught it early. That's why James is advocating for early PSA testing, particularly for men with a family history of prostate cancer. He plans to ensure his 19-year-old son is aware of the need for testing in the future.</p> <p>"We often see men wait until a friend or relative is diagnosed before thinking about getting a prostate cancer PSA test done," says Ranasinghe. "This reactive approach is dangerous. Men need to be proactive and take ownership of their prostate health and talk to their GPs about this, just like they would with cholesterol or other routine health checks."</p> <p>As prostate cancer awareness grows, James hopes his story will encourage others to get tested. It’s something no one wants to face, but the sooner you catch it, the better chance you have.</p> <p>For more information on prostate cancer screening and to find a urologist, visit the Urological Society of Australia and New Zealand (USANZ) at <a href="https://www.usanz.org.au/" target="_blank" rel="noopener">www.usanz.org.au</a>.</p> <p><em>Images: Supplied</em></p>

Caring

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Offering end of life support as part of home care is important – but may face some challenges

<div class="theconversation-article-body"> <p><em><a href="https://theconversation.com/profiles/jennifer-tieman-378102">Jennifer Tieman</a>, <a href="https://theconversation.com/institutions/flinders-university-972">Flinders University</a></em></p> <p>Earlier this month, the government announced <a href="https://theconversation.com/the-government-has-a-new-plan-for-residential-aged-care-heres-whats-changing-238765">major changes</a> to aged care in Australia, including a A$4.3 billion <a href="https://theconversation.com/what-the-governments-home-care-changes-mean-for-ageing-australians-238890">investment in home care</a>.</p> <p>Alongside a shake up of home care packages, the Support at Home program will include an important addition – an <a href="https://www.health.gov.au/sites/default/files/2024-09/support-at-home-fact-sheet.pdf">end of life pathway</a> for older Australians.</p> <p>This pathway will allow access to a <a href="https://www.health.gov.au/our-work/support-at-home/features">higher level</a> of in-home aged care services to help Australians stay at home as they come to the end of their life. Specifically, it will provide an extra A$25,000 for palliative support when a person has three months or less left to live.</p> <p>This is a positive change. But there may be some challenges to implementing it.</p> <h2>Why is this important?</h2> <p>Older people have made clear their preference to remain in their homes as they age. For <a href="https://journals.sagepub.com/doi/10.1177/0269216313487940">most people</a>, home is where they would like to be during their last months of life. The space is personal, familiar and comforting.</p> <p>However, data from the <a href="https://www.abs.gov.au/statistics/research/classifying-place-death-australian-mortality-statistics">Australian Bureau of Statistics</a> shows most people who die between the ages of 65 and 84 die in hospital, while most people aged 85 and older die in residential aged care.</p> <p>This apparent gap may reflect a lack of appropriate services. Both palliative care services and GPs have an important role in providing medical care to people living at home with a terminal illness. However, being able to <a href="https://grattan.edu.au/wp-content/uploads/2014/09/815-dying-well.pdf">die at home</a> relies on the availability of ongoing support including hands-on care and assistance with daily living.</p> <p>Family members and friends often provide this support, but this is not always possible. Even when it is, carers may <a href="https://pubmed.ncbi.nlm.nih.gov/38533612/">lack confidence and skills</a> to provide the necessary care, and may not have enough support for and respite from their carer role.</p> <p>The palliative care funding offered within Support at Home should help an older person to remain at home and die at home, if that is their preference.</p> <p>Unless someone dies suddenly, care needs are likely to increase at the end of a person’s life. Supports at home may involve help with showering and toileting, assessing and addressing symptoms, developing care plans, managing medications, wound dressing, domestic tasks, preparing meals, and communicating with the person’s family.</p> <p>Occupational therapists and physiotherapists can assist with equipment requirements and suggest home modifications.</p> <p>End of life supports may also involve clarifying goals of care, contacting services such as pharmacists for medications or equipment, liaising with organisations about financial matters, respite care or funeral planning, as well as acknowledging grief and offering spiritual care.</p> <p>But we don’t know yet exactly what services the $25,000 will go towards.</p> <h2>What do we know about the scheme so far?</h2> <p>The Support at Home program, including the end of life pathway, is scheduled to start from <a href="https://www.health.gov.au/sites/default/files/2024-09/support-at-home-fact-sheet.pdf">July 1 2025</a>.</p> <p>We know the funding is linked to a prognosis of three months or less to live, which will be determined <a href="https://www.abc.net.au/news/2024-09-15/new-payment-aims-to-make-it-easier-for-people-to-die-at-home/104347984">by a doctor</a>.</p> <p>Further information has indicated that an older person can be referred to a <a href="https://www.health.gov.au/our-work/support-at-home/features">high-priority assessment</a> to access the end of life pathway. We don’t know yet what this means, however they don’t need to be an existing Support at Home participant to be eligible.</p> <p>The pathway will allow 16 weeks to use the funds, possibly to provide some leeway around the three-month timeline.</p> <p>Although more details are coming to light, there are still some things which remain unclear.</p> <p>Home care providers will be looking for details on what can be covered by this funding and how they will work alongside primary care providers and health-care services.</p> <p>Older people and their families will want to know the processes to apply for this funding and how long applications will take to be reviewed.</p> <p>Everyone will want to know what happens if the person doesn’t die within three months.</p> <h2>Some challenges</h2> <p>Ready availability of appropriate supports and services will be crucial for older people accessing this pathway. Home care providers will therefore need to assess how an end of life pathway fits into their operational activities and how they can build the necessary skills and capacity.</p> <p>Demand for nurses with palliative care skills and allied health professionals is likely to increase. Providing end of life care can be <a href="https://pubmed.ncbi.nlm.nih.gov/33096682/">especially taxing</a> so strategies will be needed to prevent staff burnout and encourage self-care.</p> <p>How pathways are implemented in rural and remote areas and in different cultural and community groups will need to be monitored to ensure all older people benefit.</p> <p>Effective coordination and communication between home care, primary care and specialist palliative providers care will be key. Digital health systems that connect the sectors could be helpful. Family engagement will also be very important.</p> <p>Escalation pathways and referral pathways should be established to enable appropriate responses to emergencies, unexpected deterioration, and family distress.</p> <p>Finally, <a href="https://bmcpalliatcare.biomedcentral.com/articles/10.1186/s12904-023-01155-y">accurately determining</a> when someone will die can be difficult. Knowing when the last three months of life starts may not be easy, particularly where frailty, cognitive issues and multiple health concerns may be present.</p> <p>This might mean some people are not seen as being ready for this pathway. Others may not be willing to accept this prognosis. An older person may also be expected to live with a terminal illness for many months or years. Their palliative care needs would not be met under this pathway.</p> <p>Despite these challenges, the announcement of an end of life pathway within the home care program is timely and welcome. As a population we are living longer and dying older. More details will help us be better prepared to implement this scheme.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/239296/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em><a href="https://theconversation.com/profiles/jennifer-tieman-378102">Jennifer Tieman</a>, Matthew Flinders Professor and Director of the Research Centre for Palliative Care, Death and Dying, <a href="https://theconversation.com/institutions/flinders-university-972">Flinders University</a></em></p> <p><em>Image credits: Shutterstock </em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/offering-end-of-life-support-as-part-of-home-care-is-important-but-may-face-some-challenges-239296">original article</a>.</em></p> </div>

Retirement Life

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What causes food cravings? And what can we do about them?

<div class="theconversation-article-body"> <p><em><a href="https://theconversation.com/profiles/gabrielle-weidemann-91497">Gabrielle Weidemann</a>, <a href="https://theconversation.com/institutions/western-sydney-university-1092">Western Sydney University</a> and <a href="https://theconversation.com/profiles/justin-mahlberg-1634725">Justin Mahlberg</a>, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a></em></p> <p>Many of us try to eat more fruits and vegetables and less ultra-processed food. But why is sticking to your goals so hard?</p> <p>High-fat, sugar-rich and salty foods are simply so enjoyable to eat. And it’s not just you – we’ve evolved that way. These foods <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5928395/">activate</a> the brain’s reward system because in the past they were <a href="https://doi.org/10.1037/a0030684">rare</a>.</p> <p>Now, they’re all around us. In wealthy modern societies we are bombarded by <a href="https://theconversation.com/junk-food-is-promoted-online-to-appeal-to-kids-and-target-young-men-our-study-shows-234285">advertising</a> which intentionally reminds us about the sight, smell and taste of calorie-dense foods. And in response to these powerful cues, our brains respond just as they’re designed to, triggering <a href="https://doi.org/10.1111/obr.12354">an intense urge</a> to eat them.</p> <p>Here’s how food cravings work and what you can do if you find yourself hunting for sweet or salty foods.</p> <h2>What causes cravings?</h2> <p>A food craving is an intense desire or urge to eat something, <a href="https://pubmed.ncbi.nlm.nih.gov/15589112/">often focused on a particular food</a>.</p> <p>We are programmed to learn how good a food tastes and smells and where we can find it again, especially if it’s high in fat, sugar or salt.</p> <p>Something that <a href="https://doi.org/10.1111/obr.12354">reminds us</a> of enjoying a certain food, such as an eye-catching ad or delicious smell, can cause us to <a href="https://doi.org/10.1111/obr.12354">crave it</a>.</p> <p>The cue triggers a physical response, increasing saliva production and gastric activity. These responses are relatively automatic and difficult to control.</p> <h2>What else influences our choices?</h2> <p>While the effect of cues on our physical response is relatively automatic, what we do next is influenced by <a href="https://journals.sagepub.com/doi/pdf/10.1177/1090198107303308">complex</a> factors.<br />Whether or not you eat the food might depend on things like cost, whether it’s easily available, and if eating it would align with your health goals.</p> <p>But it’s usually hard to keep healthy eating in mind. This is because we tend to prioritise a more immediate reward, like the <a href="https://doi.org/10.1016/j.physbeh.2010.04.029">pleasure of eating</a>, over one that’s delayed or abstract – including health goals that will make us feel good in the long term.</p> <p><a href="https://doi.org/10.1016/S0022-3999(00)00076-3">Stress</a> can also make us eat more. When hungry, we <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8656885/">choose larger portions</a>, underestimate calories and find eating more <a href="https://www.sciencedirect.com/science/article/pii/S0195666315000793">rewarding</a>.</p> <h2>Looking for something salty or sweet</h2> <p>So what if a cue prompts us to look for a certain food, but it’s not available?</p> <p><a href="https://doi.org/10.1016/j.appet.2014.04.005">Previous research</a> suggested you would then look for anything that makes you feel good. So if you saw someone eating a doughnut but there were none around, you might eat chips or even drink alcohol.</p> <p>But our <a href="https://doi.org/10.1016/j.appet.2024.107640">new research</a> has confirmed something you probably knew: it’s more specific than that.</p> <p>If an ad for chips makes you look for food, it’s likely a slice of cake won’t cut it – you’ll be looking for something salty. Cues in our environment don’t just make us crave food generally, they prompt us to look for certain food “categories”, such as salty, sweet or creamy.</p> <h2>Food cues and mindless eating</h2> <p>Your <a href="https://core.ac.uk/download/pdf/161283824.pdf">eating history</a> and <a href="https://doi.org/10.1002/eat.24179">genetics</a> can also make it harder to suppress food cravings. But don’t beat yourself up – relying on willpower alone is <a href="https://doi.org/10.1016/j.appet.2015.01.004">hard</a> for almost everyone.</p> <p>Food cues are so powerful they can prompt us to <a href="https://doi.org/10.1177/0956797613484043">seek</a> out a certain food, even if we’re not overcome by a particularly <a href="https://doi.org/10.1177/0956797613484043">strong urge</a> to eat it. The effect is more intense if the food is easily available.</p> <p>This helps explain why we can eat an entire large bag of chips that’s in front of us, even though our pleasure decreases as we <a href="https://doi.org/10.1016/0031-9384(81)90310-3">eat</a>. Sometimes we use finishing the packet as the signal to stop <a href="https://doi.org/10.1016/j.physbeh.2015.03.025">eating</a> rather than hunger or desire.</p> <h2>Is there anything I can do to resist cravings?</h2> <p>We largely don’t have control over cues in our environment and the cravings they trigger. But there are some ways you can try and control the situations you make food choices in.</p> <ul> <li> <p><strong>Acknowledge your craving and think about a healthier way to satisfy it</strong>. For example, if you’re craving chips, could you have lightly-salted nuts instead? If you want something sweet, you could try fruit.</p> </li> <li> <p><strong>Avoid shopping when you’re hungry, and make a list beforehand</strong>. Making the most of supermarket “click and collect” or delivery options can also help avoid ads and impulse buys in the aisle.</p> </li> <li> <p><strong>At home, have fruit and vegetables easily available – and easy to see</strong>. Also have other nutrient dense, fibre-rich and unprocessed foods on hand such as nuts or plain yoghurt. If you can, remove high-fat, sugar-rich and salty foods from your environment.</p> </li> <li> <p><strong>Make sure your goals for eating are <a href="https://www.aafp.org/pubs/fpm/issues/2018/0300/p31.html">SMART</a></strong>. This means they are specific, measurable, achievable, relevant and time-bound.</p> </li> <li> <p><strong>Be kind to yourself</strong>. Don’t beat yourself up if you eat something that doesn’t meet your health goals. Just keep on trying.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/237035/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> </li> </ul> <p><em><a href="https://theconversation.com/profiles/gabrielle-weidemann-91497">Gabrielle Weidemann</a>, Associate Professor in Psychological Science, <a href="https://theconversation.com/institutions/western-sydney-university-1092">Western Sydney University</a> and <a href="https://theconversation.com/profiles/justin-mahlberg-1634725">Justin Mahlberg</a>, Research Fellow, Pyschology, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a></em></p> <p><em>Image credits: Shutterstock </em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/what-causes-food-cravings-and-what-can-we-do-about-them-237035">original article</a>.</em></p> </div>

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The Adventures of Priscilla, Queen of the Desert at 30: more important, enjoyable and vital than ever

<div class="theconversation-article-body"><em><a href="https://theconversation.com/profiles/stephen-gaunson-265553">Stephen Gaunson</a>, <a href="https://theconversation.com/institutions/rmit-university-1063">RMIT University</a></em></p> <p>The lavender painted bus named “Priscilla” continues to pick up new fans while never going out of favour with its legion of original devotees, 30 years after its release.</p> <p>The Adventures of Priscilla, Queen of the Desert was shot on location in and around Sydney, Broken Hill, Coober Pedy, Kings Canyon and Alice Springs over six weeks in 1993.</p> <p>Directed by Stephan Elliott, the film screened in the <em>Un Certain Regard</em> section of the May 1994 Cannes Film Festival, winning <a href="https://nla.gov.au/nla.obj-885750952/view?sectionId=nla.obj-888737519&amp;searchTerm=priscilla+queen+of+the+desert&amp;partId=nla.obj-885806856#page/n13/mode/1up">critical and popular acclaim</a> for its positive portrayal of LGBTQI+ characters.</p> <p>Awards came, most notably for costume designers Tim Chappel and Lizzy Gardiner who won the Academy Award for Best Costume Design for their sparkly, sequin-filled costumes.</p> <p>The film’s cultural brilliance lies in juxtaposing the extreme flamboyance of the costumes and props against the equally extreme rural natural desert landscape. The unexpected revelation for audiences was how perfectly these contrasting elements harmonised.</p> <figure><iframe src="https://www.youtube.com/embed/wGWWeourHUg?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <h2>A smash hit</h2> <p>Less than 12 months since its release, the Film Finance Corporation (FFC) of Australia had the rare success of <a href="https://if.com.au/why-the-adventures-of-priscilla-queen-of-the-desert-turned-the-government-a-profit-in-just-12-months/">fully recouping</a> its A$1.67 million investment.</p> <p>Initially hesitant due to Elliott’s disappointing box office return on his debut feature, Frauds (1993), the FFC was convinced after the screenplay gained attention at Cannes. The film exceeded predictions, grossing more than $16 million in Australia.</p> <p>The film was socially and critically embraced as an instant classic.</p> <p>Ask cinema employees from this time and they will all share similar memories of lines of people outside the cinema doors eager to watch and rewatch (and rewatch again) the musical road movie about a pair of drag queens (Hugo Weaving and Guy Pearce) and a transgender woman (Terence Stamp) as they set out from Sydney on a bus journey across the Australian outback.</p> <figure><iframe src="https://www.youtube.com/embed/tex3dw3RjGk?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <p>The casting today seems like more of a sure bet than it did in 1994. Stamp was a British actor of <a href="https://www.theguardian.com/film/2015/mar/12/terence-stamp-i-was-in-my-prime-but-when-the-60s-ended-i-ended-with-it">legendary status</a>, having gained critical accolades in the 1960s in films such as Billy Budd, The Collector and Far from the Madding Crowd. However Stamp was equally a <a href="https://www.dailymail.co.uk/home/event/article-4869258/Terence-Stamp-79-bids-farewell-active-sex-life.html">regular tabloid subject</a> for his high-profile romances with film star Julie Christie and supermodel Jean Shrimpton.</p> <p>Would audiences be willing to go with the idea of this playboy as the trandsgender woman, Bernadette?</p> <p>It is now impossible to consider any actor better able to deliver Terrance Stamps’s deadpan sardonic lines so perfectly: “Don’t ‘Darling’, me, Darling. Look at you. You’ve got a face like a cat’s arse.”</p> <p>Pearce and Weaving also were a risk. Neither were box office marquee stars at the time. Pearce was known as a lovable Mike from the popular television soap opera, Neighbours. Weaving was a critically respected actor known more for his quirky small parts than as a star in his own right.</p> <p>Both were perfect casting, launching them onto Hollywood careers. Pearce as Adam was a remarkable revelation.</p> <figure><iframe src="https://www.youtube.com/embed/p2QiCFAQ-qQ?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <p>The chemistry and connection between the three lead actors makes the film truly succeed.</p> <p>Never do their performances seem showboating or forced. Each has their own arc, personality and journey. And when they climb Kings Canyon in full drag regalia at the film’s end there is something moving about what they have been able to accomplish together.</p> <p>A film that begins to be a slight and joyful comedy about drag performers becomes a deeper essay on the importance of lived experience and friendship (or, dare I say, mateship).</p> <h2>More vital than ever</h2> <p>Drag has a <a href="https://www.remindmagazine.com/article/3894/drag-through-decades-film-tv/">long history</a> in mainstream cinema with its own codes and references.</p> <p>The Adventures of Priscilla, Queen of the Desert is crucially different by it not being about “straight” men masquerading as women to mask their actual identity. Instead, Elliot’s film rather positively celebrates these characters in drag as their true and authentic selves.</p> <p>This film stood with others as a wave of Australian cinema in the 1990s unashamedly wanting to celebrate an Australia juxtaposing the blokey masculine stereotype.</p> <p>Ocker characters (men and women) appear in this film, but ultimately they are publicly humiliated for their homophobia. Bernadette kneeing in the groin the vicious and vulgar Frank (Kenneth Radley) often receives a big cheer from cinema audiences: “Stop flexing your muscles, you big pile of budgie turd,” Bernadette scorns.</p> <figure><iframe src="https://www.youtube.com/embed/9nc12yOA4jM?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <p>Perhaps the true star of the film were not the actors as much as the iconic 1976 Japanese model Hino Freighter Priscilla bus that became the set for several scenes in the film. Because the bus interior was such a small set, there was no room for the crew with many actually in shot, hiding under clothes and other props.</p> <p>The bus, long thought to be lost, was <a href="https://www.abc.net.au/news/2024-04-12/bid-to-restore-priscilla-queen-of-the-desert-bus/103699136">rediscovered</a> on a farm in New South Wales in 2019. The bus is currently being restored, with an aim to have it on display at the National Motor Museum in Birdwood, Adelaide Hills, in 2026. Perhaps it will be good timing for shooting the recently announced <a href="https://deadline.com/2024/04/priscilla-queen-of-the-desert-movie-sequel-original-cast-1235890358/">potential sequel</a>.</p> <p>30 years after its original release The Adventures of Priscilla, Queen of the Desert seems more important, enjoyable and vital than ever. All aboard, Priscilla. Long may she run.</p> <hr /> <p><em>RMIT Capitol will be hosting a <a href="https://www.rmit.edu.au/thecapitol/events/2024/september/the-adventures-of-priscilla-queen-of-the-desert">screening and introductory panel discussion</a> on September 11 with the film’s costume designer Tim Chappel, executive producer Rebel Penfold-Russell, Melbourne Queer Film Festival program director Cerise Howard, historian Kristy Kokegei and Stephen Gaunson.</em><!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/235424/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><a href="https://theconversation.com/profiles/stephen-gaunson-265553"><em>Stephen Gaunson</em></a><em>, Associate Professor in Cinema Studies, <a href="https://theconversation.com/institutions/rmit-university-1063">RMIT University</a></em></p> <p><em>Image credits: Metro Goldwyn Mayer</em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/the-adventures-of-priscilla-queen-of-the-desert-at-30-more-important-enjoyable-and-vital-than-ever-235424">original article</a>.</em></p> </div>

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Do mobile phones cause brain cancer? Science makes definitive call

<p>The question of whether mobile phones - specifically the electromagnetic radiation or radio waves emitted by these devices - cause cancer has been debated and researched for a long time, and now scientists have made a definitive call. </p> <p>A new comprehensive review commissioned by the World Health Organization has found that mobile phones are NOT linked to brain and head cancers. </p> <p>The systematic review, led by the Australian Radiation Protection and Nuclear Safety Agency (Arpansa), examined over 5,000 studies, which included 63 observational studies on humans published between 1994 and 2022 and is "the most comprehensive review to date" according to review lead author, associate prof Ken Karipidis. </p> <p>“We concluded the evidence does not show a link between mobile phones and brain cancer or other head and neck cancers," he said. </p> <p>The review, which was published on Wednesday, focused on cancers of the nervous system, salivary gland and brain tumours. </p> <p>They found no overall association between mobile phone use and cancer, even if people have used it for a long time (over 10 years) or spend a lot of time on their phones. </p> <p>“I’m quite confident with our conclusion. And what makes us quite confident is … even though mobile phone use has skyrocketed, brain tumour rates have remained stable,” Karipidis continued. </p> <p>Despite emitting electromagnetic radiation, also known as radio waves, the exposure is relatively low. </p> <p>Karipidis said people hear the word radiation and assume it is similar to nuclear radiation, “and because we use a mobile phone close to the head when we’re making calls, there is a lot of concern.”</p> <p>He clarified that “radiation is basically energy that travels from one point to another. There are many different types, for example, ultraviolet radiation from the sun." </p> <p>“We’re always exposed to low-level radio waves in the everyday environment.”</p> <p>While exposure from mobile phones is still low, it is much higher than exposure from any other wireless technology sources since they are used close to the head, Karipidis said. </p> <p>The association between mobile phones and cancers came about from early studies comparing differences between those with and without brain tumours and asking about their exposure history. </p> <p>According to Karipidis, who is also the vice-chair of the International Commission on Non-Ionizing Radiation Protection, the results from these kind of studies tend to be biased, as the group with the tumour tend to overreport their exposure. </p> <p>Based on these early studies WHO’s International Agency for Research on Cancer (IARC) designated radio-frequency fields like those from mobile phones as a possible cancer risk, but Karipidis said "this classification doesn’t mean all that much”.</p> <p>This is because the IARC has different classifications of cancer risk, with some substances classified as  a “definite” carcinogen (such as smoking), and others as “probable” or “possible” carcinogens.</p> <p>Tim Driscoll, a professor at the University of Sydney and chair of the Australian Cancer Council’s occupational and environmental cancers committee, also backed the systematic review. </p> <p>“I think people should feel reassured by this study … but it’s worthwhile just remembering that the studies aren’t perfect, but the weight of evidence certainly is that mobile phones should be considered safe to use in terms of any concerns about increased risk of cancer,” Driscoll said.</p> <p><em>Images: Shutterstock</em></p> <p> </p>

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Flu shots play an important role in protecting against bird flu. But not for the reason you might think

<div class="theconversation-article-body"><em><a href="https://theconversation.com/profiles/allen-cheng-94997">Allen Cheng</a>, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a></em></p> <p>A current strain of highly pathogenic avian influenza, commonly known as bird flu, has become a global problem. The virus has affected <a href="https://www.cdc.gov/bird-flu/situation-summary/data-map-commercial.html">many millions</a> of birds, some other <a href="https://www.cdc.gov/bird-flu/situation-summary/mammals.html">animal species</a>, and a <a href="https://cdn.who.int/media/docs/default-source/influenza/avian-and-other-zoonotic-influenza/joint-fao-oie-who-preliminary-risk-assessment-associated-with-avian-influenza-a(h5n1)-virus.pdf?sfvrsn=faa6e47e_28&amp;download=true">small number of people</a>.</p> <p>Last week, the Australian government <a href="https://www.sbs.com.au/news/article/australians-issued-new-health-risks-travel-warning-for-europe-asia-and-the-americas/gmh1hk9py">issued a warning</a> to residents travelling to Europe, North America, South America and Asia about the risk of bird flu.</p> <p>The alert, published on the <a href="https://www.smartraveller.gov.au/news-and-updates/highly-pathogenic-avian-influenza-outbreak">Smartraveller website</a>, included advice to ensure your flu vaccine is up to date. If you are about to go travelling, this generally means if you’ve had a flu jab this year, although if it has been 3–6 months since your vaccine you should discuss this with your doctor.</p> <p>But the seasonal flu vaccine we get each year doesn’t actually prevent bird flu in humans. So why is it being recommended in this context?</p> <h2>Some bird flu background</h2> <p>Smartraveller notes <a href="https://www.smartraveller.gov.au/news-and-updates/highly-pathogenic-avian-influenza-outbreak">several strains</a> of bird flu are currently circulating.</p> <p>The most concerning strain, called the <a href="https://www.nature.com/articles/s41467-023-38415-7">2.3.4.4b clade</a>, emerged a few years ago from a type of influenza A (H5, or A/H5) that has been circulating for several decades.</p> <p>Clade 2.3.4.4b primarily affects birds, including wild birds and poultry. It has had <a href="https://theconversation.com/uk-poultry-can-roam-free-outside-again-but-bird-flu-risk-hasnt-gone-away-203361">devastating effects</a> on bird populations, as well as farmers and others involved in the poultry industry.</p> <p>In recent years, clade 2.3.4.4b has adapted to <a href="https://www.nature.com/articles/s44298-024-00039-z">infect some mammals</a>. Unfortunately it seems to cause severe disease in <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11083745/">certain animals</a>. Some marine mammals have been hit particularly hard, with mass mortality events <a href="https://www.theguardian.com/environment/2023/dec/08/mass-deaths-elephant-seals-penguins-bird-flu-antarctic-ecological-disaster-aoe">reported</a> in elephant seals and sea lions. In the United States, bird flu has also spread <a href="https://www.cdc.gov/bird-flu/situation-summary/mammals.html">among dairy cows</a>.</p> <p>Compared to the huge number of animal cases, there have been a relatively small number of <a href="https://www.who.int/publications/m/item/joint-fao-who-woah-preliminary-assessment-of-recent-influenza-a(h5n1)-viruses">humans infected with bird flu</a>. Since 2003, <a href="https://www.who.int/publications/m/item/cumulative-number-of-confirmed-human-cases-for-avian-influenza-a(h5n1)-reported-to-who--2003-2023--3-october-2023">878 cases</a> of A/H5N1 influenza have been reported in humans, with a small proportion of these reported since 2020 when <a href="https://www.outbreak.gov.au/emerging-risks/high-pathogenicity-avian-influenza">clade 2.3.4.4b first emerged</a>. The reported cases have been people who have had close contact with infected animals. It does not appear to spread from person to person.</p> <p>As such, the <a href="https://www.ecdc.europa.eu/en/infectious-disease-topics/z-disease-list/avian-influenza/threats-and-outbreaks/risk-assessment-h5">risk to travellers is low</a>. There are some situations where the risk may be greater, such as for people visiting live markets, or those who are travelling specifically to work with wildlife or animals in food production.</p> <p><a href="https://www.who.int/publications/m/item/joint-fao-who-woah-preliminary-assessment-of-recent-influenza-a(h5n1)-viruses">Infections in humans</a> with H5 influenza can vary significantly in severity, from mild conjunctivitis up to fatal pneumonia. H5 influenza strains appear to be <a href="https://asm.org/articles/2024/june/what-you-should-know-about-avian-influenza-a-h5n1">sensitive to antivirals</a> (oseltamivir, also known as Tamiflu) and they are generally <a href="https://www.cdc.gov/bird-flu/hcp/novel-av-treatment-guidance/">recommended</a> as treatment for human infection, but it’s <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(24)01307-2/fulltext">not clear</a> whether they reduce the risk of death in those with severe disease.</p> <p>To date, one case of A/H5 influenza (not 2.3.4.4b) has been <a href="https://www.abc.net.au/news/2024-05-22/bird-flu-avian-influenza-human-detection/103879886">reported in Australia</a>, in a child who had recently returned from overseas.</p> <p>While <a href="https://www.fao.org/animal-health/situation-updates/global-aiv-with-zoonotic-potential/en">clade 2.3.4.4b has been detected</a> in all continents <a href="https://www.outbreak.gov.au/emerging-risks/high-pathogenicity-avian-influenza">except Australia</a>, other avian influenza strains (A/H7) <a href="https://www.outbreak.gov.au/current-outbreaks/avian-influenza">have been reported here</a> earlier this year.</p> <h2>Seasonal flu vaccines are not effective against bird flu</h2> <p>Seasonal influenza refers to the flu strains that circulate each year. Since the COVID pandemic, three different strains have circulated in various proportions – influenza A H1N1 (descended from the <a href="https://www.nature.com/articles/nature08182">2009 swine flu strain</a>), influenza A H3N2 (which has <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6149781/">circulated since 1968</a>) and an influenza B strain. Interestingly, a second influenza B strain (the Yamagata lineage) <a href="https://www.nejm.org/doi/full/10.1056/NEJMp2314801">appears to have vanished</a> during the COVID pandemic.</p> <p>Seasonal influenza vaccines contain up-to-date variants of these types (A/H1N1, A/H3N2 and B) that are recommended by the World Health Organization each year. They are <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5912669/">moderately effective</a>, reducing the risk of hospitalisation by about 40–60%.</p> <p>Influenza vaccines are quite specific in the protection that they provide. For seasonal vaccines, even the very small changes that occur in the virus from year to year are enough to allow them to “escape” vaccine-induced immunity. Therefore seasonal flu vaccines <a href="https://www.cdc.gov.au/topics/avian-influenza-bird-flu">do not provide any protection</a> against A/H5 influenza.</p> <h2>Preventing a hybrid bird-human strain</h2> <p>The rationale for recommending travellers have a flu shot in the context of the current bird flu outbreak is that seasonal flu vaccines may help reduce the risk of simultaneous infection with both A/H5 and a seasonal influenza strain.</p> <p>When this occurs, there is potential for a “recombination” of the genetic code from both viral strains. This could have the transmissibility of a seasonal human virus with the severity of an avian influenza virus. The 2009 swine flu strain <a href="https://www.nejm.org/doi/full/10.1056/NEJMra0904322">arose from the recombination</a> of several strains over years to become more transmissible in humans.</p> <p>Obviously a more effective vaccine would include a H5 strain, to generate immune responses specific to the H5 flu strain. Vaccine manufacturers have <a href="https://www.ema.europa.eu/en/medicines/human/EPAR/celldemic">developed H5 vaccines</a> over the years, but to date <a href="https://thl.fi/en/-/avian-influenza-vaccinations-begin-vaccine-to-be-offered-to-persons-at-increased-risk-of-infection">only Finland</a> has deployed a H5 vaccine in a small group of people who work closely with potentially infected animals.</p> <p>Currently the <a href="https://www.ecdc.europa.eu/en/infectious-disease-topics/z-disease-list/avian-influenza/threats-and-outbreaks/risk-assessment-h5">level of risk</a> posed by H5 to humans is not thought to be sufficient to require a specific vaccine program, as the potential benefits are small compared to the costs and the potential risks associated with any new vaccine program.</p> <h2>The value of a flu shot for travellers</h2> <p>Seasonal flu vaccines protect against influenza infection, and may also reduce the risk of simultaneous infection with human and bird flu strains. Bird flu aside, for most travellers who haven’t received a flu shot this year, reducing the risk of illness disrupting travel plans should be enough of a reason to get one.</p> <p>For those who have already received a flu shot this season, similar to COVID jabs, protection after vaccination appears to <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8499703/">wane over time</a>. So if you’re travelling to the northern hemisphere during the winter months, and it’s been more than 3–6 months since you received a flu vaccine, your doctor may recommend you have another.</p> <p>Bird flu is only a small risk to most travellers, but people may want to take sensible precautions, such as avoiding close contact with birds at markets.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/237859/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em><a href="https://theconversation.com/profiles/allen-cheng-94997">Allen Cheng</a>, Professor of Infectious Diseases, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a></em></p> <p><em>Image credits: Shutterstock </em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/flu-shots-play-an-important-role-in-protecting-against-bird-flu-but-not-for-the-reason-you-might-think-237859">original article</a>.</em></p> </div>

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The important cause that brought Ally Langdon to tears

<p>Ally Langdon teared up on Thursday's episode of <em>A Current Affair </em>during a heartfelt segment on organ donation. </p> <p>The host was discussing Jersey Day with Michael Gremmo, a father who had lost his 13-year-old son Nathan after he was critically injured when crossing the road in 2014. </p> <p>Nathan went on to become an organ donor  - a decision that saved six lives, including a baby. </p> <p>Now his father is honouring his son's legacy by encouraging others to have the important conversation about organ and tissue donation with loved ones. </p> <p>"Nathan, as most organ donors are, are healthy, fit, young people and their lives are just taken from them," Michael said.</p> <p>"Dealing with Nathan not coming back, that's the hardest, but we wanted something positive to come out of a horrible situation."</p> <p>The <em>A Current Affair</em> host fought back tears as she recalled how her own sister's life was saved after receiving a pancreas-kidney  transplant in 2014. </p> <p>"My sister is alive because of someone as special as Nathan and there’s not a day goes by that we don’t as a family think about them and think about their grief." </p> <p>“Thank you,” Langtold told Michael tearfully.</p> <p>Michael replied: “Nathan hasn’t died in vain. He’s given life. And you’ve got your sister here because of a generous donor. And it’s not just the donor, it’s their family too. When you’re in that situation it’s hard.”</p> <p>Langdon then encouraged viewers to "have the conversation" about organ donation with loved ones of all ages. </p> <p>"Tell your family your wishes, because the final decision is theirs," she said.</p> <p>Michael said despite his son's young age, Nathan had talked about organ donation and expressed his desire to be one before his passing. </p> <p>“It’s not a conversation you usually have or want to have with young children,” Michael said.</p> <p>"Nathan had actually had a conversation with us, he watched a program on TV and it was about organ recipients ... he sat us down and spoke to us about that.</p> <p>"And yeah, we just talked about what a wonderful gift that would be."</p> <p>Jersey Day falls on Friday August 30, during which students and employees at schools and workplaces across the country are encouraged to wear their favourite jersey and have the important conversation about organ donation with their loved ones.</p> <p>"It's not just the lives of the organ recipient [you save], it's the families behind that too that, you know, you don't realise how many people are affected," Michael said.</p> <p>"I suppose that's why Jersey Day is so special as it's about awareness and getting it out there and having that important conversation."</p> <p><em>To register as an organ and tissue donor, visit <a href="http://donatelife.gov.au/" target="_blank" rel="noopener">donatelife.gov.au.</a></em></p> <p><em>Images: A Current Affair/ Nine</em></p>

Caring

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"Sexy Santas" cause outrage at Seven staff meeting

<p>Channel 7 has come under fire after footage of a routine staff meeting featured scantily clad dancers dressed as Santa. </p> <p>The meeting of Seven Network parent company Seven West Media in Perth was held on Friday to introduce a series of new hires and appointments, while deputy news director Ray Kuka also discussed upcoming programming for the rest of the year. </p> <p>As part of the announcement, Kuka spoke of Perth’s annual Christmas Pageant on December 7th, when Mariah Carey’s <em>All I Want for Christmas</em> started playing and a group of female dancers wearing Santa hats and short red dresses appeared and started dancing. </p> <p>Female staff who watched on were said to have been "dumbstruck and horrified" by the bizarre scene, with <em><a href="https://www.afr.com/rear-window/stokes-woke-regime-comes-with-sexy-surprise-20240825-p5k54h" target="_blank" rel="noopener">The Australian Financial Review</a></em> claiming many likened the dancers to "sl*tty elves". </p> <p>A photo of the event was shared online and drew heavy criticism, as one worker shared that the performance prompted a mass walk out of outraged staffers. </p> <p>“OMG – if they thought that was even slightly appropriate given everything we’ve found out about them, the cultural makeup of that organisation is badly broken,” one person wrote online. </p> <p>“That will help fix the culture issues which have been uncovered.”</p> <p>A spokesman for Seven West Media defended the dancers, explaining that they were part of the Christmas Pageant, which is a “Perth institution”.</p> <p>The footage of the meeting comes as Seven faces a series of internal challenges as they fight allegations of being a "toxic" workplace. </p> <p>Ryan Stokes, the managing director of Seven Group Holdings Ltd, recently called inappropriate behaviour in media an "industry-wide problem".</p> <p>Stokes said it was "disappointing that there is a perception [that] inappropriate behaviour" was tolerated at Seven because "we don't tolerate any inappropriate behaviour".</p> <p><em>Image credits: Seven West Media</em></p>

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Richard Simmons' cause of death revealed

<p>Richard Simmons' brother, Lenny, has revealed the fitness guru's cause of death was accidental. </p> <p>Lenny confirmed the 76-year-old's cause of death in a statement on Wednesday (Thursday AEST), and also thanked fans for the "outpouring of love" following his death.</p> <p>"This morning, Richard Simmons' brother Lenny, received a call from the LA Coroner's office," family spokesperson Tom Estey said in a statement to <em>People</em> magazine. </p> <p>"The coroner informed Lenny that Richard's death was accidental due to complications from recent falls and heart disease as a contributing factor.</p> <p>"The toxicology report was negative other than medication Richard had been prescribed.</p> <p>"The family wishes to thank everyone for their outpouring of love and support during this time of great loss."</p> <p>Simmons, who rose to fame in the 80s as a TV fitness icon was found dead by his longtime housekeeper on July 13- a day after his 76th birthday. </p> <p>His brother previously paid tribute to the icon in an interview with <em>People </em>magazine. </p> <p>"I don't want people to be sad about my brother," he said just hours after the news of his brother's death. </p> <p>"I want them to remember him for the genuine joy and love he brought to people's lives. He truly cared about people.</p> <p>"He called, wrote and emailed thousands of people throughout his career to offer help.</p> <p>"So don't be sad. Celebrate his life."</p> <p><em>Images: Instagram</em></p> <p> </p>

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Distraught wife of cricket legend reveals cause of death

<p>The grieving wife of late cricket legend Graham Thorpe has spoken out after her husband's untimely <a href="https://oversixty.com.au/health/caring/tragic-news-cricket-legend-dies-at-just-55" target="_blank" rel="noopener">death</a>, revealing that he tragically took his own life. </p> <p>After an illustrious cricket career in the UK, Thorpe died on August 5th at just 55 years old, leaving behind his wife and four children. </p> <p>Thorpe had previously been hospitalised in May 2022 following a suicide attempt, but made a full recovery. </p> <p>His wife Amanda has spoken out about her husband's mental illness, sharing how he had been suffering for many years. </p> <p>“Despite having a wife and two daughters whom he loved and who loved him, he did not get better,” Amanda told <em>The Times</em>.</p> <p>“He was so unwell in recent times and he really did believe that we would be better off without him.”</p> <p>She added: “We are devastated that he acted on that and took his own life.”</p> <p>Amanda said Graham had suffered from major depression and anxiety “for a couple of years”, explaining, “Despite glimpses of hope and of the old Graham, he continued to suffer from depression and anxiety, which at times got very severe."</p> <p>“We supported him as a family and he tried many, many treatments but unfortunately none of them really seemed to work."</p> <p>“Graham was renowned as someone who was very mentally strong on the field and he was in good physical health. But mental illness is a real disease and can affect anyone.”</p> <p>Following his sudden death, the England and Wales Cricket Board released a statement that said, “It is with great sadness that we share the news that Graham Thorpe, MBE, has passed away."</p> <p>“There seem to be no appropriate words to describe the deep shock we feel at Graham’s death."</p> <p>“More than one of England’s finest-ever batters, he was a beloved member of the cricket family and revered by fans all over the world."</p> <p><em><strong>Need to talk to someone? Don't go it alone. Call Lifeline on 13 11 14 or <a href="https://www.lifeline.org.au/" target="_blank" rel="noopener noreferrer">lifeline.org.au</a></strong></em></p> <p><em>Image credits: Colorsport/Shutterstock Editorial</em></p>

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Three-time mayor's cause of death revealed

<p>Rising Labor star Sarah Carter was found dead at her home on Tuesday, when she failed to meet her partner <a href="https://www.oversixty.com.au/health/caring/tragic-new-details-emerge-after-three-time-mayor-found-dead-at-just-45" target="_blank" rel="noopener">Nikhil Roy</a> at the airport ahead of their Europe trip. </p> <p>She was treated by emergency services but could not be saved. </p> <p>Now a source close to Carter has told the <em>Daily Mail</em> that she is believed to have of a suspected brain aneurysm. </p> <p> “They suspect it’s an aneurysm ... we don’t know whether it was anything she had been aware of. It’s come as a complete shock," the source reportedly told the outlet. </p> <p>On Wednesday Carter's devastated mother Gaynor Morris spoke out about the tragedy for the first time in an interview with the Herald Sun. </p> <p>“She hadn’t made it to the airport to meet him for their holiday together,” she said. </p> <p>“He (Roy) went and checked on her and he literally found her dead.”</p> <p>Victoria police had previously confirmed that her death was not being treated as suspicious and a report is being prepared for the coroner. </p> <p>Carter was a councillor for Maribyrnong Council in Melbourne's west, covering suburbs including Footscray, Yarraville, Tottenham and Maribyrnong itself. </p> <p>She was first elected to office in 2008 and was the first woman to have served three times as mayor of Maribyrnong. </p> <p><em>Image: Instagram</em></p>

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Cause of death revealed after eight-year-old dies mid-flight

<p>The cause of death of an eight-year-old girl who died suddenly mid-flight has been revealed. </p> <p>Sydney Weston was travelling with her family from their home state of Missouri to Chicago on June 13th when she suddenly fell ill on the flight and died. </p> <p>Now, according to Peoria County Coroner Jamie Harwood, Sydney was suffering from at least five different illnesses at the time of her death. </p> <p>The eight-year-old reportedly died from complications of chronic primary adrenalitis coupled with several infections, including enterovirus and strep, as well as inflammation of the small intestines known as duodenitis and the thyroid gland inflammation commonly known as thyroiditis. </p> <p>Harwood found that the long list of conditions suggests Sydney may have suffered from an autoimmune disorder which left her body attacking its own healthy cells and harming organ operation. </p> <p>Sydney suffered a medical emergency onboard the flight, causing the plane to divert to Peoria, Illinois, for an emergency landing, but Sydney died after being rushed to a nearby hospital.</p> <p>Results from a previously released preliminary autopsy were inconclusive but found that her body showed no signs of abuse, neglect or foul play.</p> <p>At the time of her sudden and tragic death, Sydney was just a week away from celebrating her ninth birthday.</p> <p>“She will forever be remembered for the happiness she brought to every single person she encountered. Our hearts are heavy. She was our baby girl and we celebrate her beautiful life,” her obituary read.</p> <p><em>Image credits: Instagram </em></p>

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What is competition, and why is it so important for prices?

<div class="theconversation-article-body"> <p><em><a href="https://theconversation.com/profiles/paul-blacklow-1546097">Paul Blacklow</a>, <a href="https://theconversation.com/institutions/university-of-tasmania-888"><em>University of Tasmania</em></a></em></p> <p>It’s hard to remember a time before the <a href="https://theconversation.com/you-dont-have-to-be-an-economist-to-know-australia-is-in-a-cost-of-living-crisis-what-are-the-signs-and-what-needs-to-change-210373">cost-of-living crisis</a> dominated news headlines. Most of us would certainly like it to be over.</p> <p>But the fundamental question at its heart – which points to the problem we have to solve – seems simple. What determines the prices we pay?</p> <p>The cost of producing goods and services is certainly one big factor in determining how much we pay for them. So, too, is what we’re prepared to fork out.</p> <p>But when we talk about lowering prices, we often also talk about increasing competition – the number of firms vying to sell us a particular offering.</p> <p>It’s so important for efficient pricing that the government body tasked with making our markets fair is called the Australian Competition and Consumer Commission – the “ACCC” for short.</p> <p>But why does having more people trying to sell us things drive down their prices? And can companies find ways to get around this?</p> <h2>More sellers, lower prices</h2> <p>In a free market system, there are a few different types of competition.</p> <p>In the most ideal, a <strong>perfectly competitive</strong> market, firms must use resources efficiently to produce what we consumers want at the lowest possible cost.</p> <p>In <strong>perfect competition</strong>:</p> <ul> <li>the products and services traded are identical (or very similar)</li> <li>there are many buyers and sellers</li> <li>information is perfect</li> <li>firms can enter and exit freely.</li> </ul> <p>A firm charging prices well above the minimum cost will sell no goods or services and be forced to leave the market. Why? Because its competitors will be able to steal customers by charging slightly less for exactly the same thing.</p> <p>Only lower-cost firms will remain and compete prices down until they cover the lowest cost of supplying the good or service, plus an average or normal “return on capital”.</p> <p>At a high level, think of this return as an acceptable monetary reward for the business for investing the inputs and taking on the risks required to operate.</p> <p>If ever an industry is earning above-average returns given its level of risk, new firms will enter and charge less, until only normal returns are earned.</p> <p>Conversely, below-normal profits will see firms exit, decreasing supply and raising prices.</p> <p>Do perfectly competitive markets exist? There are arguably some examples that come close, such as casual labour services, some agricultural commodities like grain, livestock and fruit, and financial and currency markets.</p> <p>But there are more examples of less competitive markets.</p> <h2>The winner takes it all</h2> <p>At the opposite extreme, in <strong>monopoly</strong> markets, there is only one seller of a good or service. Typically, there is some barrier preventing new firms from entering the market and driving prices down.</p> <p>Without government regulation, monopoly firms will reduce supply, increase prices and earn above-normal profit levels.</p> <p>However, sometimes monopolies emerge naturally because it is far more efficient to have a single coordinating supplier of a particular service – such as in letter delivery, rail tracks, or internet infrastructure.</p> <p>To strike a balance, governments typically regulate or own monopolies.</p> <h2>Same same, but different</h2> <p>More common than monopoly is what’s called <strong>monopolistic competition</strong>, which is the market structure for many of our tech, entertainment and dining goods and services.</p> <p>In monopolistic competition, firms try to make their offering <em>different</em> by investing in R&amp;D and advertising, so that they do not have to compete on price alone.</p> <p>Think Apple’s iPhone versus Samsung’s Galaxy. Both are technically the same kind of product, but have created their own unique markets.</p> <p>Differentiation allows firms to price above minimum cost and earn above-normal rates of return. At least, that is, until new firms enter and imitate them, increasing supply and lowering prices and profits to normal levels.</p> <h2>A few big players hold market power</h2> <p>In Australia, many key goods and services are traded in <strong>oligopoly</strong> markets.</p> <p>Oligopolies arise when a few large firms dominate a particular industry, such as supermarkets, domestic airlines, banking, mobile telecommunications, and petrol retailing.</p> <p>Some oligopoly markets are very competitive and drive prices down to cost, plus normal return to capital. But in other more concentrated markets with a few powerful firms, firms may have significant <strong>market power</strong> and be able to keep prices above the competitive level.</p> <p>It is not illegal to possess market power, but according to Australia’s Competition and Consumer Act 2010, it is illegal to use it “for the purpose, effect, or likely effect of substantially lessening competition”.</p> <p>It is illegal, for example, for firms to explicitly work together when setting prices. This is called collusion. Neither can they force suppliers to deal with them exclusively, or set prices below cost when new firms attempt to enter a market.</p> <p>But that doesn’t mean some firms haven’t learned subtle and legal ways of reducing competition.</p> <p>For example, loyalty programs and charging special loss-leading prices can seem at first glance to be good for consumers, but can also increase the <a href="https://theconversation.com/loyalty-programs-may-limit-competition-and-they-could-be-pushing-prices-up-for-everyone-220669">cost of switching</a> to the lowest-priced firm.</p> <h2>Are we getting a good deal?</h2> <p>Still, you may have noticed the prices charged for many goods and services are very similar across different firms in the economy.</p> <p>Have these prices been driven down by competition to their cost plus a normal return to capital? Or are firms abusing their market power to lessen competition in the market?</p> <p>What can we do if firms are reducing competition through legal measures?</p> <p>These are just some of the difficult questions both government and industry are currently grappling with.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/234082/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><a href="https://theconversation.com/profiles/paul-blacklow-1546097"><em>Paul Blacklow</em></a><em>, Lecturer in Economics, <a href="https://theconversation.com/institutions/university-of-tasmania-888">University of Tasmania</a></em></p> <p><em>Image credits: Shutterstock </em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/what-is-competition-and-why-is-it-so-important-for-prices-234082">original article</a>.</em></p> </div>

Money & Banking

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Sinéad O'Connor's cause of death finally revealed

<p>One year after the tragic <a href="https://www.oversixty.com.au/news/news/sin-ad-o-connor-passes-away-at-just-56" target="_blank" rel="noopener">passing of iconic singer Sinead O'Connor</a> at just 56 years of age, the cause of her death has finally been officially revealed. According to the Irish Independent, O'Connor succumbed to complications from obstructive pulmonary disease and asthma.</p> <p>The Grammy Award-winning artist, known for her powerful voice and poignant lyrics, died from an "exacerbation of chronic obstructive pulmonary disease and bronchial asthma, together with a low-grade lower respiratory tract infection", as stated on her death certificate. This information was made public after the certificate was registered by her former husband, John Reynolds, in London on July 24.</p> <p>Julian Morris, the senior coroner for Inner South London, certified the cause of death following a thorough post-mortem examination. The news has brought a sombre clarity to the tragic loss of O'Connor, whose music touched the hearts of many worldwide.</p> <p>Sinead O'Connor, born in Dublin, was celebrated for her distinctive voice and fearless approach to music and social issues. Her legacy continues to inspire fans and musicians alike. </p> <p>O'Connor also courageously shared her battle with mental illness, revealing that she was diagnosed with bipolar disorder. In 2017, she posted a poignant video on Facebook while staying in a New Jersey motel, expressing that she was holding on for the sake of others and admitting her own struggles.</p> <p>In 2022, she faced immense heartache when her teenage son, Shane, died by suicide. O'Connor's emotional response on Twitter at the time – that there was "no point living without him" – led to her hospitalisation.</p> <p><em>Image: Instagram</em></p>

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Clint Eastwood's partner's cause of death revealed

<p>The cause of death of Clint Eastwood's partner has been revealed, just days after the actor addressed her tragic passing. </p> <p>Christina Sandera, Eastwood's partner of 10 years, died suddenly on July 18th at just 61 years of age. </p> <p>Now, a representative from the Monterey County Health Department confirmed to <em><a href="https://nypost.com/2024/07/23/entertainment/clint-eastwoods-partner-christina-sanderas-cause-of-death-revealed/" target="_blank" rel="noopener">The New York Post</a></em> that Sandera died of a heart attack. </p> <p>The death certificate listed her immediate cause as cardiac arrhythmia, which is an abnormality in the timing or pattern of the heartbeat.</p> <p>Atherosclerotic coronary artery disease was also outlined as an additional condition that led to Sandera’s death.</p> <p>Eastwood addressed the passing of his partner after the <em>Hollywood Reporter</em> confirmed her death, with the actor writing, “Christina was a lovely, caring woman, and I will miss her very much.”</p> <p>The pair met when Sandera worked as a hostess at a hotel and restaurant owned by the actor in California, before they started dating in 2014. </p> <p>The couple largely kept their romance out of the public eye with Sandera only attended a handful of events with the Hollywood icon during their 10-year relationship.</p> <p>The couple, who never married, walked their last red carpet together in November 2019 at AFI Fest in Hollywood. </p> <p>One of Eastwood's children from a previous marriage Morgan, 27, paid tribute to her father’s longtime love on Sunday by sharing a photo of herself, her father and Eastwood and her wedding, writing, “A devastating loss for our whole family. Thank you Christina for being a good partner to my Dad ❤️ you will be greatly missed.”</p> <p><em>Image credits: John McCoy/UPI/Shutterstock Editorial </em></p>

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Motive and cause of death revealed after six found dead in luxury hotel

<p>Six people found dead in a luxury hotel room in central Bangkok are believed to have died due to poisoning, according to Thai police. </p> <p>Authorities shared photo of the teacups found in the room on the fifth floor of the hotel, revealing they found traces of cyanide in the cups. </p> <p>The bodies of two American and four Vietnamese nationals were found by hotel staff on Tuesday evening, after staff entered the room to inform them it was past check out time. </p> <p>The group, made up of three men and three women aged between 37 and 56, likely drank from tea and coffee cups laced with cyanide following a dispute linked to bad investments, Thai police said on Wednesday.</p> <p>Photos of the crime scene released by authorities show a lack of struggle and violence, instead showing bodies scattered around the room among plates of untouched food, and two thermos flasks and cups.</p> <p>Initial examinations revealed the presence of cyanide in six cups, according to police.</p> <p>“The mouths and nails on all the bodies turned purple, showing a lack of air could be a joint reason of the cause of the death,” forensic doctor Kornkiat Vongpaisarnsin, told a press conference at the Chulalongkorn University.</p> <p>“We presume they all died from cyanide which causes a lack of air in some organs,” he added.</p> <p>Authorities initially said they were searching for a seventh person who was part of the hotel booking, but on Wednesday they dismissed this line of inquiry, saying they believe one of the dead people poisoned the others with the deadly fast-acting chemical cyanide.</p> <p>“We are convinced that one of the six people found dead committed this crime,” said Noppasil Poonsawas, a deputy commander of Bangkok police.</p> <p>That person, police said, had ordered the food and tea to the room and “looked under stress” when staff arrived.</p> <p>After conducting interviews with hotel staff, Noppasin said one of the members of the group was alone in the room when the food arrived and was later joined by the other guests.</p> <p>He added that the incident was likely linked to a “personal matter” and not related to organised crime as interviews carried out with relatives of the dead indicated a dispute over debt.</p> <p>“One of the relatives said one of the deceased was an investment agent and all (the deceased) invested, but the business was not going as expected. They made an appointment to discuss the matter in Thailand,” Noppasin said.</p> <p>Police believe that the woman who poisoned the group did so after she accrued huge debts related to an investment in a hospital in Japan, according to police sources cited by Vietnamese media. </p> <p>She had allegedly convinced a married couple and two other victims among the dead to pour funds into the project, but they had reportedly lost roughly $280,000 AUD as a result. </p> <p>Tran Dinh Dung, the father of one of the victims, said his 37-year-old son was due to return to Vietnam last Sunday.</p> <p>“I kept calling him but couldn’t get through so I was very worried, but I didn’t expect him to die in Thailand,” Dung said in an interview with Vietnam’s Thanh Nien newspaper.</p> <p>“Phu’s mother has fainted countless times, she could not bear this shock,” he said.</p> <p><em>Image credits: AFP/Anusak Laowilas/NurPhoto/Shutterstock Editorial</em></p>

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3 signs your diet is causing too much muscle loss – and what to do about it

<div class="theconversation-article-body"><em><a href="https://theconversation.com/profiles/nick-fuller-219993">Nick Fuller</a>, <a href="https://theconversation.com/institutions/university-of-sydney-841">University of Sydney</a></em></p> <p>When trying to lose weight, it’s natural to want to see quick results. So when the number on the scales drops rapidly, it seems like we’re on the right track.</p> <p>But as with many things related to weight loss, there’s a flip side: rapid weight loss can result in a significant loss of muscle mass, as well as fat.</p> <p>So how you can tell if you’re losing too much muscle and what can you do to prevent it?</p> <h2>Why does muscle mass matter?</h2> <p>Muscle is an important factor in determining our metabolic rate: how much energy we burn at rest. This is determined by how much muscle and fat we have. Muscle is more metabolically active than fat, meaning it burns more calories.</p> <p>When we diet to lose weight, we create a calorie deficit, where our bodies don’t get enough energy from the food we eat to meet our energy needs. Our bodies start breaking down our fat and muscle tissue for fuel.</p> <p>A decrease in calorie-burning muscle mass slows our metabolism. This quickly slows the rate at which we lose weight and impacts our ability to maintain our weight long term.</p> <h2>How to tell you’re losing too much muscle</h2> <p>Unfortunately, measuring changes in muscle mass is not easy.</p> <p>The most accurate tool is an enhanced form of X-ray called a dual-energy X-ray absorptiometry (DXA) scan. The scan is primarily used in medicine and research to capture data on weight, body fat, muscle mass and bone density.</p> <p>But while DEXA is becoming more readily available at weight-loss clinics and gyms, it’s not cheap.</p> <p>There are also many “smart” scales available for at home use that promise to provide an accurate reading of muscle mass percentage.</p> <p>However, the accuracy of these scales is questionable. <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8122302/">Researchers found</a> the scales tested massively over- or under-estimated fat and muscle mass.</p> <p>Fortunately, there are three free but scientifically backed signs you may be losing too much muscle mass when you’re dieting.</p> <h2>1. You’re losing much more weight than expected each week</h2> <p>Losing a lot of weight rapidly is one of the early signs that your diet is too extreme and you’re losing too much muscle.</p> <p>Rapid weight loss (of more than 1 kilogram per week) results in <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5702468/">greater muscle mass loss</a> than slow weight loss.</p> <p>Slow weight loss better preserves muscle mass and often has the added benefit of <a href="https://www.sciencedirect.com/science/article/pii/S0195666312000153">greater fat mass loss</a>.</p> <p>One study compared people in the obese weight category who followed either a very low-calorie diet (500 calories per day) for five weeks or a low-calorie diet (1,250 calories per day) for 12 weeks. While both groups lost similar amounts of weight, participants following the very low-calorie diet (500 calories per day) for five weeks lost <a href="https://pubmed.ncbi.nlm.nih.gov/26813524/">significantly more muscle mass</a>.</p> <h2>2. You’re feeling tired and things feel more difficult</h2> <p>It sounds obvious, but feeling tired, sluggish and finding it hard to complete physical activities, such as working out or doing jobs around the house, is another strong signal you’re losing muscle.</p> <p><a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3648712/">Research</a> shows a decrease in muscle mass may negatively impact your body’s physical performance.</p> <h2>3. You’re feeling moody</h2> <p>Mood swings and feeling anxious, stressed or depressed may also be signs you’re losing muscle mass.</p> <p><a href="https://pubmed.ncbi.nlm.nih.gov/26228522/">Research</a> on muscle loss due to ageing suggests low levels of muscle mass can negatively impact mental health and mood. This seems to stem from the relationship between low muscle mass and proteins called neurotrophins, which help regulate mood and feelings of wellbeing.</p> <h2>So how you can do to maintain muscle during weight loss?</h2> <p>Fortunately, there are also three actions you can take to maintain muscle mass when you’re following a calorie-restricted diet to lose weight.</p> <h2>1. Incorporate strength training into your exercise plan</h2> <p>While a broad exercise program is important to support overall weight loss, strength-building exercises are a surefire way to help prevent the loss of muscle mass. A <a href="https://pubmed.ncbi.nlm.nih.gov/29596307/">meta-analysis of studies</a> of older people with obesity found resistance training was able to prevent almost 100% of muscle loss from calorie restriction.</p> <p>Relying on diet alone to lose weight will reduce muscle along with body fat, slowing your metabolism. So it’s essential to make sure you’ve incorporated sufficient and appropriate exercise into your weight-loss plan to hold onto your muscle mass stores.</p> <p>But you don’t need to hit the gym. Exercises using body weight – such as push-ups, pull-ups, planks and air squats – are just as effective as lifting weights and using strength-building equipment.</p> <p>Encouragingly, moderate-volume resistance training (three sets of ten repetitions for eight exercises) <a href="https://onlinelibrary.wiley.com/doi/full/10.1111/sms.14237">can be as effective</a> as high-volume training (five sets of ten repetitions for eight exercises) for maintaining muscle when you’re following a calorie-restricted diet.</p> <h2>2. Eat more protein</h2> <p>Foods high in protein play an essential role in building and maintaining muscle mass, but <a href="https://europepmc.org/article/MED/19927027">research</a> also shows these foods help prevent muscle loss when you’re following a calorie-restricted diet.</p> <p>But this doesn’t mean <em>just</em> eating foods with protein. Meals need to be balanced and include a source of protein, wholegrain carb and healthy fat to meet our dietary needs. For example, eggs on wholegrain toast with avocado.</p> <h2>3. Slow your weight loss plan down</h2> <p>When we change our diet to lose weight, we take our body out of its comfort zone and trigger its survival response. It then counteracts weight loss, triggering <a href="https://pubmed.ncbi.nlm.nih.gov/25896063/">several physiological responses</a> to defend our body weight and “survive” starvation.</p> <p>Our body’s survival mechanisms want us to regain lost weight to ensure we survive the next period of famine (dieting). <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5764193/">Research</a> shows that more than half of the weight lost by participants is regained within two years, and more than 80% of lost weight is regained within five years.</p> <p>However, a slow and steady, stepped approach to weight loss, prevents our bodies <a href="https://pubmed.ncbi.nlm.nih.gov/38193357/">from activating defence mechanisms</a> to defend our weight when we try to lose weight.</p> <p>Ultimately, losing weight long-term comes down to making gradual changes to your lifestyle to ensure you form habits that last a lifetime.</p> <hr /> <p><em>At the Boden Group, Charles Perkins Centre, we are studying the science of obesity and running clinical trials for weight loss. You can <a href="https://redcap.sydney.edu.au/surveys/?s=RKTXPPPHKY">register here</a> to express your interest.</em><!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/223865/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><a href="https://theconversation.com/profiles/nick-fuller-219993"><em>Nick Fuller</em></a><em>, Charles Perkins Centre Research Program Leader, <a href="https://theconversation.com/institutions/university-of-sydney-841">University of Sydney</a></em></p> <p><em>Image credits: Shutterstock </em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/3-signs-your-diet-is-causing-too-much-muscle-loss-and-what-to-do-about-it-223865">original article</a>.</em></p> </div>

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Today Show entertainment reporter's cause of death revealed

<p>Beloved <em>Today</em> show and KTLA entertainment reporter Sam Rubin's cause of death has been revealed, two months after his sudden <a href="https://oversixty.com.au/health/caring/legendary-today-show-reporter-dies-unexpectedly" target="_blank" rel="noopener">passing</a>. </p> <p>The 64-year-old died in May with a new report from medical officials finding he died from a heart attack after he suffered a massive blockage to his coronary artery. </p> <p>Just hours after hosting his Hollywood news segment in the morning of May 10th, Rubin was rushed to home after collapsing in his Los Angeles home. </p> <p>The multi-Emmy Award winner was a renowned figure in the news industry on the West Coast, and had covered entertainment, movies and TV for KTLA since 1991.</p> <p>He was also well-known in Australia as a contributor to Channel Nine's <em>Today</em> show and <em>Today Extra</em>, and in the UK where he appeared on <em>This Morning</em>.</p> <p>Following his death, Karl Stefanovic paid tribute to his colleague on Instagram, saying he "adored every second with Sam on air and off over the past two decades".</p> <p>"His spirit. His laugh. His warm caring nature. He was a beautiful man. What a loss. All love to his family, and to his TV family at KTLA5 News."</p> <p><em>Today Extra</em> host David Campbell also paid tribute to Rubin, calling him a "Hollywood great".</p> <p>"He had an encyclopaedic knowledge of the industry," Campbell posted on social media.</p> <p>"For years we would cross to him and gossip and laugh," he said.</p> <p>"He would visit us Down Under, and whenever you were in LA you had to catch up. His loss is profound. My love and condolences to his family whom he adored."</p> <p style="font-size: 16px; box-sizing: border-box; margin-top: 0px; margin-bottom: 1rem; caret-color: #212529; color: #212529; font-family: -apple-system, BlinkMacSystemFont, 'Segoe UI', Roboto, 'Helvetica Neue', Arial, sans-serif, 'Apple Color Emoji', 'Segoe UI Emoji', 'Segoe UI Symbol', 'Noto Color Emoji';">"Also his KTLA team who have lost a brother. We will cross back to you some other time Sam."</p> <p><em>Image credits: Ken McKay/ITV/Shutterstock Editorial </em></p>

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Take my (bad) breath away – causes of halitosis and how to check whether you have it

<div class="theconversation-article-body"><em><a href="https://theconversation.com/profiles/dan-baumgardt-1451396">Dan Baumgardt</a>, <a href="https://theconversation.com/institutions/university-of-bristol-1211">University of Bristol</a></em></p> <p>In Greek mythology, the many-headed beast <a href="https://mythopedia.com/topics/hydra">Hydra</a> had such severe <a href="https://patient.info/oral-dental-care/bad-breath-halitosis">halitosis</a> that the stench of its breath was deadly to anyone who smelled it. Thankfully, our morning breath might not be that pungent – although eating <a href="https://www.healthline.com/health/get-rid-of-garlic-onion-breath">onions or garlic</a> can put some people in competition with the Hydra.</p> <p>Halitosis has many causes (aside from poor oral hygiene) and can indicate problems with the gut, the sinuses and even the bloodstream. In fact, breath samples can even be tested to make formal diagnoses of health conditions.</p> <p>One condition that can affect the smell of breath is <a href="https://www.diabetes.org.uk/">diabetes mellitus</a>. This is a metabolic disorder where sugar (glucose) is unable to access the body’s cells where it is needed to provide energy, and so rises in the bloodstream.</p> <p>In some instances, such as insufficient insulin dosing, or infection, the body’s response is to break down fats into compounds called ketones to act as a rapid form of fuel. This serious condition is called <a href="https://www.nhs.uk/conditions/diabetic-ketoacidosis/">diabetic ketoacidosis</a>.</p> <p>Ketones have a distinctive scent. <a href="https://www.gov.uk/government/publications/acetone-properties-and-incident-management/acetone-general-information">Acetone</a>, which is also an ingredient in some nail varnish removers, is one of these ketones and has the smell of pear drops. When ketones build up in the bloodstream they easily <a href="https://iopscience.iop.org/article/10.1088/0967-3334/32/8/N01/pdf">diffuse into the breath</a>, giving it a <a href="https://www.medicalnewstoday.com/articles/319683">fruity odour</a>.</p> <p>It’s not just diabetes that can trigger ketone production. <a href="https://pubmed.ncbi.nlm.nih.gov/36033148/">Some diets</a> are based on generating ketones from the breakdown of fats to promote weight loss. These methods, such as the <a href="https://www.healthline.com/nutrition/atkins-diet-101">Atkins diet</a>, force the body to convert fat into energy by restricting carbohydrates.</p> <figure><iframe src="https://www.youtube.com/embed/5U8IDO1fHlU?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <p>Other diets based on the same principles include the <a href="https://patient.info/healthy-living/weight-loss-weight-reduction/52-diet">5:2</a> intermittent fasting diet. On this diet, followers restrict food intake on two days of the week to significantly reduce calorie consumption – and make the body produce ketones.</p> <p>These diets may help weight loss, but the side-effects can be grim. One of the most notorious side-effects is foul breath, although there are also anecdotal reports of <a href="https://www.forbes.com/sites/brucelee/2019/03/03/is-keto-crotch-really-a-side-effect-of-the-keto-diet/">“keto crotch”</a> where some followers of keto diets complain of strong genital odour.</p> <h2>Bacteria and breath</h2> <p>Another cause of bad breath is an <a href="https://iopscience.iop.org/article/10.1088/1752-7155/4/1/017003/pdf">overgrowth of bacteria</a> that produce foul smells. There are plenty of nooks and crannies in the mouth for bacteria to hide, grow and fester, especially the hard-to-clean areas – in between the teeth, and in and around the gums and tongue – or out-of-reach places, such as right at the back of the mouth and the throat.</p> <p>The throat acts as a passage for food, fluids and air. Some patients can develop a condition called <a href="https://www.entuk.org/patients/conditions/49/pharyngeal_pouch_surgery_new">pharyngeal pouch</a>. This is where a pocket forms at the back of the pharynx (the medical name for the throat) in which food and fluids can accumulate, ferment and give breath a pungent odour.</p> <p>Bacteria can also trigger infections in the mouth, like tonsillitis and tooth abscesses where tissues become inflamed, or develop purulence (production of pus). Pus is a collection of different dead cells, including bacteria, and it too can give off a putrid smell.</p> <p>Also, <a href="https://pubmed.ncbi.nlm.nih.gov/25234037/">sinusitis</a> – which is an infection of the air-filled cavities in the skull – can drip foul-smelling infected secretions into the throat, causing bad breath.</p> <h2>Breath tests</h2> <p>Doctors can test breath for bacteria to diagnose some health conditions. For example, <em><a href="https://pubmed.ncbi.nlm.nih.gov/28891138/">Helicobacter pylori</a></em>, bacteria that can irritate the gut and lead to the development of potentially dangerous ulcers, turns the compound urea into carbon dioxide. To test for <em>H pylori</em>, a <a href="https://www.nhs.uk/conditions/stomach-ulcer/diagnosis/">diagnostic breath test</a> is performed before and after dosing a patient with urea. If the patient exhales increased levels of carbon dioxide after being dosed with urea, then the test is positive.</p> <p>Breath can also be tested for an overgrowth of bacteria in the <a href="https://www.healthline.com/health/sibo">small intestine</a> (Sibo), which can lead to symptoms like abdominal pain and bloating. Sibo produces gases like hydrogen and methane that can also be detected with a breath test.</p> <p>If you’re worried about pongy breath and don’t have any medical issues, then you can <a href="https://www.healthline.com/health/dental-and-oral-health/how-to-smell-your-own-breath">test your own breath</a>. The age-old method is to lick the back of your wrist, let it dry and then have a sniff. You can also do the same with a tongue scraper, dental floss or a sample of breath exhaled into a cupped hand.</p> <figure><iframe src="https://www.youtube.com/embed/ak5UEM8FK2s?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <p>Often, we can become used to the smell of our own breath. We might only notice when it becomes really bad, or when there are other symptoms, like a foul taste in the mouth. Or when someone plucks up the courage to finally tell you that you have a case of the breath pongs.</p> <p>Suppose someone has broken the news – what do you do now? <a href="https://www.nhs.uk/conditions/bad-breath/">Simple measures can work well</a>, including regular fluid intake – <a href="https://www.dentalhealth.org/bad-breath">dry mouth</a> can lead to bad breath so make sure you’re drinking enough water – and good oral hygiene. This involves brushing the teeth, tongue and flossing between your teeth to eliminate any bacterial hot spots, as well as regular checkups at the dentist.</p> <p>Mouthwash can be an effective temporary solution but there’s evidence that a <a href="https://theconversation.com/eating-leafy-greens-could-be-better-for-oral-health-than-using-mouthwash-221181#:%7E:text=But%20research%20has%20indicated%20that,alternative%20for%20treating%20oral%20disease.">diet rich in leafy greens</a> might be even better at countering bad breath.</p> <p>Smoking is another potential underlying <a href="https://www.healthline.com/health/how-to-get-rid-of-cigarette-breath#1-brush-teeth">cause of halitosis</a>. So if you want sweeter breath, pack in the cigarettes – yet another good reason to give up.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/231858/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em><a href="https://theconversation.com/profiles/dan-baumgardt-1451396">Dan Baumgardt</a>, Senior Lecturer, School of Physiology, Pharmacology and Neuroscience, <a href="https://theconversation.com/institutions/university-of-bristol-1211">University of Bristol</a></em></p> <p><em>Image credits: Shutterstock </em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/take-my-bad-breath-away-causes-of-halitosis-and-how-to-check-whether-you-have-it-231858">original article</a>.</em></p> </div>

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