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Incredible new pacemaker tinier than a grain of rice

Engineers from Northwestern University in the US have developed a groundbreaking pacemaker so tiny that it can fit inside the tip of a syringe – is smaller than a grain of rice – and be non-invasively injected into the body. This innovation could revolutionise cardiac care by offering a minimally invasive alternative to traditional pacemakers. "We have developed what is, to our knowledge, the world's smallest pacemaker," said bioelectronics pioneer John A Rogers, who led the development. The small, wireless device is biocompatible and designed to be gradually broken down and absorbed by the body, reducing the need for surgical extraction. Rogers and his colleagues tested the effectiveness of their tiny, temporary pacemaker in human heart tissue and animal models. Measuring just 1.8mm by 3.5mm by 1mm, the device is smaller than any previously reported pacemaker. It is paired with a soft, flexible, wireless wearable device mounted on a patient’s chest, which controls the pacing. When an irregular heartbeat is detected, the wearable device emits a light pulse that penetrates through the skin, breastbone and muscles to activate the pacemaker and regulate heart rhythm. While designed to work for hearts of all sizes, the pacemaker is particularly well-suited for newborns with congenital heart defects. Northwestern experimental cardiologist Igor Efimov, who co-led the study, said this device could play a critical role in the effective treatment of infants. "Our major motivation was children," Efimov explained. "About 1 percent of children are born with congenital heart defects, regardless of whether they live in a low-resource or high-resource country. The good news is that these children only need temporary pacing after surgery. In about seven days or so, most patients’ hearts will self-repair. But those seven days are absolutely critical." Researchers believe this pacemaker could provide a safer alternative to traditional pacemakers for temporary pacing in patients with bradycardia, a condition characterised by a resting heart rate below 60 beats per minute. Additionally, they suggest that the technology’s versatility could extend to broader medical applications in bioelectronic medicine, such as aiding nerve and bone healing and blocking pain. This revolutionary development represents a significant step forward in cardiac treatment, potentially improving outcomes for both infants and adults requiring temporary heart pacing. Images: Northwestern University

Body

Do stem cell injections for knee osteoarthritis actually work?

<div class="theconversation-article-body">More than 500 million people around the world <a href="https://www.thelancet.com/journals/lanrhe/article/PIIS2665-9913(23)00163-7/fulltext">live with osteoarthritis</a>. The knee is affected more often than any other joint, with symptoms (such as pain, stiffness and reduced movement) affecting work, sleep, sport and <a href="https://www.aihw.gov.au/reports/chronic-musculoskeletal-conditions/osteoarthritis">daily activities</a>. Knee osteoarthritis is often thought of as thinning of the protective layer of cartilage within the joint. But we now understand it affects all the structures of the joint, including the bones, muscles and nerve endings. While there are things that <a href="https://www.safetyandquality.gov.au/standards/clinical-care-standards/osteoarthritis-knee-clinical-care-standard/information-consumers-osteoarthritis-knee-clinical-care-standard">can be done to manage</a> the symptoms of knee osteoarthritis, there is no cure, and many people experience persistent pain. As a result, an opportunity exists for <a href="https://www.rheuma.com.au/stem-cell-therapy-good-bad-ugly/174">as yet unproven treatments</a> to enter the market, often before regulatory safeguards can be put in place. Stem cell injections are one such treatment. A <a href="https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD013342.pub2">new review</a> my colleagues and I published this week finds that evidence of their benefits and harms remains elusive. <h2>Stem cell treatments</h2> Stem cells are already established as treatments for some diseases – <a href="https://www.lymphoma.org.au/lymphoma/treatments/stem-cell-transplants/autologous-stem-cell-transplant/">mostly disorders of the blood</a>, bone marrow or immune system – which has led to suggestions they could be used for a much wider array of conditions. Stem cells have been touted as promising treatments for osteoarthritis because they have special properties which allow them to replicate and develop into the mature healthy cells that make up our body’s organs and other tissues, including cartilage. Stem cell treatments for osteoarthritis generally involve <a href="https://www.aboutstemcells.org/treatments">taking a sample of tissue</a> from a site that is rich in stem cells (such as bone marrow or fat), treating it to increase the number of stem cells, then injecting it into the joint. The hope is that if the right type of stem cells can be introduced into an osteoarthritic joint in the right way and at the right time, they may help to repair damaged structures in the joint, or have other effects such as reducing inflammation. But no matter how convincing the theory, we need good evidence for effectiveness and safety before a new therapy is adopted into practice. Stem cell injections have not been approved by Australia’s <a href="https://www.tga.gov.au/news/news/stem-cell-treatments-and-regulation-quick-guide-consumers#:%7E:text=Does%20the%20TGA%20regulate%20stem,does%20not%20regulate%20medical%20practice">Therapeutic Goods Administration</a> for the treatment of osteoarthritis. Nonetheless, some clinics in Australia and around the world still offer them. Because of the regulatory restrictions, we don’t have reliable numbers on how many procedures are being done. They’re not covered by Medicare, so the procedure can cost the consumer thousands of dollars. And, as with any invasive procedure, both the <a href="https://coroners.nsw.gov.au/documents/findings/2016/Findings%20Drysdale.pdf">harvest of stem cells</a> and the joint injection procedure may carry the potential for harm, such as infection. <h2>What we found</h2> Our <a href="https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD013342.pub2">new review</a>, published by the independent, international group the Cochrane Collaboration, looks at all 25 randomised trials of stem cell injections for knee osteoarthritis that have been conducted worldwide to date. Collectively, these studies involved 1,341 participants. We found stem cell injections may slightly improve pain and function compared with a placebo injection, but the size of the improvement may be too small for the patient to notice. The evidence isn’t strong enough to determine whether there is any improvement in quality of life following a stem cell injection, whether cartilage regrows, or to estimate the risk of harm. This means we can’t confidently say yet whether any improvement that might follow a stem cell injection is worth the risk (or the cost). <h2>Hope or hype?</h2> It’s not surprising we invest hope in finding a transformative treatment for such a common and disabling condition. Belief in the benefits of stem cells is widespread – more than <a href="https://www.arthroscopyjournal.org/article/S0749-8063(21)00571-5/abstract">three-quarters of Americans</a> believe stem cells can relieve arthritis pain and more than half believe this treatment to be curative. But what happens if a new treatment is introduced to practice before it has been clearly proven to be safe and effective? The use of an unproven, invasive therapy is not just associated with the risks of the intervention itself. Even if the treatment were harmless, there is the risk of unnecessary cost, inconvenience, and a missed opportunity for the patient to use existing therapies that are known to be effective. What’s more, if we need to play catch-up to try to establish an evidence base for a treatment that’s already in practice, we risk diverting scarce research resources towards a therapy that may not prove to be effective, simply because the genie is out of the bottle. <h2>Working towards a clearer answer</h2> Several more large <a href="https://anzctr.org.au/Trial/Registration/TrialReview.aspx?ACTRN=12620000870954">clinical trials</a> are currently underway, and should increase our understanding of whether stem cell injections are safe and effective for knee osteoarthritis. Our review incorporates “<a href="https://www.cochrane.org/news/cochranes-pioneering-role-living-evidence">living evidence</a>”. This means we will continue to add the results of new trials as soon as they’re published, so the review is always up to date, and offers a comprehensive and trustworthy summary to help people with osteoarthritis and their health-care providers to make informed decisions. In the meantime, there are a number of <a href="https://www.racgp.org.au/clinical-resources/clinical-guidelines/key-racgp-guidelines/view-all-racgp-guidelines/knee-and-hip-osteoarthritis/summary-plain-language">evidence-based treatment options</a>. Non-drug treatments such as physiotherapy, regular exercise, maintaining a healthy weight, and cognitive behavioural therapy can be more effective than you think. Anti-inflammatory and pain medications can also play a supporting role. Importantly, it’s not inevitable that osteoarthritic joints get worse with time. So, even though <a href="https://aoanjrr.sahmri.com/background">joint replacement surgery</a> is often highly effective, it’s the last resort and fortunately, many people never need to take this step.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/253404/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> By <a href="https://theconversation.com/profiles/samuel-whittle-2357927">Samuel Whittle</a>, ANZMUSC Practitioner Fellow, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/people-are-getting-costly-stem-cell-injections-for-knee-osteoarthritis-but-we-dont-know-if-they-work-253404">original article</a>. Image: Shutterstock </div>

Body

New study suggests shingles vaccine may lower dementia risk

A groundbreaking new study suggests that getting vaccinated against shingles could offer protection against dementia, adding to the growing evidence linking viral infections to cognitive decline. Shingles is a painful and debilitating condition that occurs in individuals who have previously had chickenpox. The varicella-zoster virus, responsible for both diseases, can lie dormant for years before reactivating as shingles. The latest research, published recently in Nature, analysed data from over 280,000 older adults in Wales. The findings reveal that individuals who received the original live-virus shingles vaccine were 20% less likely to develop dementia than those who were not vaccinated. The study was made possible by a unique public health policy in Wales, which provided an unusual opportunity to compare similar populations. On September 1, 2013, the vaccine was offered to individuals who were 79 years old on that date but not to those who had turned 80. This created two nearly identical groups, differing only in their eligibility for the vaccine, allowing researchers from Germany and Stanford University to examine its impact on dementia risk. “This study is essentially like a randomised controlled trial, which is the gold standard in research,” said Dr Pascal Geldsetzer, a Stanford University assistant professor of medicine and senior author of the study. Previous research has suggested an association between the newer Shingrix vaccine – which replaced the live-virus version in 2020 – and a lower risk of dementia, particularly in women. However, earlier studies were unable to fully account for differences between vaccinated and unvaccinated individuals, such as overall health status. Dr Allison Aiello, a professor of epidemiology at Columbia University’s Aging Centre, praised the design of the Welsh study. “It’s like having a control group compared to a treated group,” Aiello, who was not involved in the research, said. “The 20% reduction in dementia risk is a pretty strong effect, which aligns with other findings that suggest herpes viruses might influence dementia.” Scientists do not yet fully understand how the varicella-zoster virus increases dementia risk. One theory is that viral reactivation may contribute to the buildup of abnormal proteins associated with Alzheimer’s disease. Another possibility is that shingles reactivation might also trigger herpes simplex virus (HSV), which has previously been linked to dementia. Herpes viruses, including HSV-1 and HSV-2, can linger in the body indefinitely, often reactivating when the immune system weakens with age. These viruses can infiltrate the central nervous system, potentially leading to brain inflammation and neurodegenerative effects. A 2024 study found that among 70-year-olds without dementia at the outset, those diagnosed with herpes simplex were more than twice as likely to develop dementia over 15 years. While the new study offers compelling evidence, it is not a definitive randomised controlled trial, cautioned Dr William Schaffner, an infectious disease expert at Vanderbilt University Medical Centre. “It’s provocative, interesting and exciting,” he said. However, he noted that it would be unethical to randomly assign people to receive or forgo the vaccine. The US FDA may eventually review the accumulating research and consider updating the shingles vaccine label to reflect a possible dementia-protection benefit, Schaffner added. Even if such an update does not occur, doctors may use these findings to encourage more people over 50 to get vaccinated. “There are still many eligible people who haven’t received the vaccine,” Schaffner said. “If they hear that it might help prevent dementia, that could be the push they need.” Image: Supplied

Caring

Do any non-drug treatments help back pain? Here’s what the evidence says

<div class="theconversation-article-body"> Jason, a 42-year-old father of two, has been battling back pain for weeks. Scrolling through his phone, he sees ad after ad promising relief: chiropractic alignments, acupuncture, back braces, vibrating massage guns and herbal patches. His GP told him to “stay active”, but what does that even mean when every movement hurts? Jason wants to avoid strong painkillers and surgery, but with so many options (and opinions), it’s hard to know what works and what’s just marketing hype. If Jason’s experience sounds familiar, you’re not alone. Back pain is one of the most common reasons people visit a doctor. It can be challenging to manage, mainly due to widespread <a href="https://theconversation.com/having-good-posture-doesnt-prevent-back-pain-and-bad-posture-doesnt-cause-it-183732">misunderstandings</a> and the <a href="https://ebm.bmj.com/content/early/2025/03/02/bmjebm-2024-112974">overwhelming number</a> of ineffective and uncertain treatments promoted. We assessed the best available evidence of non-drug and non-surgical treatments to alleviate low back pain. <a href="https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD014691.pub2/full">Our review</a> – published today by the independent, international group the Cochrane Collaboration – includes 31 Cochrane systematic reviews, covering 97,000 people with back pain. It <a href="https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD014691.pub2/full">shows</a> bed rest doesn’t work for back pain. Some of the treatments that do work can depend on how long you’ve been in pain. <h2>Is back pain likely to be serious?</h2> There are different types of low back pain. It can: <ul> <li>be short-lived, lasting less than six weeks (acute back pain)</li> <li>linger for a bit longer, for six to twelve weeks (sub-acute)</li> <li>stick around for months and even years (chronic, defined as more than 12 weeks).</li> </ul> In <a href="https://www.thelancet.com/article/S0140-6736(16)30970-9/abstract">most cases</a> (90-95%), back pain is non-specific and cannot be reliably linked to a specific cause or underlying disease. This includes common structural changes seen in x-rays and MRIs of the spine. For this reason, imaging of the back is <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(09)60172-0/abstract">only</a> recommended in rare situations – typically when there’s a clear suspicion of serious back issues, such as after physical trauma or when there is numbness or loss of sensation in the groin or legs. Many people expect to receive <a href="https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD013815.pub2/full">painkillers</a> for their back pain or even surgery, but these are no longer the front-line treatment options due to limited benefits and the high risk of harm. International <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(18)30489-6/fulltext">clinical guidelines</a> recommend people choose non-drug and non-surgical treatments to relieve their pain, improve function and reduce the distress commonly associated with back pain. So what works for different types of pain? Here’s what <a href="https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD014691.pub2/full">our review found</a> when researchers compared these treatments with standard care (the typical treatment patients usually receive) or no treatment. <h2>What helps for short-term back pain</h2> 1. Stay active – don’t rest in bed If your back pain is new, the best advice is also one of the simplest: keep moving despite the pain. Changing the way you move and use your body to protect it, or resting in bed, can seem like to right way to respond to pain – and may have even been recommended in the past. But we know know this excessive protective behaviour can make it harder to return to meaningful activities. This doesn’t mean pushing through pain or hitting the gym, but instead, trying to maintain your usual routines as much as possible. Evidence suggests that doing so won’t make your pain worse, and may improve it. 2. Multidisciplinary care, if pain lingers For pain lasting six to 12 weeks, multidisciplinary treatment is likely to reduce pain compared to standard care. This involves a coordinated team of doctors, physiotherapists and psychologists working together to address the many factors contributing to your back pain persisting: <ul> <li> neurophysiological influences refer to how your nervous system is currently processing pain. It can make you more sensitive to signals from movements, thoughts, feelings and environment </li> <li> psychological factors include how your thoughts, feelings and behaviours affect your pain system and, ultimately, the experience of pain you have </li> <li> occupational factors include the physical demands of your job and how well you can manage them, as well as aspects like low job satisfaction, all of which can contribute to ongoing pain. </li> </ul> <h2>What works for chronic back pain</h2> Once pain has been around for more than 12 weeks, it can become more difficult to treat. But relief is still possible. Exercise therapy Exercise – especially programs tailored to your needs and preferences – is likely to reduce pain and help you move better. This could include aerobic activity, strength training or Pilates-based movements. It doesn’t seem to matter what type of exercise you do – it matters more that you are consistent and have the right level of supervision, especially early on. Multidisciplinary treatment As with short-term pain, coordinated care involving a mix of physical, occupational and psychological approaches likely works better than usual care alone. Psychological therapies Psychological therapies for chronic pain include approaches to help people change thinking, feelings, behaviours and reactions that might sustain persistent pain. These approaches are likely to reduce pain, though they may not be as effective in improving physical function. Acupuncture Acupuncture probably reduces pain and improves how well you can function compared to placebo or no treatment. While some debate remains about how it works, the evidence suggests potential benefits for some people with chronic back pain. <h2>What doesn’t work or still raises uncertainty?</h2> The review found that many commonly advertised treatments still have uncertain benefits or probably do not benefit people with back pain. Spinal manipulation, for example, has uncertain benefits in acute and chronic back pain, and it likely does not improve how well you function if you have acute back pain. Traction, which involves stretching the spine using weights or pulleys, probably doesn’t help with chronic back pain. Despite its popularity in some circles, there’s little evidence that it works. There isn’t enough reliable data to determine whether advertised treatments – such back braces, vibrating massage guns and herbal patches – are effective. <h2>How can you use the findings?</h2> If you have back pain, start by considering how long you’ve had it. Then explore treatment options that research supports and discuss them with your GP, psychologist or physiotherapist. Your health provider should reassure you about the importance of gradually increasing your activity to resume meaningful work, social and life activities. They should also support you in making informed decisions about which treatments are most appropriate for you at this stage. <img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/253122/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" />By <a href="https://theconversation.com/profiles/rodrigo-rossi-nogueira-rizzo-1544189">Rodrigo Rossi Nogueira Rizzo</a>, Postdoctoral Research Fellow, <a href="https://theconversation.com/institutions/neuroscience-research-australia-976">Neuroscience Research Australia</a> and <a href="https://theconversation.com/profiles/aidan-cashin-2355450">Aidan Cashin</a>, NHMRC Emerging Leadership Fellow, <a href="https://theconversation.com/institutions/neuroscience-research-australia-976">Neuroscience Research Australia</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/do-any-non-drug-treatments-help-back-pain-heres-what-the-evidence-says-253122">original article</a>. Image: Shutterstock </div>

Body

King Charles admitted to hospital

Reports from Buckingham palace have revealed that King Charles has been forced to cancel a series of public engagements after being admitted to hospital due to side effects from his ongoing cancer treatment. The Palace released a statement on Thursday evening confirming that the 72-year-old monarch had “experienced temporary side effects that required a short period of observation in hospital” following his scheduled and ongoing medical treatment for cancer. The King has since returned to Clarence House, where he resides with Queen Camilla. “As a precautionary measure, acting on medical advice, tomorrow’s diary program will also be rescheduled,” the Palace added. Although the Palace did not specify the exact nature of the side effects, sources suggest such occurrences are not uncommon among cancer patients. A royal insider described the medical incident to The Daily Mail as a “most minor bump in a road that’s very much heading in the right direction”. However, in order to “protect and prioritise [his] continued very positive recovery”, King Charles has regretfully cancelled his planned engagements in Birmingham on Friday. The King was diagnosed with an undisclosed form of cancer in February last year and has been receiving outpatient treatment since then. Initially, he had to withdraw from public duties for several weeks but resumed engagements in April. Since then, he has travelled internationally – including a visit to Australia last October – and hosted numerous state visits while continuing his recovery. Last week, King Charles visited Ulster University’s Pharmacy and Pharmacology department in Northern Ireland to learn about their groundbreaking cancer research. During his visit, he shared words of encouragement with fellow cancer patients, quoting Winston Churchill’s famous phrase: “Keep buggering on.” Regarding the side effects of treatment, he remarked, “You just have to push on, don’t you?” While the specific type of cancer affecting the King has not been disclosed, reports suggest that it was caught at a very early stage. Royal sources remain optimistic about his recovery, and say his treatment is progressing positively. Image: Department of the Prime Minister and Cabinet/ Millie Pilkington

Caring

"Patrons yelled to stop": Comedy Festival rocked by death on opening night

Melbourne’s comedy scene was rocked by tragedy on the opening night of this year’s International Comedy Festival, as an attendee suffered a fatal medical episode mid-show, leading to the abrupt cancellation of the event. The distressing incident occurred during comedian Michael Hing’s performance at the Palais Theatre, where a man on the upper level of the venue suffered a medical emergency. Attendees quickly responded, providing CPR in the dark while Hing remained unaware of the unfolding crisis. “[It] would have been at least 15 mins worth before the show stopping, and [it] only stopped as patrons yelled to stop performing,” one audience member told The Daily Mail. Another witness expressed dismay over the handling of the situation, stating, “I can’t believe they [the organisers] didn’t communicate with backstage to halt the show, and that it took the public to yell out to the comedian to stop for something to happen.” The Melbourne International Comedy Festival addressed the incident in a statement posted on Instagram, confirming the show’s cancellation. “Melbourne International Comedy Festival regrets to confirm that there was a medical emergency while the Opening Night Allstars Supershow was taking place this evening at the Palais Theatre,” a festival spokesman said. “The show was stopped and cancelled. The Festival will be in touch with all ticket holders tomorrow.” Victoria Police later confirmed the man’s death. “Police will prepare a report for the Coroner following the death of a man in St Kilda on Wednesday, 26 March,” a spokesman said. “Emergency services were called to a theatre on Lower Esplanade about 9pm. A man, who is yet to be formally identified, died at the scene.” Health worker Andrea Bortoli, who was sitting in the upper level’s front row, described the distressing scene as people began leaving and it “became quite obvious that someone was getting CPR. It was just obviously really distressing, so we wanted to leave,” she told The Age. Eventually, the theatre lights were turned on, and patrons were informed that the show had been cancelled. The Allstars Gala is one of the most anticipated nights of the festival, bringing together some of the biggest names in comedy. This year’s event was hosted by Hing. As news of the tragedy spread, comedian Dave Hughes expressed his sympathies on social media, writing, “All performers thoughts are with the family affected”. Images: Palais Theatre / MichaelHing.com

Caring

"Game changer": Aussie women welcome new early-detection cancer test

Australian women will soon have access to a revolutionary blood test designed to aid in the early detection of breast cancer. The innovative test, known as BREASTEST plus™, was developed by BCAL Diagnostics and is set to complement traditional breast cancer screening methods such as mammograms and ultrasounds. BCAL Diagnostics chair Jayne Shaw <a href="https://7news.com.au/news/public-health/australian-first-blood-test-to-help-early-detection-of-breast-cancer-comes-to-sydney-before-nationwide-rollout-c-18153750" target="_blank" rel="noopener">revealed to 7NEWS</a> that the development of the test had taken 15 years, describing it as a "game changer" for women worldwide. “The breast test is a game changer for all women everywhere because it’s a blood test to detect breast cancer alongside other diagnostic tools like ultrasound and a mammogram,” Shaw said. She pointed out that a similar blood test for prostate cancer, which has been available since 1989, has significantly improved clinical outcomes for men. “Early diagnosis for all cancers will lead to improved survivability rates, and it was only inevitable that a blood test would be developed to diagnose breast cancer earlier,” she added. One of the major advantages of the new blood test is its ability to detect breast cancer in women with high breast density. Around 40-50% of Australian women undergoing screening have high breast density, which can obscure abnormalities on mammograms and make diagnosis more difficult. With the ability to identify breast cancer markers, the blood test will provide a valuable additional tool for detecting cancers that might otherwise go unnoticed. Breast Cancer Network Australia director Vicki Durston welcomed the new test, especially for its potential to improve the reporting of high breast density cases, where cancers are often missed. “Breast Cancer Network Australia has long been calling for the uplift in the breast density statement nationally to see standardised reporting across the country,” Durston said. She also noted that while high breast density is just one of many risk factors, the new test represents an innovative step forward in improving early detection and treatment. Breast cancer remains the most commonly diagnosed cancer among Australian women, accounting for approximately 28% of all new cancer cases. In 2024 alone, around 21,194 people were diagnosed with breast cancer in Australia, including 20,973 women and 221 men. However, survival rates have improved dramatically, with Australia’s peak breast cancer body reporting that the five-year survival rate has risen from 78% in 1994 to 92% in 2020, with many people living long and healthy lives beyond this period. While the test currently comes at an out-of-pocket cost, there is hope that the Australian government will eventually provide subsidies under Medicare to make it more accessible. The first tests will be available at the <a href="https://www.sydneybreastclinic.com.au/" target="_blank" rel="noopener">Sydney Breast Clinic</a> starting Thursday, followed by a rollout in Melbourne, with plans to expand nationwide by the end of the year. Images: Shutterstock / 7NEWS

Body

How long will you live? New evidence says it’s much more about your choices than your genes

<div class="theconversation-article-body"> One of the most enduring questions humans have is how long we’re going to live. With this comes the question of how much of our lifespan is shaped by our environment and choices, and how much is predetermined by our genes. A study recently published in the prestigious journal <a href="https://www.nature.com/articles/s41591-024-03483-9">Nature Medicine</a> has attempted for the first time to quantify the relative contributions of our environment and lifestyle versus our genetics in how we age and how long we live. The findings were striking, suggesting our environment and lifestyle play a much greater role than our genes in determining our longevity. <h2>What the researchers did</h2> This study used data from the <a href="https://www.ukbiobank.ac.uk/">UK Biobank</a>, a large database in the United Kingdom that contains in-depth health and lifestyle data from roughly 500,000 people. The data available include genetic information, medical records, imaging and information about lifestyle. A separate part of the study used data from a subset of more than 45,000 participants whose blood samples underwent something called “<a href="https://www.nature.com/articles/s41576-022-00511-7">proteomic profiling</a>”. Proteomic profiling is a relatively new technique that looks at how proteins in the body change over time to identify a person’s age at a molecular level. By using this method researchers were able to estimate how quickly an individual’s body was actually ageing. This is called their biological age, as opposed to their chronological age (or years lived). The researchers assessed 164 environmental exposures as well as participants’ genetic markers for disease. Environmental exposures included lifestyle choices (for example, smoking, physical activity), social factors (for example, living conditions, household income, employment status) and early life factors, such as body weight in childhood. They then looked for associations between genetics and environment and 22 major age-related diseases (such as coronary artery disease and type 2 diabetes), mortality and biological ageing (as determined by the proteomic profiling). These analyses allowed the researchers to estimate the relative contributions of environmental factors and genetics to ageing and dying prematurely. <h2>What did they find?</h2> When it came to disease-related mortality, as we would expect, age and sex explained a significant amount (about half) of the variation in how long people lived. The key finding, however, was environmental factors collectively accounted for around 17% of the variation in lifespan, while genetic factors contributed less than 2%. This finding comes down very clearly on the nurture side in the “nature versus nurture” debate. It suggests environmental factors influence health and longevity to a far greater extent than genetics. Not unexpectedly, the study showed a different mix of environmental and genetic influences for different diseases. Environmental factors had the greatest impact on lung, heart and liver disease, while genetics played the biggest role in determining a person’s risk of breast, ovarian and prostate cancers, and dementia. The environmental factors that had the most influence on earlier death and biological ageing included smoking, socioeconomic status, physical activity levels and living conditions. Interestingly, being taller at age ten was found to be associated with a shorter lifespan. Although this may seem surprising, and the reasons are not entirely clear, this aligns with <a href="https://www.sciencedaily.com/releases/2014/05/140509110756.htm">previous research</a> finding taller people are more likely to die earlier. Carrying more weight at age ten and maternal smoking (if your mother smoked in late pregnancy or when you were a newborn) were also found to shorten lifespan. Probably the most surprising finding in this study was a lack of association between diet and markers of biological ageing, as determined by the proteomic profiling. This flies in the face of the extensive body of evidence showing the crucial role of <a href="https://www.nature.com/articles/s43016-023-00868-w">dietary patterns</a> in chronic disease risk and longevity. But there are a number of plausible explanations for this. The first could be a lack of statistical power in the part of the study looking at biological ageing. That is, the number of people studied may have been too small to allow the researchers to see the true impact of diet on ageing. Second, the dietary data in this study, which was self-reported and only measured at one time point, is likely to have been of relatively poor quality, limiting the researchers’ ability to see associations. And third, as the relationship between diet and longevity is likely to be complex, disentangling dietary effects from other lifestyle factors may be difficult. So despite this finding, it’s still safe to say the food we eat is one of the most important pillars of health and longevity. <h2>What other limitations do we need to consider?</h2> Key exposures (such as diet) in this study were only measured at a single point in time, and not tracked over time, introducing potential errors into the results. Also, as this was an observational study, we can’t assume associations found represent causal relationships. For example, just because living with a partner correlated with a longer lifespan, it doesn’t mean this caused a person to live longer. There may be other factors which explain this association. Finally, it’s possible this study may have underestimated the role of genetics in longevity. It’s important to recognise genetics and environment don’t operate in isolation. Rather, health outcomes are shaped by their interplay, and this study may not have fully captured the complexity of these interactions. <h2>The future is (largely) in your hands</h2> It’s worth noting there were a number of factors such as household income, home ownership and employment status associated with diseases of ageing in this study that are not necessarily within a person’s control. This highlights the crucial role of addressing the social determinants of health to ensure everyone has the best possible chance of living a long and healthy life. At the same time, the results offer an empowering message that longevity is largely shaped by the choices we make. This is great news, unless you have good genes and were hoping they would do the heavy lifting. Ultimately, the results of this study reinforce the notion that while we may inherit certain genetic risks, how we eat, move and engage with the world seems to be more important in determining how healthy we are and how long we live.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/251054/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/hassan-vally-202904">Hassan Vally</a>, Associate Professor, Epidemiology, <a href="https://theconversation.com/institutions/deakin-university-757">Deakin University</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/how-long-will-you-live-new-evidence-says-its-much-more-about-your-choices-than-your-genes-251054">original article</a>. Image: Shutterstock </div>

Caring

Shock after baseball champion's 14-year-old son dies while on family holiday

The baseball world is grieving alongside former New York Yankees outfielder Brett Gardner and his family after the tragic and unexpected passing of his 14-year-old son, Miller. Miller Gardner died in his sleep on Friday, March 21, while on holiday with his family. According to a statement released by Brett and his wife, Jessica, through the Yankees organisation, Miller had fallen ill during the trip along with several relatives. The family did not disclose further details about the nature of the illness or their location at the time. “We have so many questions and so few answers at this point,” the grieving parents shared. “Miller was a beloved son and brother, and we cannot yet comprehend our life without his infectious smile.” Brett and Jessica, who also share an older son, Hunter, requested privacy as they mourn and seek healing. They described Miller as a vibrant and adventurous young boy who had a passion for football, baseball, golf, hunting and fishing. “He lived life to the fullest every single day,” they wrote, adding their gratitude to those who have offered condolences and support. They also extended their thoughts to other families who have suffered similar losses, saying, “We share their grief.” The Yankees, where Brett Gardner spent his entire 14-season Major League Baseball career, also released a heartfelt statement. They described Miller as having “a spark in his eyes, an outgoing and feisty personality, and a warm and loving nature.” “Words feel insignificant and insufficient in trying to describe such an unimaginable loss,” the team shared on social media. “It wasn’t just Brett who literally grew up in this organisation for more than 17 years – so did his wife, Jessica, and their two boys, Hunter and Miller.” <blockquote class="twitter-tweet"> Statement from Brett and Jessica Gardner: With heavy hearts we are saddened to announce the passing of our youngest son, Miller. He was 14 years old and has left us far too soon after falling ill along with several other family members while on vacation. We have so many… <a href="https://t.co/lBCBVmKGUe">pic.twitter.com/lBCBVmKGUe</a> — New York Yankees (@Yankees) <a href="https://twitter.com/Yankees/status/1903854341737386272?ref_src=twsrc%5Etfw">March 23, 2025</a></blockquote> Brett Gardner, now 41, was a key part of the Yankees’ 2009 World Series-winning team and became a fan favourite for his grit and dedication to the game. Messages of love and prayers continue to pour in, a testament to the impact Miller had on those who knew him and to the deep respect held for Brett and his family. Images: X (formerly Twitter)

Caring

More young people are caring for a loved one with dementia. It takes a unique toll

<div class="theconversation-article-body"> Dementia is a growing health problem, affecting more than <a href="https://www.who.int/news-room/fact-sheets/detail/dementia">55 million people</a> around the world. In Australia, an estimated <a href="https://www.dementia.org.au/about-dementia/dementia-facts-and-figures">433,300 people</a> are living with dementia. This figure is projected to rise to 812,500 by 2054. Dementia <a href="https://www.dementia.org.au/about-dementia">refers to brain disorders</a> that are not a normal part of ageing. These disorders, including Alzheimer’s disease, cause a decline in cognitive function and changes in mood, memory, thinking and behaviour. Ultimately they affect a person’s ability to carry out everyday tasks. In Australia, around <a href="https://www.sydney.edu.au/news-opinion/news/2020/10/06/research-backs-home-based-program-for-people-living-with-dementi.html">75% of people with dementia</a> live at home. While dementia care at home has traditionally been associated with older spouses or middle-aged children, it seems an increasing number of young adults in their 20s and 30s, and <a href="https://www.ncb.org.uk/sites/default/files/uploads/files/young_people_caring_for_adults_with_dementia.pdf">even teenagers</a>, are stepping into this role to care for grandparents, parents or other loved ones. In Australia, 3 million people (11.9% of the population) are carers. This includes <a href="https://www.abs.gov.au/statistics/health/disability/disability-ageing-and-carers-australia-summary-findings/latest-release">391,300 under 25</a> – a sharp rise from 235,300 in 2018. How many young carers are specifically caring for a loved one with dementia is unclear, and something we need more data on. Young dementia carers remain largely invisible, with minimal recognition or support. <h2>Unique challenges and the burden of responsibility</h2> Unlike older carers, who may have more financial stability and free time, young carers often must balance caregiving with university, early-career pressures, and personal development, including maintaining social relationships, pursuing hobbies, and prioritising mental welling. In Australia, where <a href="https://aifs.gov.au/media/more-young-adults-living-parents">51% of men and 43% of women</a> aged 20–24 still live with their parents, many young carers will have limited experience in managing a household independently. They’re often thrust into <a href="https://www.ncb.org.uk/sites/default/files/uploads/files/young_people_caring_for_adults_with_dementia.pdf">complex responsibilities</a> such as cooking, housework, managing the family budget, coordinating medical appointments and administering medications. Beyond that, they may need to provide physical care such as lifting or helping their loved one move around, and personal care such as dressing, washing, and helping with toileting. All this can leave young carers <a href="https://www.tandfonline.com/doi/full/10.2147/PROM.S499063">feeling unprepared</a>, overwhelmed and isolated. While general support groups exist for dementia carers and young carers more broadly, few cater specifically to <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC9819145/">young adults caring for someone with dementia</a>. This lack of targeted support is likely to heighten <a href="https://www.tandfonline.com/doi/pdf/10.1080/13676261.2024.2390899?casa_token=6Q73sIFsfssAAAAA:1rQ7NyiiHkwTwIk4mkk6d2r5tiUxeXJDCKTfIohKmHzCue9xvuj5r4e8-fowJGQeIYKe6afuO9FT">feelings of isolation</a>, as the young person’s friends struggle to relate to the emotional and practical burdens <a href="https://link.springer.com/content/pdf/10.1186/s12877-020-01976-z.pdf">young carers face</a>. The demanding nature of caregiving, combined with the difficulty of sharing these experiences with peers, means young dementia carers can become <a href="https://journals.sagepub.com/doi/epub/10.1177/14713012211023653?src=getftr&utm_source=tfo&getft_integrator=tfo">disconnected socially</a>. <h2>The psychological toll</h2> These challenges take a profound <a href="https://www.mentalhealth.org.uk/explore-mental-health/statistics/carers-statistics">psychological toll</a> on young carers. Research shows young carers are <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC9263065/">35% more likely</a> to report mental health issues than their non-caregiving peers. These can include <a href="https://journals.sagepub.com/doi/pdf/10.1177/1471301220980243">depression</a>, anxiety and burnout. Again, we don’t have data on mental health outcomes among young dementia carers specifically. But <a href="https://www.aihw.gov.au/reports/dementia/dementia-in-aus/contents/carers-and-care-needs-of-people-with-dementia/impact-of-the-caring-role-on-carers">in Australia</a>, 75% of dementia carers reported being affected physically or emotionally by their caring role. Some 41% felt weary or lacked energy, and 31% felt worried or depressed. Also, there are negative stereotypes about ageing – that people turn forgetful, frail, and need constant care. For young carers whose loved ones have dementia, these stereotypes <a href="https://journals.sagepub.com/doi/pdf/10.1111/j.1467-8721.2009.01662.x?casa_token=w0MW7GGY80gAAAAA:szB0XyXKPG_XMqAVyAHLm0Tye8yH3TYXK9X5cFCSIRkorhsNHx4Wgurmy_oAuZKsTsskS6kBw9Vr">can be reinforced</a> by their experience. This could shape young carers’ perceptions of <a href="https://www.tandfonline.com/doi/full/10.1080/13607863.2025.2464705?utm">their own future health</a> and wellbeing and increase anxiety about ageing. Caregiving may also affect <a href="https://www.apa.org/pi/about/publications/caregivers/faq/health-effects">physical health</a>. Research suggests carers often sacrifice <a href="https://onlinelibrary.wiley.com/doi/pdf/10.1111/psyg.12354">healthy habits</a> such as exercise and a balanced diet. What’s more, <a href="https://onlinelibrary.wiley.com/doi/pdf/10.1111/psyg.12354">carers report symptoms</a> including poor sleep, fatigue, headaches and back pain due to the physical demands of caregiving. <h2>Caring for a parent – a role reversal</h2> This emotional burden is particularly acute for those caring for a parent. These young carers are likely to experience the progressive loss of parental support, while simultaneously assuming the <a href="https://journals.sagepub.com/doi/pdf/10.1177/1471301220988231">demanding role of caregiver</a>. A significant portion of young dementia carers support parents with <a href="https://www.aihw.gov.au/reports/dementia/dementia-in-aus/contents/dementia-in-priority-groups/population-health-impacts-of-younger-onset-dementi">young-onset dementia</a>, a form of dementia diagnosed before age 65. These <a href="https://journals.sagepub.com/doi/pdf/10.1177/1471301220988231">young carers</a> face the shock of a diagnosis that defies typical expectations of ageing. The burden may be compounded by fears of <a href="https://journals.sagepub.com/doi/10.1177/0038026119874280?icid=int.sj-abstract.citing-articles.9">genetic inheritance</a>. Young onset dementia often has a <a href="https://www.nia.nih.gov/health/alzheimers-causes-and-risk-factors/alzheimers-disease-genetics-fact-sheet">hereditary component</a>. This means young carers may have a higher risk of developing the condition themselves – a concern spousal carers don’t have. This fear can fuel health anxiety, alter life planning, and create a pervasive <a href="https://pubmed.ncbi.nlm.nih.gov/26560507/">sense of vulnerability</a>. <h2>How we can better support young dementia carers</h2> Despite their growing numbers, young dementia carers <a href="https://www.tandfonline.com/doi/full/10.2147/JMDH.S464195">remain largely overlooked</a> in research, policy and support services. This is partly due to the challenges in engaging this demographic in research, as these young people juggle busy lives <a href="https://onlinelibrary.wiley.com/doi/pdf/10.1002/gps.4502?casa_token=5-Ih4KcUejwAAAAA%3AOvuh8ghI_ednIzRZmHdUl-_C7EQ8IUw2Qp7E0-0Z9abjV0xT6I4WK17V2U8JMCcxMl9yvpawBeT-l7c">balancing caregiving</a> with education and work. Many young carers also <a href="https://pubmed.ncbi.nlm.nih.gov/36353077/">don’t self-identify as carers</a>, hindering their access to support and resources. This could be because of the stigmatising label, or a feeling they’re <a href="https://www.ncb.org.uk/sites/default/files/uploads/files/young_people_caring_for_adults_with_dementia.pdf">not doing enough</a> to qualify as a carer. It could even be because of cultural norms which can frame caregiving as a family obligation, rather than a distinct role. Nonetheless, young dementia carers require <a href="https://www.mdpi.com/1660-4601/20/1/127">targeted support</a> beyond generic caregiving resources. This support might include specialised <a href="https://www.tandfonline.com/doi/full/10.2147/JMDH.S464195">peer networks</a>, educational programs, and practical skills training. Tailored programs and resources should ideally be <a href="https://www.mdpi.com/1660-4601/20/1/127">co-designed with young dementia carers</a> to ensure they meet their unique needs and preferences. With dementia cases in Australia and elsewhere <a href="https://www.dementia.org.au/about-dementia/dementia-facts-and-figures">projected to increase</a>, the demand for informal carers – including young adults – will continue to grow. Without intervention, these young carers risk burnout, social isolation, and long-term health consequences. We must ensure flexible, age-appropriate support for this often invisible group. Investing in young dementia carers is not just a moral imperative – it’s a crucial step toward a sustainable, compassionate care system for the future. <a href="https://www.dementia.org.au/living-dementia/family-friends-and-carers">Dementia Australia</a> offers a national helpline, information sessions, and a peer-to-peer connection platform for carers. <a href="https://youngcarersnetwork.com.au/">The Young Carers Network</a>, run by <a href="https://www.carersaustralia.com.au/">Carers Australia</a>, offers mental health resources, financial guidance, and respite care information, plus bursaries young carers can apply for to reduce financial pressure.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/249361/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> By <a href="https://theconversation.com/profiles/katya-numbers-784650">Katya Numbers</a>, Postdoctoral Research Fellow & Lecturer, Centre for Healthy Brain Ageing, <a href="https://theconversation.com/institutions/unsw-sydney-1414">UNSW Sydney</a> and <a href="https://theconversation.com/profiles/serena-sabatini-2320527">Serena Sabatini</a>, Lecturer in Psychology, <a href="https://theconversation.com/institutions/university-of-surrey-1201">University of Surrey</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/more-young-people-are-caring-for-a-loved-one-with-dementia-it-takes-a-unique-toll-249361">original article</a>. Image: Shutterstock </div>

Caring

Beloved Neighbours star reveals his dying wishes

Ian Smith has shared his final wishes after being told he has more time to live, despite battling a rare and aggressive form of cancer. Smith, 86, was diagnosed with pulmonary pleomorphic carcinoma and given a terminal prognosis in 2024. However, in a recent interview on BBC Radio 5 Live, he revealed he had defied expectations and was still going strong. "I'm going to die soon, but I seem to be defying the odds, which is good. I'm not unhappy about that at all," Smith said. The veteran actor recalled the moment he received the unexpected news in December last year, jokingly expressing his gratitude: "The knowledge is saying, 'Ian, you are going to die in March.' I almost gave in, and then on Friday the 13th, I got this good news and, you know what? I just had to say, 'Well, thank you, someone.'" Rather than dwell on his diagnosis, Smith is determined to make the most of the time he has left. While he admits he is still unsure of how best to spend it, one thing on his bucket list is buying a new car – specifically, an electric one. "I'm now thinking of buying a new car. That's not what you do if you're going to die in March," he said with a laugh. "I'm determined to have an electric car before I die. And I was speaking to a salesman. I said, 'Now, what about the delivery? Can you get to me before I'm dead?' And he was so shocked! I said, 'Come on, let's have a giggle.'" Smith has had a long and celebrated career in television, most notably for his role as Harold Bishop on Neighbours. First joining the show in 1987, his character quickly became a fan favourite. Over the years, he made several departures and returns, with his most recent appearance in 2024 before stepping away due to ill health. Jason Herbison, Executive Producer of Neighbours, spoke highly of Smith’s contributions to the show, saying, "The way the residents of Ramsay Street feel about Harold is how we all feel about Ian – we couldn't love him more than we do." Beyond Neighbours, Smith also appeared in the cult series Prisoner and worked extensively in theatre since the 1950s. Smith has faced significant personal challenges over the years. His wife, Gail, passed away in 2019 after a battle with cancer, leaving behind a marriage that spanned over 50 years. Additionally, Smith discovered later in life that he had been adopted. He was 54 when his adoptive mother, Connie, revealed the truth shortly before her passing. Smith eventually reunited with his biological mother, Peg Kline, who had been a victim of sexual assault at age 14. They maintained a relationship until her death in 2005, also from cancer. "Dying does change how you live," he said in a previous interview with The Guardian. "I feel like I have become more forgiving, more understanding." Despite his illness, Smith remains grateful for the life he has led. "I know I have cancer because doctors keep telling me I have it. I may get very sick again one day. But I have lived the most privileged life." Images: Supplied

Caring

Thousands of Aussie women to benefit from cheaper medicine in May

A new treatment for a common painful condition, along with two other drugs, will soon become far more affordable, improving the lives of thousands of Australian women. The federal government announced on Sunday that, from May 1, about 8,500 Australian women are expected to benefit annually from the inclusion of Ryeqo on the Pharmaceutical Benefits Scheme (PBS). The drug offers a new treatment option for endometriosis sufferers who experience moderate to severe pain and have not found relief through other hormonal treatments and painkillers. Endometriosis affects one in seven Australian women, with tissue similar to the womb’s lining growing elsewhere in the body, leading to severe pain and potential fertility issues. Symptoms can vary, often causing delays in diagnosis that average six and a half years, according to Endometriosis Australia. Additional subsidies will also reduce the costs of a progestogen-only contraceptive pill and an IVF hormone therapy. Women with low hormone levels will now have earlier access to Pergoveris for $32 per treatment cycle from their first IVF attempt, rather than waiting for multiple failed cycles. An unsubsidised cycle typically costs around $3,500. Adelaide mother Helana Shehadeh <a href="https://7news.com.au/news/thousands-of-australians-to-benefit-from-cheaper-medicine-c-18056149" target="_blank" rel="noopener">told 7News</a> that she used Pergoveris during her second IVF cycle in 2023, resulting in the birth of baby Zayn. She said that it's important to get early access to fertility treatments, adding that, “Anyone who has been through IVF understands the agony of multiple cycles. Earlier access to this fertility treatment will alleviate some of the burden for women undergoing IVF.” The announcement comes as Australia’s birth rate falls to a record low of 1.5 babies per woman. Fertility Society of Australia and New Zealand president Petra Wale welcomed the move, highlighting the financial strain of assisted reproduction. “The cost of assisted reproduction remains a major barrier for many families, particularly amid the ongoing cost-of-living crisis. Fewer financial and logistical hurdles mean more families can focus on what truly matters – having a baby,” she said. “Women have asked government to take their health care seriously," said Health Minister Mark Butler, "and we have listened. These listings could save women and their families thousands of dollars across their lifetimes.” Addressing criticism over delays in listing Ryeqo, Butler defended the government’s actions, stating that all recommendations had been handled “expeditiously”. He noted that in the previous nine years under the coalition government, “not a single new pill, not a single endometriosis treatment, not a single menopause treatment” was added to the PBS. The opposition, however, criticised the government for leaving women waiting more than a year for “potentially life-changing medications”. Opposition health spokeswoman Senator Anne Ruston reaffirmed the coalition’s commitment to timely PBS listings for all Pharmaceutical Benefits Advisory Committee-recommended medicines. Additionally, the cost of the contraceptive pill Slinda, used by about 80,000 women, will also be reduced under the PBS. The annual price will drop from approximately $320 to $94 for general patients and to $22 for concession card holders. Slinda, a progestogen-only pill, is particularly suitable for older women, smokers, women at increased risk of blood clots, and those who suffer from migraines. Image: Shutterstock

Caring

An artificial heart may save your life. But it can also change you in surprising ways

<div class="theconversation-article-body"> This week, <a href="https://www.theguardian.com/australia-news/2025/mar/12/australian-man-survives-100-days-with-artificial-heart-in-world-first-success">doctors announced</a> that an Australian man with severe heart failure had left hospital with an artificial heart that had kept him alive until he could receive a donor heart. The patient, a man from New South Wales in his 40s, was not the world’s first person to receive this type of artificial heart. However, he is <a href="https://www.svhs.org.au/newsroom/news/australia-first-total-artificial-heart-implant">said to be</a> the <a href="https://www.monash.edu/news/articles/australias-first-durable-total-artificial-heart-implant-announced-as-a-success">first with one to be discharged from hospital</a> to wait for a heart transplant, which he’s since had. <blockquote class="twitter-tweet"> WARNING: GRAPHIC CONTENT This machine has enabled an Australian man with severe heart failure to be the first person in the world to leave hospital with an artificial heart transplant <a href="https://t.co/6S12mINwBm">pic.twitter.com/6S12mINwBm</a> — Reuters (@Reuters) <a href="https://twitter.com/Reuters/status/1899862954155126824?ref_src=twsrc%5Etfw">March 12, 2025</a></blockquote> I am a philosopher and bioethicist. I <a href="https://bridges.monash.edu/articles/thesis/Phenomenology_and_Artificial_Hearts/22312204">completed my PhD</a> on artificial hearts – particularly how these implants can change people’s lives in profound ways. Here’s what patients and their families need to consider. <h2>What is an artificial heart?</h2> Artificial hearts began to be developed in the 1960s, sponsored by the United States government and funded in a similar way to space and military programs. In 1982, a man named <a href="https://www.historynewsnetwork.org/article/hero-or-victim-the-25th-anniversary-of-barney-clar">Barney Clark</a> received the Jarvik-7 total artificial heart. Doctors removed his failing biological heart and replaced it with a plastic and metal device to circulate blood to his lungs and around his body. He lived for 112 days before dying from multi-organ failure. He never left hospital. In the 1980s and 1990s, medical device companies began to develop alternatives to total artificial hearts. These partial artificial hearts, known as ventricular assist devices, help out a biological heart by supplementing or replacing one of its two pumping chambers. These are more straightforward and versatile than total artificial hearts, and can be used for earlier stages of heart failure. Not all artificial hearts generate a pulse. Artificial hearts with a pulse generally mimic the biological heart. They pump blood in the same way the heart beats, by filling with blood and squeezing to circulate blood in waves or pulses. But some devices continuously push blood around the body instead of pulsing. So with these continuous-flow devices neither the patient nor their health team can <a href="https://link.springer.com/article/10.1007/s11906-017-0782-6">detect a pulse</a>. <a href="https://linkinghub.elsevier.com/retrieve/pii/S0003497524008749">In the US between 2014 and 2024</a>, almost 30,000 patients received continuous-flow ventricular assist devices. In the same period, more than 310 total artificial hearts were implanted. The total artificial hearts <a href="https://www.syncardia.com/syncardia-total-artificial-heart-stah.html">commercially</a> <a href="https://www.carmatsa.com/en/our_product/">available</a> today are licensed exclusively as bridging therapies – to keep people alive until a donor heart becomes available – rather than permanent implants. <h2>How about the device making news this week?</h2> The device in the news – the <a href="https://www.monash.edu/news/articles/australias-first-durable-total-artificial-heart-implant-announced-as-a-success">BiVACOR Total Artificial Heart</a> – was developed by a US-Australian collaboration. This device is innovative, mainly because it is the first continuous-flow device designed to replace the whole heart. Designers are also aiming for it to be the first total artificial heart suitable as a permanent transplant (known as destination therapy). A reliable, durable and responsive total artificial heart is, <a href="https://www.abc.net.au/news/2025-03-12/sydney-hospital-artificial-heart-implant-operation-success/105036154">in the words</a> of Paul Jansz, the surgeon who implanted the device, “the Holy Grail”. The BiVACOR’s clinical success so far gives us reason to be optimistic about an alternative to scarce donor hearts for responding to severe heart failure. <figure class="align-center zoomable"><a href="https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&rect=12%2C12%2C8231%2C5475&q=45&auto=format&w=1000&fit=clip"><img src="https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&rect=12%2C12%2C8231%2C5475&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&q=45&auto=format&w=600&h=400&fit=crop&dpr=1 600w, https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&q=30&auto=format&w=600&h=400&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&q=15&auto=format&w=600&h=400&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&q=45&auto=format&w=754&h=503&fit=crop&dpr=1 754w, https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&q=30&auto=format&w=754&h=503&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/655013/original/file-20250313-56-4w24qy.jpg?ixlib=rb-4.1.0&q=15&auto=format&w=754&h=503&fit=crop&dpr=3 2262w" alt="Hand holding the BiVACOR artificial heart" /></a><figcaption>This device is designed to replace the whole heart, and for now, is licensed as a temporary implant, ahead of a heart transplant. BiVACOR TIQ</figcaption></figure> <h2>Transplants can change lives</h2> However, patients do not just resume their old lives when they leave hospital with an artificial heart. While the pumping component is inside their chest, there are also <a href="https://www.instagram.com/p/BHW0ATvgFDu/?hl=en">external components</a> to manage and monitor. A thick tube perforates their abdomen and connects to an external controller unit and power supply, which the patient carries around in a bag. Controllers must be closely monitored, and batteries must be regularly recharged. My research showed that even a perfectly safe and reliable total artificial heart could transform patients’ lives in at least three major areas. 1. Is it part of me? Do I trust it? Patients must <a href="https://www.tandfonline.com/doi/10.1080/09638288.2020.1717648">trust, tolerate and receive sensory feedback about how the device is working</a> for it to feel like part of them. In the case of an artificial heart, this might mean the device feels responsive to exercise and the body’s needs. But it may be difficult for artificial hearts to meet these criteria, especially for devices that do not generate a pulse. Patients may also question whether their heart is located in their body, or in the controller unit. They may wonder if they even have a heart, particularly if they can’t feel a pulse. 2. Beeps and alarms An artificial heart also changes how patients live their lives and <a href="https://link.springer.com/article/10.1007/s11097-024-10050-7">navigate the world</a>. Interruptions from <a href="https://journals.sagepub.com/doi/10.1177/1049732317700853">loud device alarms</a> distract patients from their normal activities. And patients must switch between mains power and batteries when they <a href="https://linkinghub.elsevier.com/retrieve/pii/S0147956311002718">wake in the night and need to visit the toilet</a>. 3. Marking time Our hearts may be our <a href="https://www.frontiersin.org/journals/neurorobotics/articles/10.3389/fnbot.2014.00015/full">natural</a> <a href="https://linkinghub.elsevier.com/retrieve/pii/S0301051111000032">metronomes</a>, marking time. So removing someone’s heart rhythm can confuse their sense of time. The need for batteries to be recharged periodically can also reshape patients’ days. Waiting around for a transplant heart, or the latest software update, may change patients’ perspectives on what months and years feel like. <h2>We need to give patients the whole picture</h2> Artificial hearts are remarkable devices with great promise. But patients and families also deserve to know how these extraordinary treatments might change how they feel about themselves and the world. They need to know this before they sign up for them. Artificial hearts don’t just save lives – they also change them.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/252165/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> By <a href="https://theconversation.com/profiles/pat-mcconville-2344684">Pat McConville</a>, Lecturer in Ethics, Law, and Professionalism, School of Medicine, <a href="https://theconversation.com/institutions/deakin-university-757">Deakin University</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/an-artificial-heart-may-save-your-life-but-it-can-also-change-you-in-surprising-ways-252165">original article</a>. Image: Shutterstock </div>

Body

Devastated mum warns of flu dangers after losing 11-week-old son

A heartbroken mother is pleading with Australians to get vaccinated against the flu this season after losing her 11-week-old son to the virus. Samantha Miller, 37, from Bathurst, NSW, was in the hospital battling her own illness when she received a devastating phone call – her infant son had also fallen ill. By the time he reached the hospital, it was too late. “I came down quite ill, so I ended up in hospital, and then we got a phone call saying he’d been brought into hospital not breathing, and we found out that he’d passed away,” Ms Miller told <a href="https://www.news.com.au/lifestyle/health/health-problems/mothers-harrowing-warning-as-11-weekold-son-dies/news-story/9a453f35136ec6ec0085e10392fd5163" target="_blank" rel="noopener">news.com.au.</a> It has been 13 years since her son’s tragic death, but the pain remains fresh. Now, she is using her heartbreaking experience to encourage others to get vaccinated against the flu. “It took a little while to get some autopsy results, but it came back with bronchial pneumonia caused by H1N1 swine flu,” Ms Miller said, describing the loss as an unimaginable shock. Her son had been seen by a paediatrician the day before his death and had shown no serious signs of illness beyond a runny nose. A photo from the night before, showing the baby peacefully asleep on his grandfather’s chest, still haunts her. “It’s the hardest thing I’ve ever had to do in my entire life. I would not wish it on anybody … it’s been 13 years and it hurts as if it was yesterday,” she said. Determined to prevent other families from suffering the same heartbreak, Ms Miller is now an advocate for flu vaccinations. “I know what it’s like to lose a child to influenza, and vaccination can help prevent other families from going through what we have been through for the past 13 years,” she said. She believes that there is a lack of education about the importance of vaccinations and the dangers of influenza, leading to misinformation and vaccine hesitancy. “Why are some people pushing for anti-vaccination when the numbers are there? They need to look at the data and get a vaccination. I mean, it’s easy enough … take ten minutes out of your life to get the vaccination,” she urged. Ms Miller also highlighted the strain that preventable illnesses place on the already overwhelmed healthcare system. “Anyone who ends up in hospital is putting a strain on the health services that are already under strain,” she said. With flu season approaching, her message is clear: vaccination saves lives. She hopes that by sharing her story, she can help prevent other families from experiencing the same devastating loss. Images: Courtesy of Samantha Miller

Caring

Bruce Willis' wife issues amazing reflection on Gene Hackman's passing

As Bruce Willis continues to live with frontotemporal dementia, his wife, Emma Heming Willis, has emerged as a vocal advocate for those who support loved ones with long-term care needs. This week, she reflected on the tragic <a href="https://www.oversixty.com.au/health/caring/gene-hackman-s-cause-of-death-revealed" target="_blank" rel="noopener">deaths of legendary actor Gene Hackman and his wife</a> and caregiver, Betsy Arakawa, in a heartfelt video shared on Instagram. "So this is not something I would normally comment on, but I do really believe that there is some learning in this story," Heming Willis said of Hackman and Arakawa. The couple was found dead in their New Mexico home in late February. According to the New Mexico medical investigator’s office, Arakawa, 65, succumbed to hantavirus, a rare and often fatal disease, while Hackman, 95, passed away days later due to heart disease. Authorities revealed that Hackman had been living with Alzheimer’s disease and may not have realised he was alone after his wife's passing. Heming Willis used the heartbreaking story to highlight a crucial issue: the well-being of caregivers themselves. "It's just made me think of this broader story, and that is that caregivers need care too and that they are vital, and that it is so important that we show up for them so that they can continue to show up for their person," she stated. She further addressed a common misconception about caregivers, saying that many assume they "have it all figured out". "They've got it covered, they're good," she said before clarifying, "I don't subscribe to that. We need to be showing up for them so they can continue to show up for their person." In the caption of her video, Heming Willis reinforced her message: "Caregivers need care too. Period. Full stop. #supportcaregivers." The flood of responses to her post was immediate and heartfelt, as fans and followers shared their experiences for caring for loved ones. "I had the same terrified reaction to Gene Hackman," wrote one reader. "What must he have suffered those last days? And his wife? It’s unfathomable. My father was a dedicated caregiver to my mom. Realising that dementia takes every ounce of patience and energy, my two sisters and I were honoured to take one full day each per week (in addition to afternoon nurses two alternate days) to support my dad’s decision to keep mama at home. She’s been gone three years now and I have never once regretted those special Fridays with her. Your message is so very important." "I’m so glad you said this," wrote another reader. "Because that’s exactly what I was thinking when this happened that his wife was so devoted to him that she didn’t take care of herself and this happens a lot with women a lot! She didn’t have any children, but I’m amazed that no friends checked in on her." Heming Willis speaks from personal experience, having taken on the role of primary caregiver for her husband since his diagnosis. It was announced in 2022 that Bruce Willis would step away from his acting career due to cognitive issues, <a href="https://www.oversixty.com.au/health/caring/cruel-disease-bruce-willis-given-heartbreaking-new-diagnosis" target="_blank" rel="noopener">which were later identified as frontotemporal dementia</a> (FTD), a progressive brain disorder. The couple, who married in 2009, share two daughters, Mabel and Evelyn. Through her platform, Heming Willis remains committed to raising awareness and fostering support for those navigating the complexities of long-term care. Images: Instagram

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Aussie Olympian air-lifted to Greece after horror crash

Australian Winter Olympian Belle Brockhoff is recovering in a Greek hospital following a serious crash at a Snowboard Cross World Cup event in Eastern Europe. The 32-year-old world champion sustained a significant injury during her final race of the program on Saturday, crashing at high speed and landing on her coccyx. Brockhoff was immediately airlifted to a hospital in Tbilisi, Georgia, where scans revealed a fracture in her L1 vertebrae. While she was cleared of neurological damage, the severity of her injury required an emergency evacuation to Athens for surgery. Her partner, Georgia, quickly boarded a flight to be by her side, while an Olympic Winter Institute of Australia (OWIA) medical staffer has been assisting her throughout the ordeal. Brockhoff is expected to remain in hospital for up to two weeks before being cleared to return home to Australia. OWIA chief Geoff Lipshut assured that Brockhoff is receiving the best medical care possible. "Currently, our medical team is working with emergency service personnel and the hospital in Greece to ensure that Belle receives the best care possible," Lipshut said. "We will reassess options together with Belle following surgery over the next 48 hours." Despite the setback, Brockhoff remains in good spirits. Lipshut expressed relief that her partner could join her in Greece to provide additional support. The injury comes as another blow to Brockhoff, who recently recovered from what her surgeon described as "the worst" broken wrist he had ever seen. With just 11 months remaining until the 2026 Winter Olympics in Italy, her latest setback could not have come at a worse time. Brockhoff has a history of pushing through injuries in pursuit of Olympic success. She competed at the PyeongChang 2018 Winter Olympics with her knee heavily taped and braced after undergoing ACL removal surgery. More recently, she sought redemption after a heartbreaking fourth-place finish in the women’s snowboard cross final at the Beijing 2022 Winter Olympics. Frustrated by her near-miss, she bluntly described her result as “the loser” and expressed disappointment at missing the podium. Her Beijing campaign ended dramatically when she crashed out of the mixed team event, lying motionless on the course before being stretchered off by medics. Now, she faces yet another uphill battle in her journey back to competition. Images: Instagram

Caring

"I'm not ready to go yet": Leading Aussie doctor given months to live

Leading melanoma researcher and Australian of the Year recipient Professor Richard Scolyer has revealed that he has only months to live following the return of his brain cancer. Diagnosed with incurable glioblastoma two years ago, Professor Scolyer chose to <a href="https://www.oversixty.com.au/health/caring/doctor-beats-cancer-using-his-own-treatment" target="_blank" rel="noopener">undergo an experimental immunotherapy treatment</a>, defying his initial six-month prognosis. His remarkable journey was documented in a study published in late February, detailing the innovative approach he helped pioneer. However, after undergoing surgery last week, doctors confirmed that the aggressive cancer had returned. “Unfortunately, there is a larger volume of quickly growing brain cancer (glioblastoma, IDH WT, unmethylated etc.) in my left brain,” Professor Scolyer shared in an emotional social media post on Monday. “The prognosis is poor. Whilst some of my recurrent tumour was removed last week, some of it couldn’t be because of its site.” Despite the devastating news, Professor Scolyer remains hopeful that the experimental treatment he volunteered for could pave the way for future medical breakthroughs. Alongside his colleague and friend, Professor Georgina Long, he was named a 2024 Australian of the Year for their groundbreaking work in melanoma research and immunotherapy advancements. “I greatly appreciate the support of all my family, friends, and colleagues who have been looking after me so well and the incredible research that has been performed,” he said. “I hope the latter will change the field and patient outcomes in the not too distant future.” In a heartfelt interview on A Current Affair with Ally Langdon on Monday, the father-of-three shared his decision to forgo further experimental treatments, choosing instead to spend his remaining time with his loved ones. “It could help, but my heart’s not in the same place as what it was before,” he admitted. “It’s not fair, but there’s a lesson for everyone … make the most of every day because you don’t know what’s around the corner.” “I’m not ready to go yet. I love my life,” he said. “I guess I feel lucky that I’ve been able to go for so long without a recurrence of it.” Images: A Current Affair

Caring

Prince Robert of Luxembourg announces youngest son's death

Prince Robert of Luxembourg has announced the heartbreaking loss of his youngest son, Frederik, at the age of 22. Frederik is Prince Robert's youngest son with wife Princess Julie of Nassau. According to a statement shared on the <a href="https://polgfoundation.org/frederik/" target="_blank" rel="noopener">POLG Foundation's</a> website, Frederik died on March 1 in Paris, France, from POLG Mitochondrial disease, a rare genetic disorder that causes progressive multiple organ dysfunction and failure. "It is with a very heavy heart that my wife and I would like to inform you of the passing of our son, The POLG Foundation Founder and Creative Director, Frederik," the statement, written by Robert began. Frederik, was born in Aix-en-Provence, France, on March 18, 2002, with POLG Mitochondrial disease, but was only diagnosed at the age of 14. He and his father later co-founded the POLG Foundation, with Frederik serving as Creative Director while Robert served as Chair. "Last Friday, February 28th, on 'Rare Disease Day', our beloved son called us in to his room to speak to him for one last time," the statement continued. "Frederik found the strength and the courage to say goodbye to each of us in turn." That included his brother, 27-year-old Prince Alexandre, his sister, 29-year-old Princess Charlotte and her 27-year-old husband, Mansour Shakarchi. Frederik also said his final goodbyes to his father's sister, Princess Charlotte, her husband Marc Victor Cunningham, and their three children: Charly, Louis, and Donall. "He had already spoken all that was in his heart to his extraordinary mother, who had not left his side in 15 years," Robert continued. "After gifting each of us with our farewells – some kind, some wise, some instructive – in true Frederik fashion, he left us collectively with a final long-standing family joke. "Even in his last moments, his humour, and his boundless compassion, compelled him to leave us with one last laugh….to cheer us all up." Prior to his other goodbye messages, Robert said, Frederik asked him: "Papa, are you proud of me?" "He had barely been able to speak for several days, so the clarity of these words was as surprising as the weight of the moment was profound," Robert wrote. "The answer was very easy, and he had heard it oh so many times, but at this time, he needed reassurance that he had contributed all that he possibly could in his short and beautiful existence and that he could now finally move on." "Frederik knows that he is my Superhero, as he is to all of our family, and to so very many good friends," Robert continued, noting how his son "was born with a special capacity for positivity, joy, and determination." "When he was little, I would always say that if there is one child of ours that I would never need to worry about, it was him," he continued. "He has social skills like no other, an amazing sense of humour, an emotional intelligence and compassion that were off the charts, a sense of justice, fairness and decency that knows no bounds. He was disciplined and organised beyond belief. " "Frederik is particularly headstrong (at times, I might have used the word stubborn). This serves him well. I cite him as being here in the present because I know that, though physically absent, he is still here, inspiring us and pushing us forward. His mission is not complete, and we all have much work left to do." Robert then thanked their family for their "messages of support and love", before asking those reading the tribute to volunteer with or donate to the POLG Foundation in Frederik's memory. "With our Superhero's help we hope to turn our deep grief into positive results and therewith follow his unwavering example," he wrote. "His ultimate message is one of hope, compassion, and resilience! We are all so very proud of you, Frederik. I am so very proud of you! We love you!" Images: The POLG Foundation

Caring

Gene Hackman's cause of death revealed

The cause of death of legendary actor Gene Hackman, 95, and his wife Betsy Arakawa, 65, has finally been confirmed, more than a week after they were <a href="https://www.oversixty.com.au/health/caring/sad-new-details-emerge-in-gene-hackman-case" target="_blank" rel="noopener">discovered in their Santa Fe home</a>. The couple was found deceased in their residence on February 26, prompting an investigation by authorities. Dr Heather Jarrell, Chief Medical Examiner for New Mexico, disclosed at a press conference that Hackman succumbed to cardiovascular disease, with advanced Alzheimer’s disease being a significant contributing factor. His wife, Betsy Arakawa, died around seven days earlier from hantavirus pulmonary syndrome, a rare infectious disease contracted through exposure to rodents and their excrement. Authorities found evidence of rodent intrusion in the couple’s home, believed to be the source of Arakawa’s infection. Based on her movements and email communications, Dr Jarrell determined that Arakawa likely passed away on February 11, while Hackman is believed to have died on February 18. Santa Fe County Sheriff Adan Mendoza confirmed that Hackman was likely at home with his deceased wife for seven days before his own death. Dr Jarrell noted that Hackman was in "very poor health", and it was unclear how he managed in his final days without his wife's assistance. Given his advanced Alzheimer’s, Jarrell suggested that it was possible he was unaware that his wife had passed away. “There was no food in his stomach, which means he had not eaten recently, but he had no evidence of dehydration,” she stated. Hantavirus pulmonary syndrome is a severe and often fatal respiratory disease. According to the Mayo Clinic, it begins with flu-like symptoms and rapidly progresses to serious lung and heart complications. The best prevention is avoiding contact with rodents and properly managing rodent-prone areas. Dr Jarrell’s findings confirmed that Hackman had severe heart disease, with evidence of multiple prior heart attacks and chronic high blood pressure. A full-body post-mortem examination showed no signs of trauma. He tested negative for COVID-19, influenza and other respiratory illnesses, as well as hantavirus and carbon monoxide poisoning. Similarly, Arakawa’s autopsy revealed no external or internal trauma. Laboratory tests confirmed the presence of hantavirus, ruling it as the cause of her death. Other tests for respiratory viruses and toxins returned negative results. Dr Jarrell said that there were “no other significant natural disease findings” in either case, and that all medications found in the home were taken as prescribed and did not contribute to their deaths. The tragic passing of Gene Hackman and Betsy Arakawa marks the end of an era for Hollywood, with fans mourning the loss of the two-time Academy Award-winning actor. Hackman, known for his roles in The French Connection, Unforgiven and The Royal Tenenbaums, retired from acting in 2004. The couple had been residing in Santa Fe for many years, enjoying a quiet life away from the public eye. Image: Instagram

Caring

Lisa Curry's touching tribute to mum three years on

Lisa Curry has shared heartfelt tribute to her mum on the third anniversary of her death. The Australian swimming icon took to Instagram to reflect on their special bond, with an emotional video montage of some of her favourite memories with her mum. “Thinking of my mum all day today. 3 years since we said goodbye but she’s always around. 🤍👼🏻 Mums are amazing … make sure you call yours,” she wrote in the caption. Curry's mum passed away on March 3, 2022, at the age of 86. <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/reel/DGu7bu-TEX4/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/reel/DGu7bu-TEX4/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Lisa Curry AO (@lisacurry)</a> </div> </blockquote> The tribute received an outpouring of support from fans, who shared their condolences and love. “Thinking of you Lisa, you look like your dear mum. Too many losses in such a short time. xx,” one wrote. “She was your biggest fan may she be watching over you always," another commented. “Lisa such a beautiful daughter and Mum so much love,” commented another. “Sending you love and hugs Lisa," added another. Curry announced the heartbreaking news of her mother's passing in a social media post shared on March 2022. At the time, she expressed how deeply the loss had affected her, as well as the loss of her daughter Jaimi Lee Kenny in September 2020. “Our beautiful mum slipped away peacefully this morning,” Curry wrote at the time. “My heart is broken again. My baby girl, and now my mum.” She ended the post with a heartfelt thank you to her late mum, for being a "wonderful mother". “Have a nice long sleep mum. We love you so much and we’ll all miss you.” Images: Instagram

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