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Terminally ill 23-year-old's final request

Liam Bulman was working a shift at The Illawarra Hotel in Wollongong last month when he asked to leave early, as he felt unwell. The following week he found himself in "crippling pain" and was rushed to hospital by his friend. After days of intensive testing, the 23-year-old was diagnosed with stage-four High Grade Neuroendocrine Carcinoma — a "rare" cancer that had spread. Now, his family and friends are doing everything they can to raise funds so he can have a "chance to make memories" through ticking off items on his bucket list in this "scary, sad and unknown" time. "He's smart and kind. He's been raised to work hard and contribute to people's lives, and our community. He's incredible," Liam's colleagues from The Illawarra Hotel wrote on the<a href="https://www.gofundme.com/f/youre-friendly-local-bartender-bulmans-bucket-list" target="_blank" rel="noopener"> GoFundMe</a> page. So far they have raised over $28,000 of their $50,000 goal, with most of the money raised going towards his medical costs, medical trial attendance and palliative care, and any remaining money will help fund his bucket list. His bucket list includes activities like attending the F1 in Melbourne, going for a joyride in a helicopter, learning to surf and taking a family photo session. Liam's mum, Emily Bulman, revealed his cancer is "incurable" and he has since started a bout of chemotherapy in hope it will "buy him more time" as the cancer has spread to his lymph nodes and lower digestive tract. "This will assist us to aim for longevity, quality of life, a chance to make memories, and receive the care and comfort that suits Liam best," she said on the fundraising page. "Anything we can raise to help take the burden off Liam, we would be incredibly grateful for." Images: GoFundMe/Facebook

Caring

Dan Andrews' statue mystery unfolds

Melbourne is abuzz with a burning question: where, oh where, will the statue of former premier Daniel Andrews be placed? More importantly, will it capture his iconic North Face jacket-clad form, standing proud in the face of lockdown chaos? Yes, it appears that a bronze likeness of the man who guided (and locked) Victoria through some of its toughest years is in the works. The <a href="https://www.heraldsun.com.au/news/victoria/former-premier-dan-andrews-to-be-immortalised-in-new-statue/news-story/68d37a765269a629baca12c08cc74e1c" target="_blank" rel="noopener">Herald Sun recently revealed</a> that the Allan government has confirmed that Dan Andrews will soon be immortalised in Melbourne, joining the ranks of past Victorian premiers like John Cain Jr, Rupert Hamer and Henry Bolte – legends who, let's be honest, didn’t have to worry about memorialising themselves in the middle of a pandemic-fuelled political firestorm. But the government isn’t spilling all the tea just yet. They’ve refused to say exactly where Dan's statue will stand or how much it will cost us. Will it be along the esteemed Treasury Place? Or will they go for something a little more subtle – like a sneaky placement next to a suburban train station, so you can admire his bronzed visage while waiting for a delayed train? Or even – as one disgruntled Facebook user suggested – "at the bottom of the Yarra River"? It's been reported that the statue could easily cost north of $100,000, but what’s a little six-figure sum when you’re eternally freezing a man in time who made your curfew 8pm sharp? The statute eligibility stems from Andrews' 3000-day reign as premier, a milestone introduced by former premier Jeff Kennett, who, sadly, never reached the magic number. It’s almost like Kennett set the bar just to have Dan leap right over it. We’re picturing Kennett in his living room, shaking his fist at a calendar – he could’ve been immortalised in bronze, but alas, his term was cut short. He’s now left without the joy of being a bronzed footnote in history. Meanwhile, sculptor Peter Corlett, the mastermind behind the previous premier statues, has been pondering what pose should immortalise Andrews. Should it be the famous “fists in the air” moment from his 2022 election victory – a rare, almost gladiatorial display of emotion for the typically stoic Andrews? Or will the statue take inspiration from his pandemic press conferences, with him wrapped in his signature North Face jacket, standing solid, as if braving the cold front of public opinion? Perhaps he’ll be gesturing towards an unseen map, locking down regions in real-time. For extra authenticity, pigeons could be programmed to circle around, completing the press conference vibe. While the government is busy with this monument of monumental importance, Opposition finance spokeswoman Jess Wilson has questioned whether now is the best time for such a grand expense. After all, it’s hard to be thrilled about bronze when you’re facing surging crime and hospital wait times that feel longer than the five-day snap lockdown that wasn’t so snappy. Her argument? "Maybe taxpayers would prefer a statue of a budget surplus instead." Of course, there are some whispers suggesting that perhaps Victoria could consider honouring a leading female figure instead. You know, just to mix things up a little in the boys’ club of bronze. But for now, Dan Andrews seems set to be cast in metal. So, while we wait to discover where this shiny new addition to the Melbourne cityscape will be unveiled, we can start placing our bets. Will it be along Treasury Place? Will it be out in the suburbs, standing sentinel in the middle of a roundabout? Or – dare we dream – will it be perched right outside the North Face store, ensuring Melbourne’s most iconic premier (and jacket wearer) is always in fashion? Let the statue speculation begin. And remember, as you ponder the deep mysteries of bronze premier placement, at least this time, you’re not in lockdown. Image: Shutterstock

Domestic Travel

I feel sick. How do I know if I have the flu, COVID, RSV or something else?

<div class="theconversation-article-body"> <a href="https://theconversation.com/profiles/natasha-yates-1213624">Natasha Yates</a>, <a href="https://theconversation.com/institutions/bond-university-863">Bond University</a> You wake with a sore throat and realise you are sick. Is this going to be a two-day or a two-week illness? Should you go to a doctor or just go to bed? Most respiratory illnesses have very similar symptoms at the start: sore throat, congested or runny nose, headache, fatigue and fever. This may progress to a dry cough. Best case scenario is that you have “<a href="https://lungfoundation.com.au/wp-content/uploads/2018/09/Factsheet-Common-Cold-Mar2016.pdf">a cold</a>” (which can be any one of hundreds of viruses, most commonly rhinovirus), which is short-lived and self-limiting. But some respiratory illnesses can be much more serious. Here is a brief guide to some important bugs to know about that are circulating this winter, and how to work out which one you have. <h2>Respiratory syncytial virus (RSV)</h2> For most people an RSV infection will feel like “a cold” – annoying, but only lasting a few days. However, for babies, older adults and people with immune issues, it can lead to <a href="https://www.rch.org.au/kidsinfo/fact_sheets/bronchiolitis/">bronchiolitis</a> or pneumonia, and even become life-threatening. RSV isn’t seasonal, which means you are just as likely to get it in summer as in winter. However, it is highly contagious so we noticed it <a href="https://pubmed.ncbi.nlm.nih.gov/32986804">disappearing almost completely</a> during COVID lockdowns. There is now a <a href="https://www.tga.gov.au/sites/default/files/2024-02/covid-19-rapid-antigen-self-tests-are-approved-australia-ifu-406813.PDF">rapid-antigen test (RAT) for RSV</a> which also checks for influenza and COVID, and is the best way of finding out if RSV is what is causing symptoms. Recently, a preventative immune therapy has become available for high risk babies (<a href="https://www.schn.health.nsw.gov.au/respiratory-syncytial-virus-rsv-monoclonal-antibody-factsheet">nirsevimab</a>) and there are also <a href="https://ncirs.org.au/ncirs-fact-sheets-faqs-and-other-resources/respiratory-syncytial-virus-rsv-frequently-asked">vaccines for higher risk adults</a>. Nirsevimab is also available to all babies for free in <a href="https://www.health.wa.gov.au/Articles/N_R/Respiratory-syncytial-virus-RSV-immunisation">Western Australia</a> and <a href="https://www.health.qld.gov.au/clinical-practice/guidelines-procedures/diseases-infection/immunisation/paediatric-rsv-prevention-program">Queensland</a>. But there are no specific treatments. Adults who get it simply have to ride it out (using whatever you need to <a href="https://www.mayoclinic.org/diseases-conditions/common-cold/diagnosis-treatment/drc-20351611">manage symptoms</a>). Babies and higher risk patients need to present to an emergency department if they test positive for RSV and are also looking or feeling very unwell (this might mean rapid shallow breathing, fevers not coming down with paracetamol or ibuprofen, a baby not feeding, mottled-looking skin, or going blue around the mouth). If a patient has developed a bronchiolitis or pneumonia, they may need to be hospitalised. <h2>Influenza</h2> Once you have had the “true flu” (influenza), you will find it frustrating when people call their sniffly cold-like symptoms a “flu”. Influenza infections generally start with a sore throat and headache which quickly turns into high fevers, generalised aches and excessive fatigue. You feel like you have been hit by a truck and may struggle to get out of bed. This can last a week or more, even in people who are generally fit and healthy. Influenza is a major public health issue internationally, with 3–5 million cases of severe illness and <a href="https://www.who.int/news-room/fact-sheets/detail/influenza-(seasonal)">290,000 to 650,000 respiratory deaths annually</a>. People who are at <a href="https://immunisationhandbook.health.gov.au/contents/vaccine-preventable-diseases/influenza-flu">greater risk of complications</a> from influenza include pregnant women, children under five, adults aged 65 and over, First Nations peoples, and people with chronic or immunosuppressive medical conditions. For this reason, annual vaccination is <a href="https://www.health.gov.au/topics/immunisation/vaccines/influenza-flu-vaccine">recommended and funded</a> for vulnerable people. Vaccination is also readily available for <a href="https://www.health.gov.au/topics/immunisation/immunisation-contacts">all Australians who want it</a>, through pharmacies as well as medical clinics, usually at a cost of less than A$30. In <a href="https://www.vaccinate.initiatives.qld.gov.au/what-to-vaccinate-against/influenza#:%7E:text=The%20flu%20vaccine%20is%20free,.qld.gov.au">some states</a>, it’s free for all residents. Influenza is seasonal, with definite peaks in the winter months. This is why vaccines are offered from early autumn. If you think you may have influenza, there are now home-testing RATs: all current influenza RATs are in combination with COVID RATs, as the symptoms overlap. Treatment for most people is to manage symptoms and try to avoid spreading it around. Doctors can also <a href="https://theconversation.com/i-think-i-have-the-flu-should-i-ask-my-gp-for-antivirals-210457">prescribe antivirals</a> to vulnerable patients; these work best if started within 48 hours of symptoms. <h2>COVID</h2> It has been less than five years since COVID-19, caused by SARS-CoV-2, started to spread around the world in pandemic proportions. Although COVID is no longer a <a href="https://www.health.gov.au/news/ahppc-statement-end-of-covid-19-emergency-response">public health emergency</a>, it still causes <a href="https://www.abs.gov.au/articles/deaths-due-covid-19-influenza-and-rsv-australia-2022-may-2024">more deaths than influenza and RSV combined</a>. Unlike RSV and influenza, only those <a href="https://www.health.gov.au/topics/covid-19/protect-yourself-and-others/high-risk-groups">aged over 70</a> are in a high-risk age group for COVID. Other <a href="https://www.cdc.gov/covid/risk-factors/?CDC_AAref_Val=https://www.cdc.gov/coronavirus/2019-ncov/need-extra-precautions/people-with-medical-conditions.html">factors besides age</a> may put you at higher risk of becoming very unwell when infected by this virus. This includes having other respiratory diseases (such as asthma or chronic obstructive pulmonary disease, also known as COPD), diabetes, cancer, kidney disease, obesity or heart disease. Unlike most respiratory viruses, SARS-CoV-2 tends to set off inflammation beyond the respiratory system. This can involve a range of other organs including the heart, kidneys and blood vessels. Although most people are back to their usual work or study after a week or two, a significant proportion go on to experience extended symptoms such as fatigue, breathlessness, brain fog and mood changes. When these last <a href="https://aci.health.nsw.gov.au/statewide-programs/critical-intelligence-unit/post-acute-sequelae">more than 12 weeks</a>, without any other explanation for symptoms, it’s called <a href="https://www.healthdirect.gov.au/covid-19/post-covid-symptoms-long-covid">long COVID</a>. COVID vaccines can prevent serious illness and have been <a href="https://pubmed.ncbi.nlm.nih.gov/38282394/">monitored</a> for several years now for their safety and effectiveness. Current vaccination recommendations are <a href="https://www.health.gov.au/resources/publications/atagi-statement-on-the-administration-of-covid-19-vaccines-in-2024?language=en">based on age and immune status</a>. It’s worth discussing them with your doctor if you are unsure whether you would benefit or not. <a href="https://www.health.gov.au/topics/covid-19/oral-treatments">Antivirals</a> can treat COVID in higher-risk people who contract it, whether vaccinated or not. Specific advice about what to do if you test positive on a RAT will vary according to your current state guidelines and workplace, however the <a href="https://www.health.gov.au/topics/covid-19/testing-positive">general principles</a> are always: avoid spreading the virus to others, and give yourself time to rest and recover. <hr /> <iframe id="ConNR" class="tc-infographic-datawrapper" style="border: 0;" src="https://datawrapper.dwcdn.net/ConNR/" width="100%" height="400px" frameborder="0" scrolling="no"></iframe> <hr /> <h2>What if it’s not one of those?</h2> So you’ve done your combined RSV/flu/COVID RAT and the result is negative. But you still have symptoms. What else could it be? More than 200 different viruses can cause cold and flu symptoms, including rhinovirus (mentioned above), adenovirus and sometimes even <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2130424/">undefined pathogens</a>. If an illness progresses to a cough which will not go away, and/or you start coughing up sputum, this could be a bacterial infection, such as pertussis (whooping cough), streptococcus pneumoniae, haemophilus influenzae or moraxella catarrhalis. So it’s worth <a href="https://www.racgp.org.au/getattachment/0a637812-c8f0-45a2-af9c-fa215b64f8e4/attachment.aspx">getting assessed by a GP</a> who may do a chest Xray and/or <a href="https://www.rcpa.edu.au/Manuals/RCPA-Manual/Pathology-Tests/M/MCS-sputum">test your sputum</a>, particularly if they suspect pneumonia. You also may also start out with what is clearly a viral infection but then get a secondary bacterial infection later. So if you are getting more unwell over time, it’s worth getting tested, in case antibiotics will help. However, taking antibiotics for a purely viral illness will not only be useless, it can contribute to harmful <a href="https://www.nps.org.au/consumers/antibiotic-resistance-the-facts">antibiotic resistance</a> and give you unwanted side effects.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/234266/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/natasha-yates-1213624">Natasha Yates</a>, General Practitioner, PhD Candidate, <a href="https://theconversation.com/institutions/bond-university-863">Bond University</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/i-feel-sick-how-do-i-know-if-i-have-the-flu-covid-rsv-or-something-else-234266">original article</a>. </div>

Body

Major breakthrough solves 44-year-old cold case

A decades-old mystery surrounding a human jawbone found on Umina Beach on the NSW Central Coast has been solved, thanks to advances in DNA analysis techniques. The jawbone, discovered by a dog walker in June 2020, was recently identified as belonging to a teenager who tragically drowned nearly half a century ago. Police announced the breakthrough in a statement on Sunday, revealing that the jawbone belonged to Henry Coleman, a 17-year-old who died in an accidental drowning on the Central Coast in August 1980. The remains, though blackened, were remarkably well-preserved and still contained some teeth when found on the beach. Detectives initially determined the remains were human and male, but extensive traditional inquiries failed to establish an identity. It wasn't until March 2023 that a major breakthrough was made using new DNA analysis technology. This technology, which combines advanced DNA analysis with traditional genealogy, allowed investigators to use commercial DNA databases to identify suspects and unknown remains. The key to solving the case was a genetic link to a possible living relative. A voluntary DNA sample from this relative confirmed the jawbone belonged to Coleman. The young man had been laid to rest in the 1980s, but it was not known at the time that part of his jaw was missing. Detective Inspector Ritchie Sim highlighted the importance of public participation in resolving missing persons cases. "This investigation showcases the importance of DNA testing in missing person cases," he said. "Without the combined efforts of our detectives, scientists, and analysts, we would not have been able to return these remains to their resting place and provide closure to Mr Coleman’s family." Sim went on to urge those who have reported a family member missing to police, and who have the Event number, to provide their DNA at a local police station. "Just like in Mr Coleman’s case, your relationship with the missing person can be several generations apart," he added. Images: 7News

Legal

Mysterious leg pain that’s quietly killing older Australians

Peripheral Artery Disease (PAD) has long been overshadowed by its more widely recognised counterparts, such as heart attacks and strokes. Often referred to as the “poor cousin” or “Cinderella” of cardiovascular diseases, PAD affects one in five older Australians, yet it remains largely under-diagnosed and misunderstood. However, a wave of optimism is sweeping through the medical community with the launch of <a href="https://www.hri.org.au/our-research/centreforpad" target="_blank" rel="noopener">The Centre for Peripheral Artery Disease</a>, an Australian-first facility dedicated to pioneering research and improving patient outcomes. Spearheaded by the Heart Research Institute, the Centre for Peripheral Artery Disease, marks a significant step forward in addressing the challenges posed by PAD. The centre aims to fill critical gaps in our understanding of the disease, which is responsible for a limb amputation in Australia every two hours. This initiative promises to enhance diagnosis, transform patient care, and raise awareness about PAD. Associate Professor Mary Kavurma, the Centre Lead, is at the forefront of this ground-breaking effort. “We’re supercharging research into PAD because there are still many unknowns about the disease’s biology that could unlock new methods for early detection and better management,” she explains. This research is particularly urgent given the prevalence of PAD among women and First Nations Australians, groups that remain disproportionately affected by this condition. The centre’s mission is to develop a simple blood test for early diagnosis and explore novel therapies that could reduce the need for limb amputations and significantly improve patients’ quality of life. Unlike current treatments that primarily focus on symptom management, this new approach seeks to prevent the disease from progressing. One of the most inspiring aspects of the centre is its commitment to involving patients and their families in the research process. At the inaugural consumer meeting, nearly 20 patients and their carers shared their personal experiences with PAD. Take the story of Simon Josephson, a renowned advertising guru – who famously designed the Solo logo. PAD almost cost him his life after taking more than five years to diagnose. He woke up one morning with a sore leg, thinking he’d overdone it exercising but the 73-year-old – who was otherwise healthy and active – unknowingly had Peripheral Artery Disease, caused by a build-up of plaque in his arteries causing them to narrow and stiffen. It wasn’t until a trip to the hospital emergency department years later that doctors would discover his aorta had expanded to more than twice the usual size and was at risk of rupturing. He immediately underwent open heart surgery and has faced a lengthy recovery. The launch of <a href="https://www.hri.org.au/our-research/centreforpad" target="_blank" rel="noopener">The Centre for Peripheral Artery Disease</a> heralds a new era of hope and progress in the fight against PAD. Through world-leading research, community engagement, and a commitment to patient-centred care, the CPAD is poised to make a profound impact on the lives of many Australians. As Assoc Prof Kavurma aptly put it, “By understanding more about this debilitating condition, we are paving the way for better health outcomes and a brighter future for all those affected by PAD.” Images: CPAD

Caring

Do you have a mental illness? Why some people answer ‘yes’, even if they haven’t been diagnosed

<div class="theconversation-article-body"> <a href="https://theconversation.com/profiles/jesse-tse-1429151">Jesse Tse</a>, <a href="https://theconversation.com/institutions/the-university-of-melbourne-722">The University of Melbourne</a> and <a href="https://theconversation.com/profiles/nick-haslam-10182">Nick Haslam</a>, <a href="https://theconversation.com/institutions/the-university-of-melbourne-722">The University of Melbourne</a> Mental illnesses such as depression and anxiety disorders have become more prevalent, especially among <a href="https://www.aihw.gov.au/mental-health/overview/prevalence-and-impact-of-mental-illness#changeovertime">young people</a>. Demand for treatment is surging and prescriptions of some <a href="https://pubmed.ncbi.nlm.nih.gov/35176912/">psychiatric medications</a> have climbed. These upswinging prevalence trends are paralleled by rising public attention to mental illness. Mental health messages saturate traditional and social media. Organisations and governments are developing awareness, prevention and treatment initiatives with growing urgency. The mounting cultural focus on mental health has obvious benefits. It increases awareness, reduces stigma and promotes help-seeking. However, it may also have costs. Critics worry <a href="https://www.bacp.co.uk/bacp-journals/therapy-today/2023/april-2023/the-big-issue/">social media</a> sites are incubating mental illness and that ordinary unhappiness is being pathologised by the overuse of diagnostic concepts and “<a href="https://www.bustle.com/wellness/is-therapy-speak-making-us-selfish">therapy speak</a>”. British psychologist <a href="https://www.psych.ox.ac.uk/team/lucy-foulkes">Lucy Foulkes</a> argues the trends for rising attention and prevalence are linked. Her “<a href="https://www.sciencedirect.com/science/article/pii/S0732118X2300003X">prevalence inflation hypothesis</a>” proposes that increasing awareness of mental illness may lead some people to diagnose themselves inaccurately when they are experiencing relatively mild or transient problems. Foulkes’ hypothesis implies that some people develop overly broad concepts of mental illness. Our research supports this view. In a new study, <a href="https://www.sciencedirect.com/science/article/pii/S2666560324000318?via%3Dihub">we show</a> that concepts of mental illness have broadened in recent years – a phenomenon we call “<a href="https://www.tandfonline.com/doi/full/10.1080/1047840X.2016.1082418">concept creep</a>” – and that <a href="https://bmcpsychiatry.biomedcentral.com/articles/10.1186/s12888-023-05152-6">people differ</a> in the breadth of their concepts of mental illness. <h2>Why do people self-diagnose mental illnesses?</h2> In our new <a href="https://doi.org/10.1016/j.ssmmh.2024.100326">study</a>, we examined whether people with broad concepts of mental illness are, in fact, more likely to self-diagnose. We defined self-diagnosis as a person’s belief they have an illness, whether or not they have received the diagnosis from a professional. We assessed people as having a “broad concept of mental illness” if they judged a wide variety of experiences and behaviours to be disorders, including relatively mild conditions. We asked a nationally representative sample of 474 American adults if they believed they had a mental disorder and if they had received a diagnosis from a health professional. We also asked about other possible contributing factors and demographics. Mental illness was common in our sample: 42% reported they had a current self-diagnosed condition, a majority of whom had received it from a health professional. Unsurprisingly, the strongest predictor of reporting a diagnosis was experiencing relatively severe distress. The second most important factor after distress was having a broad concept of mental illness. When their levels of distress were the same, people with broad concepts were substantially more likely to report a current diagnosis. The graph below illustrates this effect. It divides the sample by levels of distress and shows the proportion of people at each level who report a current diagnosis. People with broad concepts of mental illness (the highest quarter of the sample) are represented by the dark blue line. People with narrow concepts of mental illness (the lowest quarter of the sample) are represented by the light blue line. People with broad concepts were much more likely to report having a mental illness, especially when their distress was relatively high. People with greater mental health literacy and less stigmatising attitudes were also more likely to report a diagnosis. Two interesting further findings emerged from our study. People who self-diagnosed but had not received a professional diagnosis tended to have broader illness concepts than those who had. In addition, younger and politically progressive people were more likely to report a diagnosis, consistent with some <a href="https://www.sciencedirect.com/science/article/pii/S2666560321000438">previous research</a>, and held broader concepts of mental illness. Their tendency to hold these more expansive concepts partially explained their higher rates of diagnosis. <h2>Why does it matter?</h2> Our findings support the idea that expansive concepts of mental illness promote self-diagnosis and may thereby increase the apparent prevalence of mental ill health. People who have a lower threshold for defining distress as a disorder are more likely to identify themselves as having a mental illness. Our findings do not directly show that people with broad concepts over-diagnose or those with narrow concepts under-diagnose. Nor do they prove that having broad concepts causes self-diagnosis or results in actual increases in mental illness. Nevertheless, the findings raise important concerns. First, they suggest that rising mental health awareness may <a href="https://www.newscientist.com/article/mg25934573-900-why-being-more-open-about-mental-health-could-be-making-us-feel-worse/">come at a cost</a>. In addition to boosting mental health literacy it may increase the likelihood of people incorrectly identifying their problems as pathologies. Inappropriate self-diagnosis can have adverse effects. Diagnostic labels may become identity-defining and self-limiting, as people come to believe their problems are enduring, <a href="https://www.sciencedirect.com/science/article/abs/pii/S0165032724002489?via%3Dihub">hard-to-control</a> aspects of who they are. Second, unwarranted self-diagnosis may lead people experiencing relatively mild levels of distress to seek help that is unnecessary, inappropriate and ineffective. Recent <a href="https://pubmed.ncbi.nlm.nih.gov/37844607/">Australian research</a> found people with relatively mild distress who received psychotherapy worsened more often than they improved. Third, these effects may be particularly problematic for young people. They are most liable to hold broad concepts of mental illness, in part due to <a href="https://www.sciencedirect.com/science/article/pii/S0010440X22000682?via%3Dihub">social media</a> <a href="https://www.tandfonline.com/doi/full/10.1080/10810730.2023.2235563">consumption</a>, and they experience mental ill health at relatively high and rising rates. Whether expansive concepts of illness play a role in the youth mental health crisis remains to be seen. Ongoing cultural shifts are fostering increasingly expansive definitions of mental illness. These shifts are likely to have mixed blessings. By normalising mental illness they may help to remove its stigma. However, by pathologising some forms of everyday distress, they may have an unintended downside. As we wrestle with the mental health crisis, it is crucial we find ways to increase awareness of mental ill health without inadvertently inflating it.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/231687/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/jesse-tse-1429151">Jesse Tse</a>, PhD Candidate at Melbourne School of Psychological Sciences, <a href="https://theconversation.com/institutions/the-university-of-melbourne-722">The University of Melbourne</a> and <a href="https://theconversation.com/profiles/nick-haslam-10182">Nick Haslam</a>, Professor of Psychology, <a href="https://theconversation.com/institutions/the-university-of-melbourne-722">The University of Melbourne</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/do-you-have-a-mental-illness-why-some-people-answer-yes-even-if-they-havent-been-diagnosed-231687">original article</a>. </div>

Mind

"I am the Bicycle Bandit": Terminally-ill ex-cop confesses to 20-year-old mystery

In a startling twist to a 20-year-old mystery, 73-year-old Kym Allen Parsons, a terminally-ill former police officer and firefighter, has admitted to being the notorious "Bicycle Bandit" who terrorised South Australian banks and residents for a decade. Parsons' confession came just days after receiving approval for voluntary assisted dying (VAD) and being provided with a VAD kit by SA Health. Parsons, who has stage 4 cancer and who had previously denied the charges, changed his plea to guilty during a Supreme Court session on Monday, ending years of speculation and investigation. His sudden admission of guilt follows a plea bargain brokered by the Office of the Director of Public Prosecutions and his counsel after the VAD approval was granted. In a tearful apology read to the court, Parsons expressed deep remorse for his actions, acknowledging that his behaviour was both irrational and without excuse. "I have no excuse for my behaviour," he told the court. "My reasoning was illogical and irrational over that time, and over the past 10 years I have tried to rehabilitate, seek help and forgiveness and demonstrate my shame in distressing actions. "I was fearful of confessing my past and destroying their [my wife and family's] love and trust in the person they knew. "I do not expect your forgiveness, and I humbly ask you accept my sincerest apology and deepest remorse." Despite Parsons' request for bail ahead of his sentencing, Justice Sandi McDonald deemed his crimes too severe for continued freedom and ordered his immediate custody. His access to the VAD kit while in custody remains uncertain. The courtroom was filled with Parsons' victims and their supporters, many of whom had worked at the banks he robbed. Some were victimised multiple times. One victim described the lasting impact of being robbed at gunpoint, detailing the immense trauma and the development of an auto-immune disease likely induced by stress. Other victims recounted struggles with PTSD, anxiety, depression, and ongoing trust issues. Parsons had been scheduled for trial in February on charges of armed robbery, attempted armed robbery, and firearms offences, with prosecutors alleging he stole over $250,000 from 11 banks between 2004 and 2014. DNA evidence was cited as a link to the crimes. His guilty plea and impending death are expected to ignite a legal battle over his $2.4 million estate, involving prosecutors, his heirs, and his victims. Previously, Parsons had been granted home detention due to his terminal stage 4 cancer diagnosis, after significant weight loss while in custody. His defence lawyer, James Marcus, stated that Parsons pleaded guilty to provide closure to the victims and their families. Parsons' sentencing is scheduled for June 28, marking the conclusion of a complex and emotional case that has gripped the state for years. Images: ABC News / SA Police

Legal

AFL great's son in induced coma after mystery brain infection

Geelong great Peter Riccardi has revealed his son, Osca, was briefly put on life support after suffering a mystery infection on the brain. Speaking on the podcast Beyond The Boundary, the former AFL player revealed that his son became suddenly ill a fortnight ago. “A couple of Sundays ago (Osca) came home, been out with a few of his mates, he’d been to the beach, went out for dinner, went out to play 10-pin bowling ... and said he was going to bed,” Peter Riccardi said. “Then halfway through the night he was up, he was vomiting, he was feeling a bit crook ... we just thought he was run down. “But come lunchtime, he couldn’t talk, he could hardly walk.” He added that they were extremely lucky his wife Mel worked from home that day and rushed Osca straight to hospital, where they found some "swelling" on his brain following a scan. Doctors also found that he had a sinus and ear infection and glandular fever all “rolled into one”. “Whether the swim did something with his ears and went into his brain, I’m not 100 per cent sure, yet,” Riccardi said. “They put him an induced coma for three days. He was in ICU (Intensive Care Unit) for four days. “But he’s back home now recovering ... you wouldn’t know that two weeks ago, watching him on life support, and seeing him now, it’s amazing what they do in there.” The podcast hosts then asked how scary the situation was for Riccardi and his wife, and he responded: “It was, yeah ... obviously they have got to prepare you for the worst (outcome)." “That was probably the worst thing to hear, because we didn’t know how he was going to come out of it. “But again, like I said, if Mel had gone to work that day, he wouldn’t be here today. “We’re pretty lucky, we’re pretty lucky. “It must have been a mother’s intuition or mother’s instinct to stay at home that day.” Image: Facebook/ Geelong Cats

Caring

Why it’s still a scientific mystery how some can live past 100 – and how to crack it

<div class="theconversation-article-body"><a href="https://theconversation.com/profiles/richard-faragher-224976">Richard Faragher</a>, <a href="https://theconversation.com/institutions/university-of-brighton-942">University of Brighton</a> and <a href="https://theconversation.com/profiles/nir-barzilai-1293752">Nir Barzilai</a>, <a href="https://theconversation.com/institutions/albert-einstein-college-of-medicine-3638">Albert Einstein College of Medicine</a> A 35-year-old man <a href="https://pubmed.ncbi.nlm.nih.gov/18544745/">only has a 1.5% chance of dying in the next ten years</a>. But the same man at 75 has a 45% chance of dying before he reaches 85. Clearly, ageing is bad for our health. On the bright side, we have made unprecedented progress in understanding the fundamental mechanisms that control ageing and late-life disease. A few tightly linked biological processes, sometimes called the <a href="https://pubmed.ncbi.nlm.nih.gov/23746838/">“hallmarks of ageing”</a>, including our supply of stem cells and communication between cells, act to keep us healthy in the early part of our lives – with <a href="https://theconversation.com/the-secret-to-staying-young-scientists-boost-lifespan-of-mice-by-deleting-defective-cells-54068">problems arising as these start to fail</a>. <a href="https://pubmed.ncbi.nlm.nih.gov/34699859/">Clinical trials are ongoing</a> to see if targeting some of these hallmarks can improve <a href="https://pubmed.ncbi.nlm.nih.gov/31542391/">diabetic kidney disease</a>, <a href="https://pubmed.ncbi.nlm.nih.gov/29997249/">aspects of</a> <a href="https://pubmed.ncbi.nlm.nih.gov/33977284/">immune function</a> and age-related <a href="https://pubmed.ncbi.nlm.nih.gov/30616998/">scarring of the lungs</a> among others. So far, so good. Unfortunately, big, unanswered questions remain in the biology of ageing. To evaluate what these are and how to address them, the <a href="https://www.afar.org/">American Federation For Aging Research</a>, a charity, recently convened a series of <a href="https://www.afar.org/imported/AFAR_GeroFuturesThinkTankReport_November2021.pdf">meetings for leading scientists and doctors</a>. The experts agreed that understanding what is special about the biology of humans who survive more than a century is now a key challenge. These centenarians <a href="https://www.statista.com/chart/18826/number-of-hundred-year-olds-centenarians-worldwide/">comprise less than 0.02% of the UK population</a> but have exceeded the life expectancy of their peers by almost 50 years (babies born in the 1920s typically had a life expectancy of less than 55). How are they doing it? We know that centenarians live so long because they are unusually healthy. They remain in good health for about 30 years longer than most normal people and when they finally fall ill, they are only sick for a very short time. This <a href="https://pubmed.ncbi.nlm.nih.gov/27377170/">“compression of morbidity”</a> is clearly good for them, but also benefits society as a whole. In the US, the medical care costs for a centenarian in their last two years of life <a href="https://www.cdc.gov/nchs/data/series/sr_10/sr10_198.pdf">are about a third of those of someone who dies in their seventies</a> (a time when most centenarians don’t even need to see a doctor). The children of centenarians are also much healthier than average, indicating they are inheriting something beneficial from their parents. But is this genetic or environmental? <h2>Centenarians aren’t always health conscious</h2> Are centenarians the poster children for a healthy lifestyle? For the general population, watching your weight, not smoking, drinking moderately and eating at least five servings of fruit and vegetables a day can <a href="https://pubmed.ncbi.nlm.nih.gov/27296932/">increase life expectancy by up to 14 years</a> compared with someone who does none of these things. This difference <a href="https://publications.parliament.uk/pa/ld5801/ldselect/ldsctech/183/18305.htm#_idTextAnchor012">exceeds that seen</a> between the least and most deprived areas in the UK, so intuitively it would be expected to play a role in surviving for a century. But astonishingly, this needn’t be the case. <a href="https://pubmed.ncbi.nlm.nih.gov/21812767/">One study</a> found that up to 60% of Ashkenazi Jewish centenarians have smoked heavily most of their lives, half have been obese for the same period of time, less than half do even moderate exercise and under 3% are vegetarians. The children of centenarians appear no more health conscious than the general population either. Compared to peers with the same food consumption, wealth and body weight, however, <a href="https://pubmed.ncbi.nlm.nih.gov/29050682/">they have half the prevalence of cardiovascular disease</a>. There is something innately exceptional about these people. <h2>The big secret</h2> Could it be down to rare genetics? If so, then there are two ways in which this could work. Centenarians might carry unusual genetic variants that extend lifespan, or instead they might lack common ones that cause late-life disease and impairment. Several studies, including our own work, <a href="https://pubmed.ncbi.nlm.nih.gov/32860726/">have shown</a> that centenarians have just as many bad genetic variants as the general population. Some even carry two copies of the largest known common risk gene for Alzheimer’s disease (APOE4), but still don’t get the illness. So a plausible working hypothesis is that centenarians carry rare, beneficial genetic variations rather than a lack of disadvantageous ones. And the best available data is consistent with this. Over 60% of centenarians have genetic changes that alter the genes which regulate growth in early life. This implies that these remarkable people are human examples of a type of lifespan extension observed in other species. Most people know that <a href="https://pubmed.ncbi.nlm.nih.gov/28803893/">small dogs tend to live longer than big ones</a> but fewer are aware that this is a general phenomenon across the animal kingdom. <a href="https://pubmed.ncbi.nlm.nih.gov/26857482/">Ponies can live longer than horses</a> and many strains of laboratory mice with dwarfing mutations <a href="https://pubmed.ncbi.nlm.nih.gov/29653683/">live longer than their full-sized counterparts</a>. One potential cause of this is reduced levels of a growth hormone called IGF-1 – although human centenarians <a href="https://pubmed.ncbi.nlm.nih.gov/28630896/">are not necessarily shorter than the rest of us</a>. Obviously, growth hormone is necessary early on in life, but there is increasing evidence that high levels of IGF-1 in mid to late life <a href="https://pubmed.ncbi.nlm.nih.gov/18316725/">are associated with increased late-life illness</a>. The detailed mechanisms underlying this remain an open question, but even among centenarians, women with the lowest levels of growth hormone <a href="https://pubmed.ncbi.nlm.nih.gov/24618355/">live longer than those with the highest</a>. They also have better cognitive and muscle function. That doesn’t solve the problem, though. Centenarians are also different from the rest of us in other ways. For example, they tend to have good cholesterol levels – hinting there may several reasons for their longevity. Ultimately, centenarians are “natural experiments” who show us that it is possible to live in excellent health even if you have been dealt a risky genetic hand and chose to pay no attention to health messages – but only if you carry rare, poorly understood mutations. Understanding exactly how these work should allow scientists to develop new drugs or other interventions that target biological processes in the right tissues at the right time. If these become a reality perhaps more of us than we think will see the next century in. But, until then, don’t take healthy lifestyle tips from centenarians.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/172020/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/richard-faragher-224976">Richard Faragher</a>, Professor of Biogerontology, <a href="https://theconversation.com/institutions/university-of-brighton-942">University of Brighton</a> and <a href="https://theconversation.com/profiles/nir-barzilai-1293752">Nir Barzilai</a>, Professor of Medicine and Genetics, <a href="https://theconversation.com/institutions/albert-einstein-college-of-medicine-3638">Albert Einstein College of Medicine</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/why-its-still-a-scientific-mystery-how-some-can-live-past-100-and-how-to-crack-it-172020">original article</a>. </div>

Retirement Life

Police appeal for help in solving discovery of mystery body

Last July, a farmer was clearing bushland on a property between Clifton Beach and Goats Beach in Tasmania, when he made a grisly discovery. He found skeletal remains along with a number of personal items believed to belong to the deceased. “Police attended and recovered the remains, which showed no signs of physical injuries,” Sergeant Lee Taylor said. The remains had been in the area between five and 50 years, according to police. “A forensic examination at the scene located several items with the remains, including a pair of black dress shoes with several store-bought orthotics in the heel, a short sleeve top and unreadable identification paperwork, and an old campsite nearby which may be related,” Taylor said. Forensic analysis of the remains found that they belonged to a Caucasian male , but no DNA matches were made, with police having to use facial reconstruction and releasing the image to the public, in hopes that someone could identify him. “With DNA taken from the remains not matching to any DNA comparisons, state or nationally, Australian Federal Police have provided a craniofacial reconstruction of the man’s potential appearance,” Taylor said. Anybody with information or who can identify the man is asked to contact the police. Images: 7News

Legal

3 common travel illnesses (and how to avoid them)

Nobody wants to fall sick when they’re on holidays but it happens and is actually quite common. Not every travel illness is foreseeable, but the most prevalent ones usually can be managed if you’re prepared and know what to look out for. Here are three of the most common illnesses travellers experience and what you can do to avoid them. Traveller’s diarrhoea It may be an unpleasant topic of conversation, but as diarrhoeais the most common travel sickness, it’s important to be prepared. It is estimated diarrhoeais experienced by almost half of travellers at some point on their holiday, but mainly by those visiting developing countries. It’s contracted by eating or drinking contaminated food and water and in severe cases can last for days. How to avoid it: Stick to bottled or purified water, freshly cooked meals and fruits and vegetables you can peel yourself. Talk to your doctor for antibiotics you can take in case you are struck with traveller’s diarrhoea. Motion sickness Whether it’s by boat, plane, or car, many travellers experience motion sickness. This occurs when your eyes see motion but your body doesn’t register it, leading to a conflict of the senses. It often results in nausea, vomiting, headaches, and sweating. How to avoid it: If flying, try to sit near the wings of plane. If cruising, get an outside cabin in the middle of ship, and if in a car, sit up front. Don’t play with your devices, as looking at a small screens often exacerbates the problem; instead try to look far to the horizon. Have a light meal before travelling and avoid spicy, greasy or rich foods. You can talk to your doctor about over-the-counter medication that can help motion sickness as well. Bug bites There are all sorts of infectious diseases like malaria, dengue, chikungunya and yellow fever you can pick up from bug bites, especially in developing nations. While you should always talk to your doctor about the types of vaccines you need to take for your travel destination, it is always advisable to protect against insect bites. How to avoid it: Apply insect repellent, wear long sleeves and pants where possible and try to avoid outside activity around dust and dawn when mosquitos are active. If sleeping outdoors, it is advisable to use curtain nettings. Image credits: Getty Images

Travel Tips

Families including someone with mental illness can experience deep despair. They need support

<div class="theconversation-article-body"> <a href="https://theconversation.com/profiles/amanda-cole-1484502">Amanda Cole</a>, <a href="https://theconversation.com/institutions/edith-cowan-university-720">Edith Cowan University</a> In the aftermath of the <a href="https://theconversation.com/bondi-attacker-had-mental-health-issues-but-most-people-with-mental-illness-arent-violent-227868">tragic Bondi knife attack</a>, Joel Cauchi’s parents have <a href="https://www.theguardian.com/australia-news/video/2024/apr/15/bondi-junction-stabbings-joel-cauchis-father-extremely-sorry-for-victims-video">spoken</a> about their son’s long history of mental illness, having been diagnosed with schizophrenia at age 17. They said they were “devastated and horrified” by their son’s actions. “To you he’s a monster,” said his father. “But to me he was a very sick boy.” Globally, one out of every eight people <a href="https://www.who.int/news-room/fact-sheets/detail/mental-disorders">report a mental illness</a>. In Australia, <a href="https://www.aihw.gov.au/reports/mental-health-services/mental-health">one in five people experience a mental illness</a> in their lifetime. Mental illness and distress affects not only the person living with the condition, but <a href="https://www.aihw.gov.au/reports/australias-health/chronic-conditions-and-multimorbidity">family members and communities</a>. As the prevalence of mental health problems grows, the flow-on effect to family members, including caregivers, and the impact on families as a unit, is also rising. While every family is different, the words of the Cauchis draw attention to how families can experience distress, stress, fear, powerlessness, and still love, despite the challenges and trauma. How can they help a loved one? And who can they turn to for support? <h2>The role of caregivers</h2> Informal caregivers help others <a href="https://www.aihw.gov.au/reports/australias-welfare/informal-carers">within the context of an existing relationship</a>, such as a family member. The care they provide goes beyond the usual expectations or demands of such relationships. Around <a href="https://www.aihw.gov.au/reports/australias-welfare/informal-carers">2.7 million Australians</a> provide informal care. For almost a third of these the person’s primary medical diagnosis is psychological or psychiatric. It has <a href="https://journals.sagepub.com/doi/10.1177/1074840708323598">long been acknowledged</a> that those supporting a family member with ongoing mental illness need support themselves. In the 1980s, interest grew in caregiving dynamics within families of people grappling with mental health issues. Subsequent research recognised <a href="http://www.aihw.gov.au/chronic-diseases/">chronic health conditions</a> not only affect the quality of life and wellbeing of the people experiencing them, but also impose burdens that reverberate within relationships, caregiving roles, and family dynamics over time. Past studies have shown families of those diagnosed with chronic mental illness are increasingly forced to <a href="https://pubmed.ncbi.nlm.nih.gov/24943714/">manage their own depression</a>, experience elevated levels of <a href="https://pubmed.ncbi.nlm.nih.gov/23692348/">emotional stress</a>, negative states of mind and <a href="https://pubmed.ncbi.nlm.nih.gov/21165597/">decreased overall mental health</a>. Conditions such as depression, anxiety disorders, bipolar disorder, and schizophrenia can severely impact daily functioning, relationships, and <a href="https://pubmed.ncbi.nlm.nih.gov/36875411/">overall quality of life</a>. Living with mental illness is often accompanied by a myriad of challenges. From stigma and discrimination to difficulty accessing adequate health care and support services. Patients and their families navigate a complex and often isolating journey. <h2>The family is a system</h2> The concept of <a href="http://apps.who.int/iris/bitstream/10665/40336/1/16937_eng.pdf">family health</a> acknowledges the physical and psychological wellbeing of a person is significantly affected by the family. Amid these challenges, <a href="https://www.frontiersin.org/journals/psychology/articles/10.3389/fpsyg.2024.1330720/full">family support</a> emerges as a beacon of hope. Research consistently demonstrates strong familial relationships and support systems play a pivotal role in mitigating the adverse effects of mental illness. Families provide emotional support, practical assistance, and a sense of belonging that are vital for people struggling with mental illness. My recent <a href="https://www.collegianjournal.com/article/S1322-7696(24)00004-0/fulltext">research</a> highlights the profound impact of mental illness on family dynamics, emphasising the resilience and endurance shown by participants. Families struggling with mental illness often experience heightened emotional fluctuations, with extreme highs and lows. The enduring nature of family caregiving entails both stress and adaptation over an extended period. Stress associated with caregiving and the demands on personal resources and coping mechanisms builds and builds. Yet families I’ve <a href="https://www.collegianjournal.com/article/S1322-7696(24)00004-0/fulltext">interviewed</a> find ways to live “a good life”. They prepare for the peaks and troughs, and show endurance and persistence. They make space for mental illness in their daily lives, describing how it spurs adaptation, acceptance and inner strength within the family unit. When treating a person with mental illness, health practitioners need to consider the entire family’s needs and engage with family members. By fostering open and early dialogue and providing comprehensive support, health-care professionals can empower families to navigate the complexities of mental illness while fostering resilience and hope for the future. Family members <a href="https://www.collegianjournal.com/article/S1322-7696(24)00004-0/fulltext">express stories</a> of an inner struggle, isolation and exhaustion. <h2>Shifting the focus</h2> There is a pressing need for a shift in research priorities, from illness-centered perspectives to a <a href="https://shop.elsevier.com/books/child-youth-and-family-health-strengthening-communities/barnes/978-0-7295-4155-8">strengths-based focus</a> when considering families “managing” mental illness. There is transformative potential in harnessing strengths to respond to challenges posed by mental illnesses, while also <a href="https://www.collegianjournal.com/article/S1322-7696(24)00004-0/fulltext">supporting family members</a>. For people facing mental health challenges, having <a href="https://www.sane.org/information-and-resources/facts-and-guides/families-friends-carers">loved ones who listen without judgement</a> and offer empathy can alleviate feelings of despair. Beyond emotional support, families often serve as crucial caregivers, assisting with <a href="https://www.blackdoginstitute.org.au/emergency-help/helping-someone-else/">daily tasks, medication management and navigating the health-care system</a>. As the Cauchi family so painfully articulated, providing support for a family member with mental illness is intensely challenging. Research <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4804270/">shows</a> caregiver burnout, financial strain and strained relationships are common. Health-care professionals should prioritise support for family members at an early stage. In Australia, there are various support options available for families living with mental illness. <a href="https://www.carergateway.gov.au/?utm_source=google&utm_medium=paid-search&utm_campaign=10626744435&utm_adgroup=102994881737&utm_term=carer%20gateway%20wa&gad_source=1&gclid=EAIaIQobChMIt8T6pJzIhQMVjAyDAx2KiQl1EAAYASAAEgLj-fD_BwE">Carer Gateway</a> provides information, support and access to services. <a href="https://www.headspace.com/?utm_source=google&utm_medium=search&utm_campaign=HS_Headspace_Brand-Exact_Search_AU-INT_Google_NA&utm_content=&utm_term=headspace&gad_source=1&gclid=EAIaIQobChMI4uKKvpzIhQMVFheDAx1bZgk8EAAYASAAEgLy6vD_BwE">Headspace</a> offers mental health services and supports to young people and their families. Beyond these national services, GPs, nurses, nurse practitioners and local community health centres are key to early conversations. Mental health clinics and hospitals often target family involvement in treatment plans. While Australia has made strides in recognising the importance of family support, challenges persist. Access to services can vary based on geographic location and demand, leaving some families under-served or facing long wait times. And the level of funding and resources allocated to family-oriented mental health support often does not align with the demand or complexity of need. In the realm of mental illness, family support serves as a lifeline for people navigating the complexities of their conditions. <hr /> If this article has raised issues for you, or if you’re concerned about someone you know, call <a href="https://www.lifeline.org.au/">Lifeline</a> on 13 11 14.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/228007/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/amanda-cole-1484502">Amanda Cole</a>, Lead, Mental Health, <a href="https://theconversation.com/institutions/edith-cowan-university-720">Edith Cowan University</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/families-including-someone-with-mental-illness-can-experience-deep-despair-they-need-support-228007">original article</a>. </div>

Caring

Leap of imagination: how February 29 reminds us of our mysterious relationship with time and space

<a href="https://theconversation.com/profiles/emily-ohara-874665">Emily O'Hara</a>, <a href="https://theconversation.com/institutions/auckland-university-of-technology-1137">Auckland University of Technology</a> If you find it intriguing that February 28 will be followed this week by February 29, rather than March 1 as it usually is, spare a thought for those alive in 1582. Back then, Thursday October 4 was followed by Friday October 15. Ten whole days were snatched from the present when Pope Gregory XIII issued a papal bull to “restore” the calendar from discrepancies that had crept into the Julian calendar, introduced by Julius Caesar in 45 BCE. The new Gregorian calendar returned the northern hemisphere’s vernal equinox to its “proper” place, around March 21. (The equinox is when the Earth’s axis is tilted neither toward nor away from the sun, and is used to determine the date of Easter.) The Julian calendar had observed a leap year every four years, but this meant time had drifted out of alignment with the dates of celestial events and astronomical seasons. In the Gregorian calendar, leap days were added only to years that were a multiple of four – like 2024 – with an exception for years that were evenly divisible by 100, but not 400 – like 1700. Simply put, leap days exist because it doesn’t take a neat 365 days for Earth to orbit the Sun. It takes 365.2422 days. Tracking the movement of celestial objects through space in an orderly pattern doesn’t quite work, which is why we have February – time’s great mop. <h2>Time and space</h2> This is just part of the history of how February – the shortest month, and originally the last month in the Roman calendar – came to have the job of absorbing those inconsistencies in the temporal calculations of the world’s most commonly used calendar. There is plenty of <a href="https://theconversation.com/leap-day-fixing-the-faults-in-our-stars-54032">science</a>, <a href="https://theconversation.com/explainer-the-science-behind-leap-years-and-how-they-work-54788">maths</a> and <a href="https://theconversation.com/how-a-seasonal-snarl-up-in-the-mid-1500s-gave-us-our-strange-rules-for-leap-years-132659">astrophysics</a> explaining the relationship between time and the planet we live on. But I like to think leap years and days offer something even more interesting to consider: why do we have calendars anyway? And what have they got to do with how we understand the wonder and strangeness of our existence in the universe? Because calendars tell a story, not just about time, but also about space. Our reckoning of time on Earth is through our spatial relationship to the Sun, Moon and stars. Time, and its place in our lives, sits somewhere between the scientific, the celestial and the spiritual. It is <a href="https://shop.whitechapelgallery.org/products/time">notoriously slippery, subjective and experiential</a>. It is also marked, tracked and determined in myriad ways across different cultures, from tropical to solar to <a href="https://www.stuff.co.nz/pou-tiaki/300062097/matariki-and-the-maramataka-the-mori-lunar-calendar">lunar</a> calendars. It is the Sun that measures a day and gives us our first reference point for understanding time. But it is the <a href="https://librarysearch.aut.ac.nz/vufind/Record/1145999?sid=25214690">Moon</a>, as a major celestial body, that extends our perception of time. By stretching a span of one day into something longer, it offers us a chance for philosophical reflection. The Sun (or its effect at least) is either present or not present. The Moon, however, goes through phases of transformation. It appears and disappears, changing shape and hinting that one night is not exactly like the one before or after. The Moon also has a distinct rhythm that can be tracked and understood as a pattern, giving us another sense of duration. Time is just that – overlapping durations: instants, seconds, minutes, hours, days, weeks, months, years, decades, lifetimes, centuries, ages. <h2>The elusive Moon</h2> It is almost impossible to imagine how time might feel in the absence of all the tools and gadgets we use to track, control and corral it. But it’s also hard to know what we might do in the absence of time as a unit of productivity – a measurable, dispensable resource. The closest we might come is simply to imagine what life might feel like in the absence of the Moon. Each day would rise and fall, in a rhythm of its own, but without visible reference to anything else. Just endless shifts from light to dark. Nights would be almost completely dark without the light of the Moon. Only stars at a much further distance would puncture the inky sky. The world around us would change – trees would grow, mammals would age and die, land masses would shift and change – but all would happen in an endless cycle of sunrise to sunset. The light from the Sun takes <a href="https://www.skyatnightmagazine.com/space-science/how-take-light-from-sun-reach-earth">eight minutes</a> to reach Earth, so the sunlight we see is always eight minutes in the past. I remember sitting outside when I first learned this, and wondering what the temporal delay might be between me and other objects: a plum tree, trees at the end of the street, hills in the distance, light on the horizon when looking out over the ocean, stars in the night sky. Moonlight, for reference, takes about <a href="https://www.pbs.org/seeinginthedark/astronomy-topics/light-as-a-cosmic-time-machine.html">1.3 seconds</a> to get to Earth. Light always travels at the same speed, it is entirely constant. The differing duration between how long it takes for sunlight or moonlight to reach the Earth is determined by the space in between. Time on the other hand, is anything but constant. There are countless ways we characterise it. The mere fact we have so many calendars and ways of describing perceptual time hints at our inability to pin it down. Calendars give us the impression we can, and have, made time predictable and understandable. Leap years, days and seconds serve as a periodic reminder that we haven’t.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/224503/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/emily-ohara-874665">Emily O'Hara</a>, Senior Lecturer, Spatial Design + Temporary Practices, <a href="https://theconversation.com/institutions/auckland-university-of-technology-1137">Auckland University of Technology</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/leap-of-imagination-how-february-29-reminds-us-of-our-mysterious-relationship-with-time-and-space-224503">original article</a>.

Technology

"I'm lowkey dying": Brave young woman with terminal illness shares her final wish

Samantha Bulloch was given three years to live after she was diagnosed with gut-wrenching stage four bowel cancer at the young age of 28. A year later, Bulloch has shared a heartfelt plea on social media in hopes of meeting her idol- pop star Taylor Swift. The Swiftie has scored a ticket to Taylor's final show in Sydney on the 26th of February, but she’s calling on “anyone to hook a sister up” so she can meet-and-greet the singer backstage. “I’m low key dying and honestly this would just make my year,” she said in a video shared to TikTok. “I’m going out on a limb here so I’m just shooting my shot and we’re going to see what happens. “If anyone has any connections... I would love you forever.” Bulloch has been a fan of the megastar since she was 15 years old. “Taylor means so much to me, and I’d love the opportunity to tell her just how much of an impact she’s made on my life,” she told 7Life. “I’ve loved her since I was 15, and her music has seen me through so many chapters in my life — including this one. “I love that her music transcends all kinds of walks of life, and so many of us connect with it so personally, despite the differences in our situations. “She has a real talent for making you feel less alone.I recently got a new tattoo of the lyric, ‘For the hope of it all’, from her song called August. “I adopted that lyric during my experience with cancer. I’m choosing to live for the hope of it all.” As she faces terminal cancer, Bulloch said that she is determined to live the rest of her life to the fullest. "I’m hoping and praying for many more years than what I’ve been given. But if not, I intend to try and maximise these few I’ve got left to the best of my ability," she said. “Thankfully I’ve always been quite a positive and hopeful person, and that hasn’t left me during this experience.” Bulloch was diagnosed with terminal cancer in 2023, after experiencing low iron levels, fatigue and blood in her stool. She is currently on a chemotherapy regime and an immunotherapy drug and added that she also hopes to tick off many of her bucket list destinations this year, including visiting UK, Paris, New York and Tasmania. “My doctor has said I can, providing the treatment I’m on now works," the hopeful 29-year-old said. “Thankfully treatment has been working so hopefully in a few months I’ll be able to do that." Images: Samantha Bulloch

Caring

Woman heartlessly steals terminally-ill man's pokies win after he collapsed

Thomas Purtill, 80, had “decided to go up and throw a few dollars in the pokies" at his local pub in Caboolture, Brisbane. To his surprise, the pensioner won $300. As the terminally-ill pensioner was about to collect his winnings, he collapsed during a medical episode and dropped his ticket. “I remember landing on the floor,” Purtill, who only has six months to live, told 7News. “I think I fell over before I got the ticket into the machine (to cash it in).” CCTV footage from the pub showed the alleged thief looking concerned as she rushed to call pub staff. When she returned, Purtill can be seen pointing to something by the machine, and this was when she noticed the ticket he had dropped. The woman then moved over to the machine and allegedly pocketed the pensioner's winnings. While Purtill received medical attention, the alleged thief escaped and used another machine to withdraw the cash. Now, the CCTV footage has been released to help police identify the mystery woman, as they urge anyone with information to come forward. The pensioner spend two days in hospital after the incident on November 27, and has since been diagnosed with terminal cancer. “I don’t want to die,” he said. “But I’m going to.” He was told he has between three to six months to live, and has since returned for treatment. When Purtill was asked what he thought of the alleged thief's actions he said: “I can’t really say on TV. It wouldn’t be allowed.” “If you’re that desperate … If someone came up to me and said, ‘You’ve had a win, can I have $10 to buy a loaf of bread, a bottle of milk for the kids?’, I would have given it to them.” He then went on to call her "weak", and urged anyone who might know her to “give her up”. Staff members have offered to reimburse Purtill for his stolen winnings. Images: 7News

Legal

"Do you hear it?": Worldwide hum global mystery baffles scientists

A perplexing phenomenon known as "The Worldwide Hum" has been capturing the attention of scientists and citizens alike, as an unusual low-frequency noise continues to puzzle experts. This mysterious hum, first recorded in 2012, has been reported by thousands of people worldwide, sparking investigations, online discussions and even <a href="https://www.thehum.info/" target="_blank" rel="noopener">the creation of an interactive map</a> documenting instances of the enigmatic sound. As researchers strive to unravel the mystery, individuals share their experiences, raising questions about its origin and effects. Described as a low rumbling or droning sound, "the hum" is often likened to the idling of a car or truck engine. What makes this phenomenon particularly intriguing is that it is not universally heard, with reports of the hum being exclusive to certain individuals. Some claim it is more pronounced at night than during the day, and louder indoors than outdoors. One Reddit user even compared it to the low-frequency vibrations felt when a passenger jet flies overhead. Since its first documentation, more than 6,500 instances of the hum have been reported globally, with new cases continually emerging. The interactive user-generated World Hum Map and Database Project captures the experiences of those who have encountered the sound, providing a comprehensive overview of its widespread occurrence. In some regions, authorities such as the Environment Protection Authority (EPA) have conducted investigations, as was the case in the NSW Waverley Council ten years ago. Despite these efforts, the source of the hum remains elusive. Individuals affected by the mysterious noise often find solace in online communities, where they share their experiences and discuss possible explanations. Some describe feeling as though they are "going insane", and say that the psychological impact of the persistent hum is actually very severe. Facebook support groups have become a platform for individuals to connect, share anecdotes and speculate about the origin of the sound. Theories range from the mundane – such as the use of headphones causing collective tinnitus – to more complex environmental factors. While tinnitus, a symptom of auditory system issues, has been proposed as a potential explanation, it does not account for the collective experience of the hum. Various theories, including industrial plants, ocean waves, lightning strikes and the proliferation of mobile phone towers, have been suggested over the years. However, none of these explanations have gained widespread acceptance or provided a conclusive answer. Dr Glen MacPherson, who initiated the World Hum Map and Database Project, experienced the hum firsthand on Canada's Sunshine Coast. Having debunked the idea of "hum hotspots", Dr MacPherson theorises that the hum may be a subjective phenomenon, akin to tinnitus, originating from within the individual rather than an external source. His 11 years of research highlight the complexity of the mystery, challenging initial assumptions and pointing towards the need for further investigation. As "The Worldwide Hum" continues to captivate the curiosity of scientists and citizens worldwide, the quest for understanding remains elusive. While theories abound, the true origin of the hum remains unknown, leaving both experts and individuals alike intrigued by a phenomenon that transcends geographic boundaries and defies conventional explanations. Image: Getty

Body

Hunt for Cinderella! Mystery shoe left at Prince Christian's party sparks search

A mystery shoe left at Prince Christian's <a href="https://oversixty.com.au/lifestyle/family-pets/unseen-pics-of-prince-christian-mark-his-18th-birthday" target="_blank" rel="noopener">18th birthday party</a> inside Christianborg Palace has sparked a search for a real life Cinderella. In the hours after the ball, which saw royalty from around the world attend, the Danish royal household posted a photo of the gold stiletto that was left behind from one of the high profile party guests. The post read, "Is it Cinderella who forgot her shoe last night?" The caption continued, "When the guests at Her Majesty the Queen's gala dinner yesterday had gone home, this lonely stiletto shoe was left at Christiansborg Castle." "The owner is welcome to contact you to get it back." <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-permalink="https://www.instagram.com/p/CydeENrNum2/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/p/CydeENrNum2/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by DET DANSKE KONGEHUS 🇩🇰 (@detdanskekongehus)</a> </div> </blockquote> The ball was attended by Crown Princess Mary and Crown Prince Frederik, along with Christian's younger siblings Princess Isabella, 16, Prince Vincent and Princess Josephine, both 12. His uncle Prince Joachim, who relocated to America in August, was also there with his wife Princess Marie and their three youngest children Count Felix, 21, Count Henrik, 14, and Countess Athena, 12. A number of future monarchs were also present including royals from Sweden, Norway, the Netherlands and Belgium. Thankfully, the owner of the shoe was found, with Anne-Sofie Tørnsø Olesen, from Denmark's Egedal region, coming forward to claim the golden stiletto. And it turns out, she left it at the palace on purpose after being inspired by the story of Cinderella who marries her prince after long search. "I thought it was a bit funny myself, and I talked to my family and friends about it before, and they agreed that I should do it," Tørnsø Olesen, 18, told local Danish publication Se & Hør. "It's such a chance you won't get again." She said she was keen to get the shoe back because it was "a memory from a great evening". The lost shoe, by Danish brand Deichmann's Catwalk collection, sparked an immediate flurry of comments on the royal family's Instagram page. The shoe brand said, "If the princess comes from a long way, we will gladly give her a new pair". Image credits: Getty Images / Instagram

Beauty & Style

Bec Judd's son taken to hospital with mystery illness

Bec Judd's seven-year-old son, Tom, has been hospitalised due to an undisclosed medical condition, as the concerned mother shared on her Instagram account. In the recent post, the 40-year-old WAG posted a heartwarming picture of Tom comfortably lying in a hospital bed. In the photo, Tom flashed a smile at the camera, clutching his teddy bear and savouring an ice cream treat. However, Bec did not reveal the specific reason for Tom's hospitalisation in her social media update. With a hopeful tone, Bec wrote, "Onwards and upwards, Tom," and expressed gratitude to Nurse Becky Bell for her care and support during this challenging time. Bec Judd, a mother of four, shares her children with her ex-AFL star husband, Chris Judd. Alongside her recent family concerns, Bec has also been actively involved in charitable endeavours. She disclosed her plans to contribute 25,000 meals to Food Bank Victoria, an organisation dedicated to addressing food insecurity. Bec's involvement extends beyond her vocal support, as she is using her design skills to create a unique spatula for fundraising purposes. The entirety of the proceeds from the spatula sales will be donated to Food Bank. In Bec's own words, each spatula sold translates into an impressive "25 meals on the table", with the ambitious goal of providing "250,000 extra meals by Christmas". She encouraged her substantial following to support the cause by considering the spatula as a unique Christmas gift option. Not only will buyers acquire an original Bec Judd creation, but they will also make a significant contribution to their community. This charitable campaign features other notable personalities, including New Zealand culinary expert Ben Shewry, radio and TV presenter Chrissie Swan, and Australian pastry chef Kirsten Tibballs. In addition to her philanthropic efforts, Bec Judd has been making appearances on the celebrity edition of The Amazing Race, where she teams up with her sister, Kate Twigley. During a recent interview on KIIS FM's Jase and Lauren show, Bec recounted some of the show's more challenging moments, including an encounter with elephant dung. "It was interesting; they really threw us into these challenges that were quite foul, like shovelling poo, and I was like, where are the gloves?" Bec shared. "You know, I worked at The Alfred Hospital; I have four kids, I've got a puppy. I'm used to vomit and poo and secretions, but we always had gloves and masks, so it was okay. But this is bare hands, and I was not happy about it." In a recent trailer for the Ten reality show, Bec also revealed her germophobic tendencies, adding a layer of intrigue to her adventures on The Amazing Race. Images: Instagram

Caring

Cancer is rising in under-50s – but the causes are a mystery

<a href="https://theconversation.com/profiles/ashleigh-hamilton-1468163">Ashleigh Hamilton</a>, <a href="https://theconversation.com/institutions/queens-university-belfast-687">Queen's University Belfast</a> Cancer is often thought of as a disease that mostly affects older people. But worrying new research shows that cancer in younger adults is a growing problem. The study found there’s been a nearly 80% increase in the number of under-50s being <a href="https://bmjoncology.bmj.com/content/2/1/e000049">diagnosed with cancer</a> globally in the last three decades. Also of concern are the types of cancers being seen in younger adults – with this latest study and previous research showing that cancers thought of as typical of older age groups are now increasingly being diagnosed in younger people. These include <a href="https://pubmed.ncbi.nlm.nih.gov/31105047/">bowel cancer</a>, <a href="https://pubmed.ncbi.nlm.nih.gov/31331685/">stomach cancer</a>, <a href="https://pubmed.ncbi.nlm.nih.gov/32144720/">breast cancer</a>, <a href="https://pubmed.ncbi.nlm.nih.gov/30733056/">uterine cancer</a> and <a href="https://pubmed.ncbi.nlm.nih.gov/35053447/">pancreatic cancer</a>. This is worrying because some of these cancers – particularly <a href="https://www.cancerresearchuk.org/about-cancer/pancreatic-cancer/survival">pancreatic</a> and <a href="https://www.cancer.org/cancer/types/stomach-cancer/detection-diagnosis-staging/survival-rates.html">stomach</a> cancer – have low survival rates, due to the fact they’re often diagnosed at a late stage. Research has also shown that bowel cancer tends to be <a href="https://pubmed.ncbi.nlm.nih.gov/29564176/">diagnosed at a more advanced stage</a> in young people compared with older adults. While it’s clear from this latest study that cancer is becoming more common in those under 50, experts still aren’t entirely sure what’s causing this rise. <h2>Early-onset cancer</h2> The study investigated cancer cases in people under the age of 50 (termed “early-onset cancer”) from 204 countries and regions. The data analysed was collected between 1990 and 2019. The researchers were interested in knowing not only the incidence of early-onset cancer, but what types of cancer had the highest burden in under-50s. They found that in 2019, there were 3.26 million cases of early-onset cancer diagnosed worldwide – a 79% increase since 1990. The authors also predicted that by 2030, the number of under-50s diagnosed with cancer would increase by a further 31%. Breast cancer was the most common early-onset cancer in 2019, but incidences of prostate and throat cancers increased at the fastest rate since 1990. Liver cancer decreased the fastest over the same time period. The number of deaths due to early-onset cancers also increased from 1990 to 2019 – although less quickly than the rate of diagnosis, with 1.06 million deaths worldwide in 2019, an increase of 28%. The cancers with the highest number of deaths in 2019 were breast, lung, bowel and stomach cancers. The age group at greatest risk of early-onset cancer were those in their 40s. In 2019, early-onset breast cancer had the highest burden for women, while early-onset lung cancer the highest burden for men. Women were disproportionately affected in terms of death and poor health from early-onset cancer in low- and middle-income countries. The study also shows that while the highest number of early-onset cancer cases were in developed countries such as western Europe, North America and Australasia, many cases were also seen in low- and middle-income countries. Death rates were also higher in low- and middle-income countries. The main limitation of this paper is the variability of the data collected by different countries, making it difficult to measure its completeness. Nonetheless, it is still useful in getting a picture of global health. <h2>Unknown causes</h2> There’s no single explanation for why cancers are rising in under-50s. Some cancers in younger people happen as a result of a genetic condition – but these only <a href="https://aacrjournals.org/cancerres/article/80/16_Supplement/1122/641186">account for a small number of cases</a> (around 20%). Lifestyle factors such as the foods we eat, whether we drink alcohol or smoke, and being overweight are all linked to an <a href="https://www.cancer.gov/about-cancer/causes-prevention/risk">increased risk</a> of many types of cancer. Research indicates that these factors may be contributing to a rise in <a href="https://pubmed.ncbi.nlm.nih.gov/33524598/">early-onset colorectal cancer</a>, for example. Whether this is true for other types of early-onset cancer remains unknown. Some people affected by early-onset cancers may live healthy lifestyles. This suggests there are probably other reasons for the increase that have not yet been discovered. It’s clear from this research that the landscape of cancer is changing. While the incidence of early-onset cancers is increasing, cancer in this age group is still much less common than for those over-50. Early-onset cancers account for only around a tenth of <a href="https://www.cancerresearchuk.org/health-professional/cancer-statistics/incidence/age">new cases in the UK</a>. But though the numbers are still relatively low, this doesn’t mean the trend we’re seeing isn’t of concern. It will be crucial now to ensure there’s greater awareness of early-onset cancers. Most younger people, and even healthcare professionals, don’t necessarily put cancer at the top of the list when symptoms develop. It’s important for people to see their GP if they notice any new symptoms, as detecting cancer at an early stage leads to a better prognosis. Urgent research into early-onset cancer is also needed at a national and international level. The underlying causes are probably different depending on a person’s sex, ethnicity and where they live. On a personal level, there are many things you can do to reduce your risk of developing cancer. <a href="https://www.cancerresearchuk.org/about-cancer/causes-of-cancer/can-cancer-be-prevented">Following a healthy lifestyle</a> remains important. This includes eating a healthy diet, stopping smoking, exercising regularly, reducing your alcohol intake, being safe in the sun and maintaining a healthy weight. If something doesn’t feel right with your body or you experience any new symptoms, it’s important to see a doctor as soon as you can.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/212834/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/ashleigh-hamilton-1468163">Ashleigh Hamilton</a>, Academic Clinical Lecturer, Centre for Public Health, <a href="https://theconversation.com/institutions/queens-university-belfast-687">Queen's University Belfast</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/cancer-is-rising-in-under-50s-but-the-causes-are-a-mystery-212834">original article</a>.

Body

Terminally ill teacher convicted of child abuse granted end-of-life permit

A convicted child abuser from Adelaide, who was imprisoned for his acts of paedophilia against students during his tenure as a music teacher, has been authorised to pursue assisted dying, according to an exclusive report by <a href="https://www.9news.com.au/national/exclusive-adelaide-news-jailed-paedophile-teacher-malcolm-day-given-end-of-life-permit-voluntary-assisted-dying/cab7e95c-f3b1-4dbd-ae0d-cc8dbfee22c0" target="_blank" rel="noopener">9News</a>. Malcolm Day, aged 81, has emerged as the first incarcerated individual in Australia to receive approval for voluntary assisted dying following a terminal illness diagnosis, reportedly linked to cancer. Having received a 20-year prison sentence last June, Day's remaining term spans 17 years. Navigating the 11-step process required for accessing voluntary assisted dying in South Australia, Day's application is reported to be in its concluding stages, potentially reaching completion within the next few days. Dr Philip Nitschke, the director of the pro-euthanasia organisation Exit International, acknowledged that an incarcerated individual availing themselves of this scheme was an inevitable eventuality. "By the sounds of it, he satisfies all the conditions of the South Australian assisted dying legislation," Dr Nitschke told 9News. "So there should be no impediment… he should be given the option that any other person would have if they were terminally ill." During the 1980s, Day, while serving as a music teacher in South Australia, inflicted profound and lasting harm upon two of his students. After grooming and exploiting his victims, he vehemently refuted all allegations when investigated by educational authorities. When Day was sentenced, his legal representative, Stephen Ey, acknowledged the real possibility of his client passing away behind bars, saying at the time that it was "a real prospect... given his age." According to the latest data from SA Health, since the initiation of voluntary assisted dying in January of this year, 39 terminally ill residents of South Australia have opted to peacefully conclude their lives after being granted the necessary permits. Image: Nine News

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Cancer is rising in under-50s – but the causes are a mystery

Terminally ill teacher convicted of child abuse granted end-of-life permit

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