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For type 2 diabetes, focusing on when you eat – not what – can help control blood sugar

<div class="theconversation-article-body"> <a href="https://theconversation.com/profiles/evelyn-parr-441878">Evelyn Parr</a>, <a href="https://theconversation.com/institutions/australian-catholic-university-747">Australian Catholic University</a> and <a href="https://theconversation.com/profiles/brooke-devlin-2237174">Brooke Devlin</a>, <a href="https://theconversation.com/institutions/the-university-of-queensland-805">The University of Queensland</a> Type 2 diabetes affects <a href="https://www.aihw.gov.au/reports/diabetes/diabetes/contents/how-common-is-diabetes/type-2-diabetes">1.2 million Australians</a> and accounts for <a href="https://www.diabetesaustralia.com.au/about-diabetes/type-2-diabetes/">85-90%</a> of all diabetes cases. This chronic condition is characterised by high blood glucose (sugar) levels, which carry serious <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(17)30058-2/abstract">health</a> risks. <a href="https://www.nature.com/articles/nrendo.2017.151">Complications</a> include heart disease, kidney failure and vision problems. Diet is an important way people living with type 2 diabetes manage blood glucose, alongside exercise and medication. But while we know individualised, professional dietary advice improves blood glucose, it can be <a href="https://linkinghub.elsevier.com/retrieve/pii/S0168822717317588">complex</a> and is not always <a href="https://www.publish.csiro.au/py/PY13021">accessible</a>. <a href="https://www.sciencedirect.com/science/article/pii/S0168822724008039">Our new study</a> looked at the impact of time-restricted eating – focusing on when you eat, rather than what or how much – on blood glucose levels. We found it had similar results to individualised advice from an accredited practising dietitian. But there were added benefits, because it was simple, achievable, easy to stick to – and motivated people to make other positive changes. <h2>What is time-restricted eating?</h2> Time-restricted eating, also known as <a href="https://www.annualreviews.org/content/journals/10.1146/annurev-nutr-082018-124320">the 16:8 diet</a>, became popular for weight loss around 2015. Studies have since shown it is also an <a href="https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2811116">effective way</a> for people with type 2 diabetes to manage blood glucose. Time-restricted eating involves limiting when you eat each day, rather than focusing on what you eat. You restrict eating to a window during daylight hours, for example between 11am and 7pm, and then fast for the remaining hours. This can sometimes naturally lead to also eating less. Giving your body a break from constantly digesting food in this way helps align eating with natural <a href="https://doi.org/10.1111/jne.12886">circadian rhythms</a>. This <a href="https://doi.org/10.1111/jnc.15246">can help</a> regulate metabolism and improve overall health. For people with type 2 diabetes, there may be specific benefits. They often have their <a href="https://doi.org/10.2337/dc12-2127">highest blood glucose</a> reading in the morning. Delaying breakfast to mid-morning means there is time for physical activity to occur to help reduce glucose levels and prepare the body for the first meal. <h2>How we got here</h2> We ran an <a href="https://www.mdpi.com/2072-6643/12/11/3228">initial study</a> in 2018 to see whether following time-restricted eating was achievable for people with type 2 diabetes. We found participants could easily stick to this eating pattern over four weeks, for an average of five days a week. Importantly, they also had improvements in blood glucose, spending less time with high levels. <a href="https://www.mdpi.com/2072-6643/12/2/505">Our previous research</a> suggests the reduced time between meals may play a role in how the hormone insulin is able to reduce glucose concentrations. <a href="https://doi.org/10.1001/jamanetworkopen.2023.39337">Other studies</a> have confirmed these findings, which have <a href="https://doi.org/10.1186/s12986-021-00613-9">also shown</a> notable improvements in HbA1c. This is a <a href="https://www.ncbi.nlm.nih.gov/books/NBK304271/">marker</a> in the blood that represents concentrations of blood glucose over an average of three months. It is the <a href="https://journals.sagepub.com/doi/10.4137/BMI.S38440">primary clinical tool</a> used for diabetes. However, these studies provided intensive support to participants through weekly or fortnightly meetings with researchers. While we know this level of support <a href="https://www.nature.com/articles/0802295">increases</a> how likely people are to stick to the plan and improves outcomes, it is not readily available to everyday Australians living with type 2 diabetes. <h2>What we did</h2> In our <a href="https://www.sciencedirect.com/science/article/pii/S0168822724008039">new study</a>, we compared time-restricted eating directly with advice from an <a href="https://dietitiansaustralia.org.au/working-dietetics/standards-and-scope/role-accredited-practising-dietitian">accredited practising dietitian</a>, to test whether results were similar across six months. We recruited 52 people with type 2 diabetes who were currently managing their diabetes with up to two oral medications. There were 22 women and 30 men, aged between 35 and 65. Participants were randomly divided into two groups: diet and time-restricted eating. In both groups, participants received four consultations across the first four months. During the next two months they managed diet alone, without consultation, and we continued to measure the impact on blood glucose. In the diet group, consultations focused on changing their diet to control blood glucose, including improving diet quality (for example, eating more vegetables and limiting alcohol). In the time-restricted eating group, advice focused on how to limit eating to a nine-hour window between 10am and 7pm. Over six months, we measured each participant’s blood glucose levels every two months using the HbA1c test. Each fortnight, we also asked participants about their experience of making dietary changes (to what or when they ate). <h2>What we found</h2> We found time-restricted eating was as effective as the diet intervention. Both groups had reduced blood glucose levels, with the greatest improvements occurring after the first two months. Although it wasn’t an objective of the study, some participants in each group also lost weight (5-10kg). When surveyed, participants in the time-restricted eating group said they had adjusted well and were able to follow the restricted eating window. Many told us they had family support and enjoyed earlier mealtimes together. Some also found they slept better. After two months, people in the time-restricted group were looking for more dietary advice to further improve their health. Those in the diet group were less likely to stick to their plan. Despite similar health outcomes, time-restricted eating seems to be a simpler initial approach than making complex dietary changes. <h2>Is time-restricted eating achievable?</h2> The main barriers to following time-restricted eating are social occasions, caring for others and work schedules. These factors may prevent people eating within the window. However, there are many benefits. The message is simple, focusing on when to eat as the main diet change. This may make time-restricted eating more translatable to people from a wider variety of socio-cultural backgrounds, as the types of foods they eat don’t need to change, just the timing. Many people don’t have access to more individualised support from a dietitian, and receive nutrition advice from their GP. This makes time-restricted eating an alternative – and equally effective – strategy for people with type 2 diabetes. People should still try to stick to <a href="https://www.eatforhealth.gov.au/guidelines/guidelines">dietary guidelines</a> and prioritise vegetables, fruit, wholegrains, lean meat and healthy fats. But our study showed time-restricted eating may also serve as stepping stone for people with type 2 diabetes to take control of their health, as people became more interested in making diet and other positive changes. Time-restricted eating might not be appropriate for everyone, especially people on medications which don’t recommend fasting. Before trying this dietary change, it’s best speak to the healthcare professional who helps you manage diabetes.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/241472/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/evelyn-parr-441878">Evelyn Parr</a>, Research Fellow in Exercise Metabolism and Nutrition, Mary MacKillop Institute for Health Research, <a href="https://theconversation.com/institutions/australian-catholic-university-747">Australian Catholic University</a> and <a href="https://theconversation.com/profiles/brooke-devlin-2237174">Brooke Devlin</a>, Lecturer in Nutrition and Dietetics, School of Human Movement and Nutrition Sciences, <a href="https://theconversation.com/institutions/the-university-of-queensland-805">The University of Queensland</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/for-type-2-diabetes-focusing-on-when-you-eat-not-what-can-help-control-blood-sugar-241472">original article</a>. </div>

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Rare coin hidden for decades to fetch eye-watering sum

Three sisters from the US who inherited a dime coin kept it in a bank vault for more than 40 years, and while they know it had some value, they didn't know just how much until a few years ago. The rare coin, struck by the US Mint in San Francisco in 1975, could be worth more than $US500,000 ($748,000), according to Ian Russell, president of GreatCollections, the auction house selling the coin. What makes the coin depicting President Franklin D. Roosevelt so valuable is that it is just one of two coins missing the "S" mint mark for San Francisco. The other dime sold for $US682,000 (over $1 million) at a 2019 auction and then again months later to a private collector. While avid coin collectors have known about the existence of these two extraordinarily rare coins, their whereabouts had remained a mystery since the late 1970s. “They were hidden for decades,” Russell said. “Most major collectors and dealers have never seen one.” The three sisters from Ohio, who want to remain anonymous, inherited one of the two dimes after the recent death of their of their brother, Russell said. They told Russell that their brother and mother bought the first error coin discovered in 1978 for $27,225, which would amount to roughly $135,000 today. Their parents, who ran a dairy farm, saw the coin as a financial safety net, and it was only until last year that one of the sisters saw the coin first-hand. Russell also said that their brother had reached out to him about seven years ago and told him about the coin, but he too kept it a secret. When Russell told one of the sisters about the potential value of the coin, she told him: “is that really possible?”. The coin, known as the “1975 ‘no S’ proof dime,” will be displayed at a coin show beginning on Wednesday in Tampa, Florida, and before <a href="https://www.greatcollections.com/Coin/1655587" target="_blank" rel="noopener">the auction</a> closes late next month, Russell said. The current highest bidder has offered $US250,000 ($374,000). Images: Great Collections/ Professional Coin Grading Services

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What is type 1.5 diabetes? It’s a bit like type 1 and a bit like type 2 – but it’s often misdiagnosed

<div class="theconversation-article-body"> <a href="https://theconversation.com/profiles/emily-burch-438717">Emily Burch</a>, <a href="https://theconversation.com/institutions/southern-cross-university-1160">Southern Cross University</a> and <a href="https://theconversation.com/profiles/lauren-ball-14718">Lauren Ball</a>, <a href="https://theconversation.com/institutions/the-university-of-queensland-805">The University of Queensland</a> While you’re likely familiar with type 1 and type 2 diabetes, you’ve probably heard less about type 1.5 diabetes. Also known as latent autoimmune diabetes in adults (LADA), type 1.5 diabetes has features of <a href="https://diabetesjournals.org/diabetes/article/69/10/2037/16062/Management-of-Latent-Autoimmune-Diabetes-in-Adults">both type 1 and type 2 diabetes</a>. More people became aware of this condition after <a href="https://www.imdb.com/name/nm0004726/">Lance Bass</a>, best known for his role in the iconic American pop band NSYNC, <a href="https://www.foxnews.com/health/pop-singer-lance-bass-type-1-5-diabetes-know-disease">recently revealed</a> he has it. So, what is type 1.5 diabetes? And how is it diagnosed and treated? <h2>There are several types of diabetes</h2> Diabetes mellitus is a group of conditions that arise when the levels of glucose (sugar) in our blood are higher than normal. There are actually <a href="https://diabetesjournals.org/care/article/37/Supplement_1/S81/37753/Diagnosis-and-Classification-of-Diabetes-Mellitus">more than ten types</a> of diabetes, but the <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(23)01301-6/fulltext">most common</a> are type 1 and type 2. Type 1 diabetes is an <a href="https://diabetesjournals.org/care/article/44/11/2589/138492/The-Management-of-Type-1-Diabetes-in-Adults-A">autoimmune condition</a> where the body’s immune system attacks and destroys the cells in the pancreas that make the hormone insulin. This leads to very little or no insulin production. Insulin is important for moving glucose from the blood into our cells to be used for energy, which is why people with type 1 diabetes need <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6995794/">insulin medication daily</a>. Type 1 diabetes usually <a href="https://www.nature.com/articles/s41390-024-03107-5">appears</a> in children or young adults. Type 2 diabetes is not an autoimmune condition. Rather, it happens when the body’s cells become resistant to insulin over time, and the pancreas is no longer able to make enough insulin to <a href="https://diabetesjournals.org/care/article/47/Supplement_1/S20/153954/2-Diagnosis-and-Classification-of-Diabetes">overcome this resistance</a>. Unlike type 1 diabetes, people with type 2 diabetes still produce some insulin. Type 2 is more common in adults but is <a href="https://www.sciencedirect.com/science/article/abs/pii/S1871402121002733">increasingly</a> seen in children and young people. Management <a href="https://www.racgp.org.au/getattachment/41fee8dc-7f97-4f87-9d90-b7af337af778/Management-of-type-2-diabetes-A-handbook-for-general-practice.aspx">can include</a> behavioural changes such as nutrition and physical activity, as well as oral medications and insulin therapy. <h2>How does type 1.5 diabetes differ from types 1 and 2?</h2> Like type 1 diabetes, type 1.5 occurs when the immune system attacks the pancreas cells that make insulin. But people with type 1.5 often don’t need insulin <a href="https://diabetesjournals.org/diabetes/article/69/10/2037/16062/Management-of-Latent-Autoimmune-Diabetes-in-Adults">immediately</a> because their condition develops more slowly. Most people with type 1.5 diabetes will need to use insulin within <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5111528/">five years</a> of diagnosis, while those with type 1 typically require it from diagnosis. Type 1.5 diabetes is usually diagnosed in people <a href="https://diabetesjournals.org/diabetes/article/69/10/2037/16062/Management-of-Latent-Autoimmune-Diabetes-in-Adults">over 30</a>, likely due to the slow progressing nature of the condition. This is older than the typical age for type 1 diabetes but younger than the usual diagnosis age for type 2. Type 1.5 diabetes shares <a href="https://www.nature.com/articles/nrendo.2017.99">genetic and autoimmune risk factors</a> with type 1 diabetes such as specific gene variants. However, evidence has also shown it may be influenced by lifestyle factors such as <a href="https://pubmed.ncbi.nlm.nih.gov/29589073/">obesity</a> and <a href="https://www.frontiersin.org/journals/physiology/articles/10.3389/fphys.2019.00320/full">physical inactivity</a> which are more commonly associated with type 2 diabetes. <h2>What are the symptoms, and how is it treated?</h2> The symptoms of type 1.5 diabetes are highly variable between people. Some have no symptoms at all. But generally, people may experience the following <a href="https://www.ncbi.nlm.nih.gov/books/NBK557897/">symptoms</a>: <ul> <li>increased thirst</li> <li>frequent urination</li> <li>fatigue</li> <li>blurred vision</li> <li>unintentional weight loss.</li> </ul> Typically, type 1.5 diabetes is <a href="https://diabetesjournals.org/diabetes/article/69/10/2037/16062/Management-of-Latent-Autoimmune-Diabetes-in-Adults">initially treated</a> with oral medications to keep blood glucose levels in normal range. Depending on their glucose control and the medication they are using, people with type 1.5 diabetes may need to monitor their blood glucose levels regularly throughout the day. When average blood glucose levels increase beyond normal range even with oral medications, treatment may progress to insulin. However, there are <a href="https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2022.959011/full">no universally accepted</a> management or treatment strategies for type 1.5 diabetes. <h2>Type 1.5 diabetes is often misdiagnosed</h2> Lance Bass said he was initially diagnosed with <a href="https://www.healthline.com/health-news/lance-bass-misdiagnosed-diabetes">type 2 diabetes</a>, but later learned he actually has type 1.5 diabetes. This is <a href="https://journals.lww.com/jaanp/abstract/2009/03000/latent_autoimmune_diabetes_of_adulthood__lada___an.4.aspx">not entirely uncommon</a>. Estimates suggest type 1.5 diabetes is misdiagnosed as type 2 diabetes <a href="https://www.sciencedirect.com/science/article/abs/pii/S1043276018301292">5–10% of the time</a>. There are a few possible reasons for this. First, accurately diagnosing type 1.5 diabetes, and distinguishing it from other types of diabetes, requires special <a href="https://journals.lww.com/jaanp/abstract/2009/03000/latent_autoimmune_diabetes_of_adulthood__lada___an.4.aspx">antibody tests</a> (a type of blood test) to detect autoimmune markers. Not all health-care professionals necessarily order these tests routinely, either due to cost concerns or because they may not consider them. Second, type 1.5 diabetes is commonly found in adults, so doctors might wrongly assume a person has developed type 2 diabetes, which is more common in this age group (whereas type 1 diabetes usually affects children and young adults). Third, people with <a href="https://diabetesjournals.org/diabetes/article/69/10/2037/16062/Management-of-Latent-Autoimmune-Diabetes-in-Adults">type 1.5 diabetes</a> often initially make enough insulin in the body to manage their blood glucose levels without needing to start insulin medication. This can make their condition appear like type 2 diabetes, where people also produce some insulin. Finally, because type 1.5 diabetes has <a href="https://www.ncbi.nlm.nih.gov/books/NBK557897/">symptoms</a> that are similar to type 2 diabetes, it may initially be treated as type 2. <h2>We’re still learning about type 1.5</h2> Compared with type 1 and type 2 diabetes, there has been much less research on how common type 1.5 diabetes is, especially in <a href="https://www.sciencedirect.com/science/article/abs/pii/S1043276018301292">non-European populations</a>. In 2023, it was estimated type 1.5 diabetes represented <a href="https://pubmed.ncbi.nlm.nih.gov/37428296/">8.9%</a> of all diabetes cases, which is similar to type 1. However, we need more research to get accurate numbers. Overall, there has been a limited awareness of type 1.5 diabetes and <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10683931/">unclear diagnostic criteria</a> which have slowed down our understanding of this condition. A misdiagnosis can be stressful and confusing. For people with type 1.5 diabetes, being misdiagnosed with type 2 diabetes might mean they don’t get the insulin they need in a timely manner. This can lead to worsening health and a greater likelihood of complications down the road. Getting the right diagnosis helps people receive the most appropriate treatment, save money, and reduce <a href="https://diabetesjournals.org/diabetes/article/73/Supplement_1/55-OR/155112/55-OR-Diabetes-Distress-among-Persons-Living-with">diabetes distress</a>. If you’re experiencing symptoms you think may indicate diabetes, or feel unsure about a diagnosis you’ve already received, monitor your symptoms and chat with your doctor.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/237041/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/emily-burch-438717">Emily Burch</a>, Accredited Practising Dietitian and Lecturer, <a href="https://theconversation.com/institutions/southern-cross-university-1160">Southern Cross University</a> and <a href="https://theconversation.com/profiles/lauren-ball-14718">Lauren Ball</a>, Professor of Community Health and Wellbeing, <a href="https://theconversation.com/institutions/the-university-of-queensland-805">The University of Queensland</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/what-is-type-1-5-diabetes-its-a-bit-like-type-1-and-a-bit-like-type-2-but-its-often-misdiagnosed-237041">original article</a>. </div>

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Does eating ham, bacon and beef really increase your risk of developing type 2 diabetes?

<div class="theconversation-article-body"><a href="https://theconversation.com/profiles/duane-mellor-136502">Duane Mellor</a>, <a href="https://theconversation.com/institutions/aston-university-1107">Aston University</a> That lunchtime staple, the humble ham sandwich, has come in for a bashing in the press recently. According to <a href="https://www.theguardian.com/society/article/2024/aug/20/two-slices-of-ham-a-day-can-raise-type-2-diabetes-risk-by-15-research-suggests">many</a> <a href="https://www.dailymail.co.uk/health/article-13761253/Eating-ham-daily-linked-increase-risk-diabetes.html">reports</a>, eating two slices of ham a day can increase your risk of developing type 2 diabetes. But what’s the science behind these headlines? The research offers a more complex picture. <a href="https://www.thelancet.com/journals/landia/article/PIIS2213-8587(24)00179-7/fulltext">A new study</a> from the University of Cambridge highlighted an association between developing type 2 diabetes and eating processed meat like ham and bacon, and red meat such as beef and lamb. This led to headlines suggesting the risk was mainly linked to <a href="https://www.telegraph.co.uk/news/2024/08/21/ham-sandwich-processed-meat-fresh-risk-link-type-2-diabetes/">ham sandwiches</a>. This seems to have come from the <a href="https://www.cam.ac.uk/research/news/red-and-processed-meat-consumption-associated-with-higher-type-2-diabetes-risk">press release</a>, which used ham as the example to quantify the amount of processed meat associated with a 15% increased risk of developing type 2 diabetes over ten years. The research found that this risk was linked to eating an extra 50g of processed meat every day, which happens to equate to two slices of ham. A useful example thus appears to have been taken up by the media as the main cause, perhaps ignoring some of the key messages coming from the study. So, can processed and red meat really increase your risk of developing type 2 diabetes? The <a href="https://www.diabetes.org.uk/diabetes-the-basics/types-of-diabetes/type-2/diabetes-risk-factors">biggest risk factors</a> linked to developing type 2 diabetes are being over 40, having family members with type 2 diabetes, being of South Asian or African descent, or having a higher body weight – and especially a larger waist. <figure><iframe src="https://www.youtube.com/embed/EsOBcx2bJqU?wmode=transparent&start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> The Cambridge study used data from nearly 2 million people from 31 studies. Participants were followed for an average of ten years. During this time, around one in 20 people developed type 2 diabetes. The research suggested that a 10% increase in the probability of developing type 2 diabetes was associated with every 100g of additional red meat eaten daily. Eating half as much extra processed meat every day was linked to an even greater increased risk of developing the disease. This is not the <a href="https://ajcn.nutrition.org/article/S0002-9165(23)66119-2/abstract">first time</a> that both processed and red meats have been linked with an increased risk of developing type 2 diabetes. However, the key strength of the Cambridge study was that it tried to control for many of the other factors linked to the disease, including smoking, having a higher body weight, dietary intake and exercise. However, the size of the increased risk is modest, considering few people included in the study ate 50g or more processed meat per day – meaning moderate ham consumption is likely to have <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9908545/">no meaningful effect</a> on your risk. <h2>What’s the link?</h2> Processed meat has been linked to increased risk of type 2 diabetes because of its nitrate and salt content – additives that are used to cure many processed meats. Nitrates and salt in processed meats have also <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6893523/">been linked to</a> an increased risk of developing colon cancer. In fact, the World Health Organization classifies the additives as <a href="https://www.who.int/news-room/questions-and-answers/item/cancer-carcinogenicity-of-the-consumption-of-red-meat-and-processed-meat">group 1 carcinogens</a>, which means they can cause a range of cancers. The mechanism linking processed meat to cancer seems to be similar to how it might be linked to type 2 diabetes. During digestion, processed meat produces <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6294997/">N-nitroso chemicals</a>, which can damage cells. This can lead to inflammation and affects how insulin, the hormone that controls blood glucose (sugar), works. This in turn can lead to <a href="https://www.niddk.nih.gov/health-information/diabetes/overview/what-is-diabetes/prediabetes-insulin-resistance">insulin resistance</a>, when cells in your muscles, fat and liver don’t respond well to insulin and can’t easily take up glucose from your blood. Red meat, meanwhile, is <a href="https://www.healthline.com/nutrition/healthy-iron-rich-foods">rich in iron</a>. Research suggests that people with <a href="https://www.diabetes.org.uk/diabetes-the-basics/related-conditions/haemochromatosis-diabetes#:%7E:text=So%20a%20rise%20of%20iron,GP%20as%20soon%20as%20possible.">high levels of iron</a> are more likely to develop type 2 diabetes. However, <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8744124/#:%7E:text=The%20WHO%20has%20recognised%20iron,being%20affected%20with%20this%20condition.">low levels of iron</a> are more of a health concern for the general population. Another potential link regarding red meat could be the way it is cooked. <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5521980/">Previous studies</a> have suggested that charred meat, cooked over an open flame or at high temperature, is also linked to an increased risk of developing <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5911789/">type 2 diabetes</a>. Charring meat leads to formation of toxic chemicals such as <a href="https://www.sciencedirect.com/topics/pharmacology-toxicology-and-pharmaceutical-science/heterocyclic-amine#:%7E:text=Heterocyclic%20amines%20are%20aromatic%20compounds,of%20reactions%20called%20Maillard%20reactions.">heterocyclic aromatic amines</a> and harmful compounds like <a href="https://www.healthline.com/nutrition/advanced-glycation-end-products">advanced glycation end products</a>, both of which have been linked to <a href="https://pubmed.ncbi.nlm.nih.gov/21709297/">insulin resistance and type 2 diabetes</a>. <h2>Bye-bye barbecues and bacon butties?</h2> The key message is reduction, rather than avoidance. The UK government nutritional recommendations offer sound advice: limit your combined intake of red and processed meat to no more than <a href="https://www.nhs.uk/live-well/eat-well/food-types/meat-nutrition/#:%7E:text=Red%20meat%20and%20processed%20meat&text=If%20you%20currently%20eat%20more,%2C%20veal%2C%20venison%20and%20goat.">an average of 70g per day</a>. But these guidelines also suggest that red meat can be a valuable source of iron. So, if you decide to stop eating red meat, you should eat alternative sources of iron such as beans, lentils, dark green vegetables and fortified cereals. This needs to be done as part of a carefully planned diet. Non-meat sources of iron are more difficult for our bodies to absorb so should be eaten with a source of vitamin C, found in green vegetables and citrus fruit. The best advice to reduce your risk of developing type 2 diabetes is to maintain a healthy weight – consider losing weight if you have a higher body weight – and be as physically active as possible. A healthy diet should be based on plenty of vegetables, fruit, beans, peas, lentils, nuts and seeds, along with some wholegrain foods, some dairy products, fish and white meat (or vegetarian alternatives) – plus moderate amounts of red meat and minimal processed meat. This will help reduce your risk of type 2 diabetes, <a href="https://www.bhf.org.uk/informationsupport/support/healthy-living/healthy-eating">heart disease</a>, and <a href="https://www.cancerresearchuk.org/about-cancer/causes-of-cancer/diet-and-cancer/does-having-a-healthy-diet-reduce-my-risk-of-cancer">many cancers</a> – as well being more <a href="https://www.bda.uk.com/static/539e2268-7991-4d24-b9ee867c1b2808fc/a1283104-a0dd-476b-bda723452ae93870/one%20blue%20dot%20reference%20guide.pdf">environmentally sustainable</a>. But if you have a penchant for ham sandwiches, rest assured you can continue to indulge as an occasional treat. It’s your overall lifestyle and diet that really matter for your health and risk of developing type 2 diabetes.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/237346/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/duane-mellor-136502">Duane Mellor</a>, Visiting Academic, Aston Medical School, <a href="https://theconversation.com/institutions/aston-university-1107">Aston University</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/does-eating-ham-bacon-and-beef-really-increase-your-risk-of-developing-type-2-diabetes-237346">original article</a>. </div>

Body

$2 billion lotto win tears family apart

A man who won one of the biggest lottery jackpots in American history has been accused of cutting his family out of their promised share after winning $2 billion (AUD) in the Mega Millions jackpot. The unidentified man has been in a legal battle with his daughter’s mum since November, after he accused her of violating a nondisclosure agreement by telling the rest of the family about his fortune before their daughter's 18th birthday in 2032, according to the Independent. He bought the winning ticket in Lebanon, Maine on January 13 2023. The mum – identified by a pseudonym, Sara Smith – claimed that he was the one who told his family about his lotto winnings, not her. The man's father supported Smith's claim and said that his son told him about the win and all the things he planned to do with his new-found fortune, which he collected through an LLC in a lump sum of over $750 million. “February or March of 2023, my son came to my house … and informed me and my wife that he won a large amount of money in the Maine State Lottery,” his father wrote in new court documents. “I understand that my son has stated that he told me nothing about his money ‘other than the simple fact that I had won’,” the dad wrote. “That is not true.” He also claimed that he didn't ask his son for any money, but the lotto-winner allegedly made a bunch of promises, including building his dad a garage to fix up old cars, buying his childhood home, setting up a million-dollar trust fund and funding future medical expenses for his dad and stepmum. The lotto-winner also allegedly demanded his father to not talk to Smith. "I told him … ‘You are not the son I knew’,” his dad wrote in the filing. “He got angry, calling me a ‘dictator’ and an ‘a**ehole’. I have not heard from my son since, and he has not done any of [the] things he promised.” The half-billionaire refuted his dad and Smith's claims. “I made the mistake of telling my father that I had won the lottery without having him sign a confidentiality agreement,” he wrote. “Our relationship deteriorated quickly thereafter,” he continued. “I did not tell him what I was doing with my money, how I was going to benefit my daughter, or any facts other than the simple fact that I had won.” He also accused his ex-partner of trying to reveal his identity to the world and that she wrongly accused him of trying to kidnap their daughter after he refused to pay for her and her new boyfriend's vacation. Image: Shutterstock

Legal

Could not getting enough sleep increase your risk of type 2 diabetes?

<div class="theconversation-article-body"> <a href="https://theconversation.com/profiles/giuliana-murfet-1517219">Giuliana Murfet</a>, <a href="https://theconversation.com/institutions/university-of-technology-sydney-936">University of Technology Sydney</a> and <a href="https://theconversation.com/profiles/shanshan-lin-1005236">ShanShan Lin</a>, <a href="https://theconversation.com/institutions/university-of-technology-sydney-936">University of Technology Sydney</a> Not getting enough sleep is a common affliction in the modern age. If you don’t always get as many hours of shut-eye as you’d like, perhaps you were concerned by news of a <a href="https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2815684">recent study</a> that found people who sleep less than six hours a night are at higher risk of type 2 diabetes. So what can we make of these findings? It turns out the relationship between sleep and diabetes is complex. <h2>The study</h2> Researchers analysed data from the <a href="https://www.ukbiobank.ac.uk/">UK Biobank</a>, a large biomedical database which serves as a global resource for health and medical research. They looked at information from 247,867 adults, following their health outcomes for more than a decade. The researchers wanted to understand the associations between sleep duration and type 2 diabetes, and whether a healthy diet reduced the effects of short sleep on diabetes risk. As part of their involvement in the UK Biobank, participants had been asked roughly how much sleep they get in 24 hours. Seven to eight hours was the average and considered normal sleep. Short sleep duration was broken up into three categories: mild (six hours), moderate (five hours) and extreme (three to four hours). The researchers analysed sleep data alongside information about people’s diets. Some 3.2% of participants were diagnosed with type 2 diabetes during the follow-up period. Although healthy eating habits were associated with a lower overall risk of diabetes, when people ate healthily but slept less than six hours a day, their risk of type 2 diabetes increased compared to people in the normal sleep category. The researchers found sleep duration of five hours was linked with a 16% higher risk of developing type 2 diabetes, while the risk for people who slept three to four hours was 41% higher, compared to people who slept seven to eight hours. One limitation is the study defined a healthy diet based on the number of servings of fruit, vegetables, red meat and fish a person consumed over a day or a week. In doing so, it didn’t consider how dietary patterns such as time-restricted eating or the Mediterranean diet may modify the risk of diabetes among those who slept less. Also, information on participants’ sleep quantity and diet was only captured at recruitment and may have changed over the course of the study. The authors acknowledge these limitations. <h2>Why might short sleep increase diabetes risk?</h2> In people with <a href="https://www.diabetesaustralia.com.au/about-diabetes/type-2-diabetes/">type 2 diabetes</a>, the body becomes resistant to the effects of a hormone called insulin, and slowly loses the capacity to produce enough of it in the pancreas. Insulin is important because it regulates glucose (sugar) in our blood that comes from the food we eat by helping move it to cells throughout the body. We don’t know the precise reasons why people who sleep less may be at higher risk of type 2 diabetes. But <a href="https://doi.org/10.7759/cureus.23501">previous research</a> has shown sleep-deprived people often have increased <a href="https://doi.org/10.1186/1476-511X-9-125">inflammatory markers</a> and <a href="https://doi.org/10.1007/s00125-015-3500-4">free fatty acids</a> in their blood, which <a href="https://doi.org/10.1007/s11892-018-1055-8">impair insulin sensitivity</a>, leading to <a href="https://doi.org/10.7759/cureus.23501">insulin resistance</a>. This means the body struggles to use insulin properly to regulate blood glucose levels, and therefore increases the risk of type 2 diabetes. Further, people who don’t sleep enough, as well as people who sleep in irregular patterns (such as shift workers), experience disruptions to their body’s natural rhythm, known as the <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5995632/">circadian rhythm</a>. This can interfere with the release of hormones like <a href="https://doi.org/10.1210/edrv.18.5.0317">cortisol, glucagon and growth hormones</a>. These hormones are released through the day to meet the body’s changing energy needs, and normally keep blood glucose levels nicely balanced. If they’re compromised, this may reduce the body’s ability to handle glucose as the day progresses. These factors, and <a href="https://www.science.org/doi/10.1126/sciadv.aar8590">others</a>, may contribute to the increased risk of type 2 diabetes seen among people sleeping less than six hours. While this study primarily focused on people who sleep eight hours or less, it’s possible longer sleepers may also face an increased risk of type 2 diabetes. Research has previously shown a U-shaped correlation between sleep duration and type 2 diabetes risk. A <a href="https://doi.org/10.2337/dc14-2073">review</a> of multiple studies found getting between seven to eight hours of sleep daily was associated with the lowest risk. When people got less than seven hours sleep, or more than eight hours, the risk began to increase. The reason sleeping longer is associated with increased risk of type 2 diabetes may be linked to <a href="https://doi.org/10.2337/dc15-0186">weight gain</a>, which is also correlated with longer sleep. Likewise, people who don’t sleep enough are more likely to be <a href="https://doi.org/10.1016/j.sleh.2017.07.013">overweight or obese</a>. <h2>Good sleep, healthy diet</h2> Getting enough sleep is an important part of a healthy lifestyle and may reduce the risk of type 2 diabetes. Based on this study and other evidence, it seems that when it comes to diabetes risk, seven to eight hours of sleep may be the sweet spot. However, other factors could influence the relationship between sleep duration and diabetes risk, such as individual differences in sleep quality and lifestyle. While this study’s findings question whether a healthy diet can mitigate the effects of a lack of sleep on diabetes risk, a wide range of evidence points to the benefits of <a href="https://www.who.int/initiatives/behealthy/healthy-diet">healthy eating</a> for overall health. The <a href="https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2815684">authors of the study</a> acknowledge it’s not always possible to get enough sleep, and suggest doing <a href="https://pubmed.ncbi.nlm.nih.gov/33137489/">high-intensity interval exercise</a> during the day may offset some of the potential effects of short sleep on diabetes risk. In fact, exercise <a href="https://doi.org/10.1016/j.jshs.2023.03.001">at any intensity</a> can improve blood glucose levels.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/225179/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/giuliana-murfet-1517219">Giuliana Murfet</a>, Casual Academic, Faculty of Health, <a href="https://theconversation.com/institutions/university-of-technology-sydney-936">University of Technology Sydney</a> and <a href="https://theconversation.com/profiles/shanshan-lin-1005236">ShanShan Lin</a>, Senior Lecturer, School of Public Health, <a href="https://theconversation.com/institutions/university-of-technology-sydney-936">University of Technology Sydney</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/could-not-getting-enough-sleep-increase-your-risk-of-type-2-diabetes-225179">original article</a>. </div>

Body

Mothers’ dieting habits and self-talk have profound impact on daughters − 2 psychologists explain how to cultivate healthy behaviors and body image

<a href="https://theconversation.com/profiles/janet-j-boseovski-451496">Janet J. Boseovski</a>, <a href="https://theconversation.com/institutions/university-of-north-carolina-greensboro-2069">University of North Carolina – Greensboro</a> and <a href="https://theconversation.com/profiles/ashleigh-gallagher-1505989">Ashleigh Gallagher</a>, <a href="https://theconversation.com/institutions/university-of-north-carolina-greensboro-2069">University of North Carolina – Greensboro</a> Weight loss is one of the most common health and appearance-related goals. Women and <a href="https://www.cdc.gov/nchs/products/databriefs/db340.htm">teen girls</a> are <a href="https://www.cdc.gov/nchs/products/databriefs/db313.htm">especially likely to pursue dieting</a> to achieve weight loss goals even though a great deal of research shows that <a href="https://theconversation.com/what-thin-people-dont-understand-about-dieting-86604">dieting doesn’t work over the long term</a>. We are a <a href="https://www.duck-lab.com/people">developmental psychologist</a> and a <a href="https://psy.uncg.edu/directory/ashleigh-gallagher/">social psychologist</a> who together wrote a forthcoming book, “Beyond Body Positive: A Mother’s Evidence-Based Guide for Helping Girls Build a Healthy Body Image.” In the book, we address topics such as the effects of maternal dieting behaviors on daughters’ health and well-being. We provide information on how to build a foundation for healthy body image beginning in girlhood. <h2>Culturally defined body ideals</h2> Given the strong influence of social media and other cultural influences on body ideals, it’s understandable that so many people pursue diets aimed at weight loss. <a href="https://communityhealth.mayoclinic.org/featured-stories/tiktok-diets">TikTok</a>, YouTube, Instagram and celebrity websites feature slim influencers and “how-tos” for achieving those same results in no time. For example, women and teens are engaging in rigid and extreme forms of exercise such as 54D, a program to <a href="https://54d.com/">achieve body transformation in 54 days</a>, or the <a href="https://health.clevelandclinic.org/75-hard-challenge-and-rules">75 Hard Challenge</a>, which is to follow five strict rules for 75 days. For teens, these pursuits are likely fueled by trendy body preoccupations such as the desire for “<a href="https://www.nytimes.com/2024/02/06/well/move/tiktok-legging-legs-eating-disorders.html">legging legs</a>.” Women and teens have also been been inundated with recent messaging around <a href="https://theconversation.com/drugs-that-melt-away-pounds-still-present-more-questions-than-answers-but-ozempic-wegovy-and-mounjaro-could-be-key-tools-in-reducing-the-obesity-epidemic-205549">quick-fix weight loss drugs</a>, which come with a lot of caveats. Dieting and weight loss goals are highly individual, and when people are intensely self-focused, it is <a href="https://doi.org/10.1521/jscp.2000.19.1.70">possible to lose sight of the bigger picture</a>. Although women might wonder what the harm is in trying the latest diet, science shows that dieting behavior doesn’t just affect the dieter. In particular, for women who are mothers or who have other girls in their lives, these behaviors affect girls’ emerging body image and their health and well-being. <h2>The profound effect of maternal role models</h2> Research shows that mothers and maternal figures <a href="https://doi.org/10.1016/j.brat.2017.11.001">have a profound influence on their daughters’ body image</a>. The opportunity to influence girls’ body image comes far earlier than adolescence. In fact, research shows that these influences on body image <a href="https://www.teenvogue.com/story/how-toxic-diet-culture-is-passed-from-moms-to-daughters">begin very early in life</a> – <a href="https://doi.org/10.1016/bs.acdb.2016.10.006">during the preschool years</a>. Mothers may feel that they are being discreet about their dieting behavior, but little girls are watching and listening, and they are far more observant of us than many might think. For example, one study revealed that compared with daughters of nondieting women, 5-year-old girls whose mothers dieted <a href="https://doi.org/10.1016/S0002-8223(00)00339-4">were aware of the connection between dieting and thinness</a>. Mothers’ eating behavior does not just affect girls’ ideas about dieting, but also their daughters’ eating behavior. The amount of food that mothers eat <a href="https://doi.org/10.1016/j.appet.2018.04.018">predicts how much their daughters will eat</a>. In addition, daughters whose mothers are dieters are <a href="https://doi.org/10.1016/j.appet.2018.04.018">more likely to become dieters themselves</a> and are also <a href="https://doi.org/10.1016/j.eatbeh.2007.03.001">more likely to have a negative body image</a>. Negative body image is <a href="https://theconversation.com/mounting-research-documents-the-harmful-effects-of-social-media-use-on-mental-health-including-body-image-and-development-of-eating-disorders-206170">not a trivial matter</a>. It affects girls’ and women’s mental and physical well-being in a <a href="https://doi.org/10.1177/1359105317710815">host of ways</a> and <a href="https://doi.org/10.1016/j.brat.2011.06.009">can predict the emergence of eating disorders</a>. <h2>Avoiding ‘fat talk’</h2> What can moms do, then, to serve their daughters’ and their own health? They can focus on small steps. And although it is best to begin these efforts early in life – in girlhood – it is never too late to do so. For example, mothers can consider how they think about and talk about themselves around their daughters. Engaging in “fat talk” may inadvertently send their daughters the message that larger bodies are bad, <a href="https://doi.org/10.1016/j.bodyim.2020.07.004">contributing to weight bias</a> and negative self-image. Mothers’ fat talk also <a href="https://doi.org/10.1080/15267431.2021.1908294">predicts later body dissatisfaction in daughters</a>. And negative self-talk isn’t good for mothers, either; it is associated with <a href="https://doi.org/10.1177/1359105318781943">lower motivation and unhealthful eating</a>. Mothers can instead practice and model self-compassion, which involves treating oneself the way <a href="https://doi.org/10.1016/j.bodyim.2016.03.003">a loving friend might treat you</a>. In discussions about food and eating behavior, it is important to avoid moralizing certain kinds of food by labeling them as “good” or “bad,” as girls may extend these labels to their personal worth. For example, a young girl may feel that she is being “bad” if she eats dessert, if that is what she has learned from observing the women around her. In contrast, she may feel that she has to eat a salad to be “good.” Moms and other female role models can make sure that the dinner plate sends a healthy message to their daughters by showing instead that all foods can fit into a balanced diet when the time is right. Intuitive eating, which emphasizes paying attention to hunger and satiety and allows flexibility in eating behavior, is associated with <a href="https://doi.org/10.1007/s40519-020-00852-4">better physical and mental health in adolescence</a>. Another way that women and especially moms can buffer girls’ body image is by helping their daughters <a href="https://doi.org/10.1016/j.bodyim.2021.12.009">to develop media literacy</a> and to think critically about the nature and purpose of media. For example, moms can discuss the misrepresentation and distortion of bodies, such as the use of filters to enhance physical appearance, on social media. <h2>Focusing on healthful behaviors</h2> One way to begin to focus on health behaviors rather than dieting behaviors is to develop respect for the body and to <a href="https://theconversation.com/body-neutrality-what-it-is-and-how-it-can-help-lead-to-more-positive-body-image-191799">consider body neutrality</a>. In other words, prize body function rather than appearance and spend less time thinking about your body’s appearance. Accept that there are times when you may not feel great about your body, and that this is OK. To feel and look their best, mothers can aim to stick to a <a href="https://theconversation.com/whats-the-best-diet-for-healthy-sleep-a-nutritional-epidemiologist-explains-what-food-choices-will-help-you-get-more-restful-zs-219955">healthy sleep schedule</a>, manage their stress levels, <a href="https://theconversation.com/fiber-is-your-bodys-natural-guide-to-weight-management-rather-than-cutting-carbs-out-of-your-diet-eat-them-in-their-original-fiber-packaging-instead-205159">eat a varied diet</a> that includes all of the foods that they enjoy, and <a href="https://theconversation.com/the-runners-high-may-result-from-molecules-called-cannabinoids-the-bodys-own-version-of-thc-and-cbd-170796">move and exercise their bodies regularly</a> as lifelong practices, rather than engaging in quick-fix trends. Although many of these tips sound familiar, and perhaps even simple, they become effective when we recognize their importance and begin acting on them. Mothers can work toward modeling these behaviors and tailor each of them to their daughter’s developmental level. It’s never too early to start. <h2>Promoting healthy body image</h2> Science shows that several personal characteristics are associated with body image concerns among women. For example, research shows that women who are <a href="https://doi.org/10.1016/j.bodyim.2020.02.001">higher in neuroticism</a> <a href="https://doi.org/10.1186/2050-2974-1-2">and perfectionism</a>, <a href="https://doi.org/10.3389/fpsyg.2022.983534">lower in self-compassion</a> or <a href="https://doi.org/10.1016/j.bodyim.2013.08.001">lower in self-efficacy</a> are all more likely to struggle with negative body image. Personality is frequently defined as a person’s characteristic pattern of thoughts, feelings and behaviors. But if they wish, <a href="https://doi.org/10.1002/per.1945">mothers can change personality characteristics</a> that they feel aren’t serving them well. For example, perfectionist tendencies – such as setting unrealistic, inflexible goals – can be examined, challenged and replaced with more rational thoughts and behaviors. A woman who believes she must work out every day can practice being more flexible in her thinking. One who thinks of dessert as “cheating” can practice resisting moral judgments about food. Changing habitual ways of thinking, feeling and behaving certainly takes effort and time, but it is far more likely than diet trends to bring about sustainable, long-term change. And taking the first steps to modify even a few of these habits can positively affect daughters. In spite of all the noise from media and other cultural influences, mothers can feel empowered knowing that they have a significant influence on their daughters’ feelings about, and treatment of, their bodies. In this way, mothers’ modeling of healthier attitudes and behaviors is a sound investment – for both their own body image and that of the girls they love.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/221968/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/janet-j-boseovski-451496">Janet J. Boseovski</a>, Professor of Psychology, <a href="https://theconversation.com/institutions/university-of-north-carolina-greensboro-2069">University of North Carolina – Greensboro</a> and <a href="https://theconversation.com/profiles/ashleigh-gallagher-1505989">Ashleigh Gallagher</a>, Senior Lecturer, <a href="https://theconversation.com/institutions/university-of-north-carolina-greensboro-2069">University of North Carolina – Greensboro</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/mothers-dieting-habits-and-self-talk-have-profound-impact-on-daughters-2-psychologists-explain-how-to-cultivate-healthy-behaviors-and-body-image-221968">original article</a>.

Mind

Mum slammed for being "stingy" after refusing to buy $2 snack for daughter's playdate

A mum has come under fire for being "selfish" and "stingy" after refusing to buy a $2 snack for her 11-year-old daughter’s best friend. The woman, believed to be from the US, and her daughter Ellie were invited for a playdate at an indoor playground with 12-year-old Sophie and her mum. Sophie's mum offered to put them on her membership card so that Ellie and her mum could go to the indoor playground for free. “Sophie’s mum called me... and Sophie wanted to know if Ellie could come and play," she began in a Reddit thread called Am I the a****** . "She offered to put me on her membership card so it would be free for me so I got Ellie in the car and we met them at the playground.” Trouble started when the girls got hungry after an hour of playing, and Ellie's mum only packed a snack for her daughter. “Sophie’s mum didn’t have any snacks on her,” she said. “I told her they sell snacks in the front but she claimed that she didn’t have any money on her and asked me to buy Sophie some Goldfish." Ellie's mum agreed to grab the crackers on one condition - Sophie's mum had to transfer the money to her. “She says she paid for my kid to get in so I could cover the $2 for the Goldfish. I said no, I took care of my kid and it’s not my job to take care of hers too. “I told her if she wanted me to bring snacks she should’ve told me when she invited me but I won’t be wasting $2 for a 50 cent bag of Goldfish because she was unprepared.” She added that Sophie's mum eventually managed to get snacks for her own daughter, and wondered "if she lied about not having money". She then accused Sophie's mum of being "petty" for asking her to pay back for “all the times” she's used her membership to get a guest pass at the indoor playground, adding that "they regularly pay for us to join them on outings.” Her post was met with over 2500 comments slamming her for being “selfish”, “stingy” and “ungrateful”. “You were invited to a place for free that you would otherwise have had to pay for. You only packed snacks for your child? Why didn’t you also take snacks for the other child?" one wrote. “Yes, you did not have to do so, and that child is not your responsibility, but if I was meeting someone for a playdate for my child, not paying to get in, knowing, at some point both girls were going to be hungry, I would have packed snacks for both, as a thank you for the invitation and just because," the commenter continued. “If someone asked me to transfer them $2, I’m rolling my eyes big time. It’s petty, especially when someone gave you something likely far more valuable," another added. “Seriously. I don’t even think I could tell a stranger no when it comes to feeding their hungry child, much less a person I know and spend time with," a third commented. Others called the mum a "fool", for potentially causing Ellie to lose her best friend. “Don’t be an idiot. Apologise. You might care about 50 cents. But your daughter will lose her best friend. And that is worth a lot more. Your daughter might never get a friend like that... And the fact that universe gift wrapped a friend for your daughter. And you choose to throw it in the trash. Wow, you are truly a fool," they said. Image: Getty

Family & Pets

Liam Neeson filming new movie in tiny Aussie town

The quaint, gorgeous historic Victorian town of Walhalla is gearing up for its moment in the cinematic spotlight as it gets set to become the backdrop for Liam Neeson's upcoming blockbuster sequel, Ice Road 2: Road To The Sky. Yes, apparently, the road to the sky involves a detour through rural Australia. Residents of Walhalla received a letter announcing the impending movie magic, and it's safe to say they're experiencing a mixture of excitement and confusion akin to trying to follow the plot of a Christopher Nolan epic. According to reports, the filmmakers are turning Walhalla into a bona fide Nepalese village. The town's Star Hotel and surrounding areas are getting a makeover to mimic Kodari, Nepal. Now, if you're wondering where Walhalla is on the map, don't worry, you're not alone. Even the residents seem a bit perplexed, with one local commenting online, "Interesting that this is going ahead at the height of our tourist season." Because, naturally, when you think tourist hotspots, you think Walhalla. But fear not, dear residents, for the filmmakers have assured everyone that after their Himalayan escapade, Walhalla will return to its original heritage colours. It's like the town is getting a cinematic spa day, complete with a paint job. Filming is set to take place at two main locations: the intersection of Main Rd and Right Hand Branch Rd and the top of Churchill Rd above the Fire Station Museum. And oh boy, get ready for some action, because the letter states, "During the filming period, there will be stunts involving large vehicles, special effects and prop gun use." Walhalla, known for its scenic beauty and historic charm, is about to witness the fusion of Hollywood glitz and Nepalese grit. Of course, not everyone is on board with this Hollywood invasion. One local expressed concern about the impact on other businesses in town, suggesting, "This would have been much better slotted into the quiet time in August." Clearly, they're not buying into the idea that summer is the best time for a Nepalese makeover. But fear not, skeptics! Another resident pointed out that the influx of up to 200 crew members per day will be a boon for local shops. "What a great thing for the area," they declared. And who can argue with that logic? Imagine the crew swarming the pub, devouring schnitties and downing pints of Carlton lager. This could be the most Aussie-Nepalese fusion experience since Vegemite momos. As the charming town of Walhalla braces itself for the coming storm of movie magic, we can't help but wonder: Will Liam Neeson's next iconic line be, "I will find you, even if I have to navigate the treacherous roads of rural Australia"? Buckle up, Walhalla, because the road to the sky might just be a detour through down under. Images: Visit Victoria / Netflix

Movies

Pitch to ditch the King from Aussie coins

Bob Katter is calling for a major overhaul of Australian coins, saying King Charles' image should be scrapped from the currency. The federal MP touted an alternative design for the national coins, suggesting it could feature a Kalkadoon warrior or distinguished Australian soldier Ralph Honner. “Surely you’d put Kokoda hero Ralph Honner on your coin, not some British monarch, demonstrating that you don’t believe that all people are born free and equal and that you don’t believe you’re a separate country, that you’re a nationalistic Australian,” Katter said on Monday. The Queensland MP plans to move an amendment to the Crown References Amendment Bill to omit references to the monarchy and substitute the words “sovereign people of Australia”. “For heaven’s sake, get rid of the affirmation that we believe that all people are free and equal,” Katter said. “If you’ve got a monarch on your coin, you do not believe that all people are free and equal.” Katter's pitch comes just weeks after the Royal Australian Mint last month unveiled the effigy of King Charles III, which will be seen on Australian coins by Christmas. For decades, the country’s coins have carried an image of Queen Elizabeth II, who died in 2022. The Royal Mint also recently announced the production of a <a href="https://oversixty.com.au/finance/money-banking/new-commemorative-queen-coin-worth-serious-cash" target="_blank" rel="noopener">commemorative coin</a> in honour of the late Queen Elizabeth, which is already in high demand among royal fans and avid coin collectors. Image credits: Getty Images

Money & Banking

New commemorative Queen coin worth serious cash

The Royal Australian Mint has confirmed that it will be releasing a commemorative 50c coin to celebrate the life of Queen Elizabeth II, on Thursday. The coin will feature all six effigies which have been featured on Australian coins during the late monarch’s reign, with two versions up for sale. One is an uncirculated version which will cost $15 and, the other is silver proof edition for $135. “With limited mintage, this coin is expected to be a highly prized addition to any coin collection,” the Mint said. Australian coin expert Joel Kandia said that online marketplaces are already selling the coin at “seven times the RRP”. Royal Australian Mint CEO Leigh Gordon added that this latest release is the perfect tribute to the late Queen. “Historically, coins bear witness to a Monarch’s reign with their royal effigies appearing on the obverse. In keeping with that tradition, this exceptional coin showcases the Queen Elizabeth II Memorial Effigy by Jody Clark on the obverse,” he said. “The Mint’s trademark storytelling is strongly represented on the coin’s reverse, which features a central design depicting the first six effigies, fanned above the Queen’s royal cypher.” This surprise release will be in high demand, with a “frenzy” expected for coin collectors, according to the Perth coin and bank note expert. “It is essentially the last coin commemorating the Queen,” Kandiah said in an interview with 7News. “It is extremely special because it features all six effigies of the Queen that have appeared on Australian coinage since 1954, so it unique in that respect. “There will definitely be a frenzy, which is why the RAM have reduced the allocation to just one per person through their physical store, through the phone and their authorised distributors. “There have been murmurings about the coin for a while, so collectors are really excited to see it confirmed and able for purchase.” The uncirculated coin itself will have a mintage of 25,000 and the silver proof version has an even lower mintage of 7,500. The coins will be for sale at the Royal Australian Mint in Canberra from 8.30am on Thursday November 23, through the Mint’s Contact Centre on 1300 352 020, or through the Mint’s authorised distributors. Image: Royal Australian Mint

Money & Banking

myTax is fast and free – so why do 2 in 3 Australians still pay to lodge a tax return?

<a href="https://theconversation.com/profiles/jawad-harb-1441668">Jawad Harb</a>, <a href="https://theconversation.com/institutions/rmit-university-1063">RMIT University</a> and <a href="https://theconversation.com/profiles/elizabeth-morton-1218408">Elizabeth Morton</a>, <a href="https://theconversation.com/institutions/rmit-university-1063">RMIT University</a> Ten years ago, the Australian Taxation Office (ATO) created the “myTax” portal, an easy way to lodge your tax return online. There was an “e-Tax” filing option before the 2015-16 tax year, but this was quite complicated and barely better than filling out a form online. In comparison, myTax <a href="https://resources.taxinstitute.com.au/tiausttaxforum/acceptance-of-mytax-in-australia">is simpler</a> and more automated. It’s available 24 hours a day, is free to use, and you will typically get your refund within <a href="https://www.ato.gov.au/Individuals/Your-tax-return/How-to-lodge-your-tax-return/Lodge-your-tax-return-online-with-myTax/">two weeks</a>. But the chances are you won’t be using it. In fact, slightly less than <a href="https://www.ato.gov.au/About-ATO/Research-and-statistics/In-detail/Taxation-statistics/Taxation-statistics-2020-21/?anchor=IndividualsStatistics#IndividualsStatistics">36%</a> of Australia’s 15 million taxpayers used the myTax portal in 2020-21 – the most recent tax year for which the tax office has published data. About <a href="https://www.ato.gov.au/About-ATO/Research-and-statistics/In-detail/Taxation-statistics/Taxation-statistics-2020-21/?anchor=IndividualsStatistics#IndividualsStatistics">64% of tax returns</a> were lodged through tax agents. This is one of the highest rates among 38 <a href="https://www.oecd.org/about/">Organisation for Economic Co-operation and Development</a> nations. Meanwhile, just 0.6% of Australians still used the paper-based form. <hr /> <iframe id="5Kdz0" class="tc-infographic-datawrapper" style="border: none;" src="https://datawrapper.dwcdn.net/5Kdz0/3/" width="100%" height="400px" frameborder="0"></iframe> <hr /> So why have Australians – who have quickly embraced the internet for everything from shopping to dating – been so slow to embrace myTax? For some, particularly older people, it’s about being intimidated by the technology. Others may be concerned with cybersecurity risk. But for most it’s about the perceived complexity of the tax system and the process, regardless of the technology. They see using a tax agent as easier and the way to maximise their tax refund. While in some cases this may be true, in many instances it’s simply a perception – but one the tax office will need to address if it wants to promote use of myTax. <h2>Reasons for the low uptake of myTax</h2> <a href="https://resources.taxinstitute.com.au/tiausttaxforum/acceptance-of-mytax-in-australia">Our research</a> suggests most people who have used the myTax portal think it is easy to use. We surveyed 193 taxpayers who have used the system. About three-quarters agreed the system was clear and understandable, and said they would keep using it. But of course these are people who have chosen to use the system, so their responses don’t shed much light on the reasons people don’t use myTax. Answers to that come from other published research, in particular from the <a href="https://www.igt.gov.au/">Inspector-General of Taxation</a> (the independent office investigating complaints about the tax system) as well as the House of Representatives’ Standing Committee on Tax and Revenue. Evidence submitted to these bodies indicate that Australians prefer tax agents to avoid errors in claiming deductions. The parliamentary committee’s <a href="https://parlinfo.aph.gov.au/parlInfo/download/committees/reportrep/024169/toc_pdf/TaxpayerEngagementwiththeTaxSystem.pdf;fileType=application%2Fpdf">2018 inquiry</a> into the tax system was told the use of tax agents ballooned from about 20% in the 1980s, peaking at about 74% of all taxpayers: "The Tax Commissioner considered that the size of the TaxPack had probably contributed to that rise, driving many people with simple tax affairs to a tax agent because it looked daunting." In short, habits are hard to break. Having come to rely on tax agents, most Australians keep using them, despite the system being vastly improved. For example, the myTax system now simplifies the process by <a href="https://www.ato.gov.au/Individuals/Your-tax-return/How-to-lodge-your-tax-return/Lodge-your-tax-return-online-with-myTax/Pre-fill-availability/?=redirected_myGov_prefill">pre-filling</a> data from government agencies, health funds, financial institutions and your own employer. About 80% of our survey respondents said this was helpful. <h2>Taking care of the digital divide</h2> This suggests the main barrier to increasing use of the myTax system is mostly habit and the perception the tax system is too complicated to navigate without an expert. There is also a small percentage of people who feel uncomfortable with computers. This is reflected in the minority of respondents in our study who said they were unlikely to use myTax again, as well as the tax office’s data showing some people continue to stick with paper lodgement. Those more likely to find the system daunting are the elderly, those with low English skills, people with disabilities and those with low educational attainment. These people’s needs should not be forgotten as the Australian <a href="https://www.dta.gov.au/digital-government-strategy">Digital Government Strategy</a> aims to making Australia a “world-leading” digital government by 2025, delivering “simple, secure and connected public services”. Even with the greatest online system in the world, it’s unlikely there will ever be a complete transition.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/207305/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/jawad-harb-1441668">Jawad Harb</a>, PhD Candidate, <a href="https://theconversation.com/institutions/rmit-university-1063">RMIT University</a> and <a href="https://theconversation.com/profiles/elizabeth-morton-1218408">Elizabeth Morton</a>, Research Fellow of the RMIT Blockchain Innovation Hub, Lecturer Taxation, <a href="https://theconversation.com/institutions/rmit-university-1063">RMIT University</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/mytax-is-fast-and-free-so-why-do-2-in-3-australians-still-pay-to-lodge-a-tax-return-207305">original article</a>.

Money & Banking

What happens in our body when we encounter and fight off a virus like the flu, SARS-CoV-2 or RSV?

<a href="https://theconversation.com/profiles/lara-herrero-1166059">Lara Herrero</a>, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> and <a href="https://theconversation.com/profiles/wesley-freppel-1408971">Wesley Freppel</a>, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> <a href="https://www.labcorp.com/coronavirus-disease-covid-19/covid-news-education/covid-19-vs-flu-vs-rsv-how-tell-difference">Respiratory viruses</a> like influenza virus (flu), SARS-CoV-2 (which causes COVID) and respiratory syncytial virus (RSV) can make us sick by infecting our respiratory system, including the nose, upper airways and lungs. They spread from person to person through respiratory droplets when someone coughs, sneezes, or talks and can cause death in serious cases. But what happens in our body when we first encounter these viruses? Our immune system uses a number of strategies to fight off viral infections. Let’s look at how it does this. <h2>First line of defence</h2> When we encounter respiratory viruses, the <a href="https://www.sciencedirect.com/science/article/pii/S193131281600038X?via%3Dihub/">first line of defence</a> is the physical and chemical barriers in our nose, upper airways, and lungs. Barriers like the mucus lining and hair-like structures on the surface of cells, work together to trap and remove viruses before they can reach deeper into our respiratory system. Our defence also includes our behaviours such as coughing or sneezing. When we blow our nose, the mucus, viruses, and any other pathogens that are caught within it are expelled. But sometimes, viruses manage to evade these initial barriers and sneak into our respiratory system. This activates the cells of our innate immune system. <h2>Patrolling for potential invaders</h2> While our acquired immune system develops over time, our innate immune system is present at birth. It generates “non-specific” immunity by identifying what’s foreign. The cells of innate immunity act like a patrol system, searching for any invaders. These innate cells patrol almost every part of our body, from our skin to our nose, lungs and even internal organs. Our respiratory system has different type of innate cells such – as macrophages, neutrophils and natural killer cells – which patrol in our body looking for intruders. If they recognise anything foreign, in this case a virus, they will initiate an attack response. Each cell type plays a slightly different role. Macrophages, for example, will not only engulf and digest viruses (phagocytosis) but also release a cocktail of different molecules (cytokines) that will warn and recruit other cells to <a href="https://onlinelibrary.wiley.com/doi/full/10.1111/cmi.12580">fight against the danger</a>. In the meantime, natural killer cells, aptly named, attack infected cells, and stop viruses from multiplying and <a href="https://www.nature.com/articles/s41577-021-00558-3">invading our body further</a>. Natural killer cells also promote inflammation, a <a href="https://www.hindawi.com/journals/jir/2018/1467538/">crucial part of the immune response</a>. It helps to recruit more immune cells to the site of infection, enhances blood flow, and increases the permeability of blood vessels, allowing immune cells to reach the infected tissues. At this stage, our immune system is fighting a war against viruses and the result can cause inflammation, fevers, coughs and congestion. <h2>Launching a specific attack</h2> As the innate immune response begins, another branch of the immune system called the adaptive immune system is <a href="https://www.ncbi.nlm.nih.gov/books/NBK21070/">activated</a>. The adaptive immune system is more specific than the innate immune system, and it decides on the correct tools and strategy to fight off the viral invaders. This system plays a vital role in eliminating the virus and providing long-term protection against future infections. Specialised cells called T cells and B cells are key players in acquired immunity. T cells (specifically, helper T cells and cytotoxic T cells) recognise viral proteins on the surface of infected cells: <ul> <li> helper T cells release molecules that <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3764486/">further activate immune cells</a> </li> <li> cytotoxic T cells directly kill infected cells with a very great precision, <a href="https://www.frontiersin.org/articles/10.3389/fimmu.2018.00678/full">avoiding any healthy cells around</a>. </li> </ul> B cells produce antibodies, which are proteins that can bind to viruses, neutralise them, and mark them for <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7247032/">destruction by other immune cells</a>. B cells are a critical part of memory in our immune system. They will remember what happened and won’t forget for years. When the same virus attacks again, B cells will be ready to fight it off and will neutralise it faster and better. Thanks to the adaptive immune system, vaccines for respiratory viruses such as the COVID mRNA vaccine keep us protected from <a href="https://www.health.gov.au/our-work/covid-19-vaccines/our-vaccines/how-they-work">being sick or severely ill</a>. However, if the same virus became mutated, our immune system will act as if it was a new virus and will have to fight in a war again. <h2>Neutralising the threat</h2> As the immune response progresses, the combined efforts of the innate and adaptive immune systems helps control the virus. Infected cells are cleared, and the virus is neutralised and eliminated from the body. As the infection subsides, symptoms gradually improve, and we begin to feel better and to recover. But recovery varies depending on the specific virus and us as individuals. Some respiratory viruses, like rhinoviruses which cause the common cold, may cause relatively mild symptoms and a quick recovery. Others, like the flu, SARS-CoV-2 or severe cases of RSV, may lead to more severe symptoms and a longer recovery time. Some viruses are very strong and too fast sometimes so that our immune system does not have the time to develop a proper immune response to fight them off. <img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/207023/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/lara-herrero-1166059">Lara Herrero</a>, Research Leader in Virology and Infectious Disease, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> and <a href="https://theconversation.com/profiles/wesley-freppel-1408971">Wesley Freppel</a>, Research Fellow, Institute for Glycomics, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/what-happens-in-our-body-when-we-encounter-and-fight-off-a-virus-like-the-flu-sars-cov-2-or-rsv-207023">original article</a>.

Body

Bees have appeared on coins for millennia, hinting at an age-old link between sweetness and value

<a href="https://theconversation.com/profiles/adrian-dyer-387798">Adrian Dyer</a>, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a> In 2022, the Royal Australian Mint issued a $2 coin decorated with honeybees. Around 2,400 years earlier, a mint in the kingdom of Macedon had the same idea, creating a silver obol coin with a bee stamped on one side. Over the centuries between these two events, currency demonstrating a symbolic link between honey and money is surprisingly common. In a recent study in <a href="https://s3.ap-southeast-2.amazonaws.com/assets.mmxgroup.com.au/ACR/Bee+Article.pdf">Australian Coin Review</a>, I trace the bee through numismatic history – and suggest a scientific reason why our brains might naturally draw a connection between the melliferous insects and the abstract idea of value. <figure class="align-center "><img src="https://images.theconversation.com/files/536400/original/file-20230709-15-2u5ywn.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/536400/original/file-20230709-15-2u5ywn.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=600&fit=crop&dpr=1 600w, https://images.theconversation.com/files/536400/original/file-20230709-15-2u5ywn.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=600&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/536400/original/file-20230709-15-2u5ywn.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=600&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/536400/original/file-20230709-15-2u5ywn.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=754&fit=crop&dpr=1 754w, https://images.theconversation.com/files/536400/original/file-20230709-15-2u5ywn.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=754&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/536400/original/file-20230709-15-2u5ywn.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=754&fit=crop&dpr=3 2262w" alt="" /><figcaption>A Royal Australian Mint 2022 two-dollar coin representing 200 years since the introduction of the honeybee to Australia.</figcaption></figure> <h2>What is currency and why is it important?</h2> Money is a store of value, and can act as a medium of exchange for goods or services. Currency is a physical manifestation of money, so coins are a durable representation of value. Coins have had central role in many communities to enable efficient trade since ancient times. Their durability makes them important time capsules. Ancient Malta was famous for its honey. The modern 3 Mils coin (<a href="https://en.numista.com/catalogue/pieces1775.html">1972-81</a>) celebrates this history with images of a bee and honeycomb. According to the information card issued with the coin set, <blockquote> A bee and honeycomb are shown on the 3 Mils coin, symbolising the fact that honey was used as currency in Ancient Malta. </blockquote> <figure class="align-center "><img src="https://images.theconversation.com/files/536403/original/file-20230709-23-drk2lj.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/536403/original/file-20230709-23-drk2lj.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=582&fit=crop&dpr=1 600w, https://images.theconversation.com/files/536403/original/file-20230709-23-drk2lj.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=582&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/536403/original/file-20230709-23-drk2lj.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=582&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/536403/original/file-20230709-23-drk2lj.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=732&fit=crop&dpr=1 754w, https://images.theconversation.com/files/536403/original/file-20230709-23-drk2lj.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=732&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/536403/original/file-20230709-23-drk2lj.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=732&fit=crop&dpr=3 2262w" alt="" /><figcaption>A circulating 3 Mils coin from Malta showing a honeybee on honeycomb.</figcaption></figure> In ancient Greece, bees were used on some of the earliest coins made in Europe. A silver Greek obol coin minted in Macedon between 412 BCE and 350 BCE, now housed in the British Museum, shows a bee on one side of the coin. <figure class="align-center "><img src="https://images.theconversation.com/files/536411/original/file-20230709-182252-v4evxr.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/536411/original/file-20230709-182252-v4evxr.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=293&fit=crop&dpr=1 600w, https://images.theconversation.com/files/536411/original/file-20230709-182252-v4evxr.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=293&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/536411/original/file-20230709-182252-v4evxr.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=293&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/536411/original/file-20230709-182252-v4evxr.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=368&fit=crop&dpr=1 754w, https://images.theconversation.com/files/536411/original/file-20230709-182252-v4evxr.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=368&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/536411/original/file-20230709-182252-v4evxr.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=368&fit=crop&dpr=3 2262w" alt="" /><figcaption>An ancient obol from Macedon, dated between 412 BCE and 350 BCE, shows a bee one side.</figcaption></figure> Bees also feature on coins minted elsewhere in the ancient Greek world, such as a bronze coin minted in Ephesus dated between 202 BCE and 133 BCE. <figure class="align-center "><img src="https://images.theconversation.com/files/536407/original/file-20230709-27-a2jvo3.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/536407/original/file-20230709-27-a2jvo3.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=546&fit=crop&dpr=1 600w, https://images.theconversation.com/files/536407/original/file-20230709-27-a2jvo3.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=546&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/536407/original/file-20230709-27-a2jvo3.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=546&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/536407/original/file-20230709-27-a2jvo3.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=686&fit=crop&dpr=1 754w, https://images.theconversation.com/files/536407/original/file-20230709-27-a2jvo3.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=686&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/536407/original/file-20230709-27-a2jvo3.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=686&fit=crop&dpr=3 2262w" alt="" /><figcaption>A bronze coin minted in Ephesus, dated between 202BCE and 133BCE, featuring a honeybee.</figcaption></figure> The use of bees on ancient coins extended for many centuries including widely circulated bronze coins, and new varieties <a href="https://coinweek.com/bee-all-that-you-can-bee-honeybees-on-ancient-coins/">continue to be discovered</a>. <h2>Why we might like bees on coins</h2> Why have bees appeared so often on coins? One approach to this question comes from the field of neuro-aesthetics, which seeks to understand our tastes by understanding the basic brain processes that underpin aesthetic appreciation. From this perspective, it seems likely the sweet taste of honey – which indicates the large amount of sugar it delivers – promotes positive neural activity <a href="https://brill.com/view/journals/artp/10/1/article-p1_2.xml">associated with bees and honey</a>. Indeed, primatologist Jane Goodall once proposed that obtaining high-calorie nutrition from bee honey may have been <a href="https://www.sciencedirect.com/science/article/abs/pii/S0066185668800032">an important step</a> in the cognitive development of primates. Our brain may thus be pre-adapted to liking bees due to their association with the sweet taste of honey. Early usage of bees on coins may have been a functional illustration of the link between a known value (honey) and a new form of currency: coins as money. <h2>The bee on modern coins</h2> <figure class="align-center "><img src="https://images.theconversation.com/files/536393/original/file-20230709-17-jywq3f.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/536393/original/file-20230709-17-jywq3f.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=588&fit=crop&dpr=1 600w, https://images.theconversation.com/files/536393/original/file-20230709-17-jywq3f.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=588&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/536393/original/file-20230709-17-jywq3f.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=588&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/536393/original/file-20230709-17-jywq3f.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=738&fit=crop&dpr=1 754w, https://images.theconversation.com/files/536393/original/file-20230709-17-jywq3f.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=738&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/536393/original/file-20230709-17-jywq3f.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=738&fit=crop&dpr=3 2262w" alt="" /><figcaption>A 1920 Italian bronze ten-centesimi coin featuring featuring an Italian honeybee on a flower.</figcaption></figure> The use of bees as a design feature has persisted from ancient to modern times. A honeybee visiting a flower is shown on a series of ten-centesimi bronze coins issued in Italy from <a href="https://en.numista.com/catalogue/pieces1960.html">1919 to 1937</a>. (As an aside, the world’s last stock of pure Italian honeybees is found in Australia, on Kangaroo Island, which was declared a sanctuary for Ligurian bees by an <a href="https://www.legislation.sa.gov.au/home/historical-numbered-as-made-acts/1885/0342-Lingurian-Bees-Act-No-342-of-48-and-49-Vic,-1885.pdf">act of parliament</a> in 1885.) <figure class="align-center "><img src="https://images.theconversation.com/files/536416/original/file-20230709-15-60yst8.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/536416/original/file-20230709-15-60yst8.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=586&fit=crop&dpr=1 600w, https://images.theconversation.com/files/536416/original/file-20230709-15-60yst8.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=586&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/536416/original/file-20230709-15-60yst8.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=586&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/536416/original/file-20230709-15-60yst8.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=737&fit=crop&dpr=1 754w, https://images.theconversation.com/files/536416/original/file-20230709-15-60yst8.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=737&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/536416/original/file-20230709-15-60yst8.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=737&fit=crop&dpr=3 2262w" alt="" /><figcaption>A coin from Tonga showing 20 honeybees emerging from a hive.</figcaption></figure> More recently, a 20-seniti coin from the Pacific nation of Tonga shows 20 honeybees flying out of a hive. This coin was part of a series initiated by the Food and Agriculture Organization of the United Nations to promote sustainable agricultural and cultural development around the world. Bees are relevant here because their pollinating efforts contribute to about one-third of the food required to feed the world, with a value in excess of <a href="https://zenodo.org/record/2616458">US$200 billion per year</a>, and they are threatened by climate change and other environmental factors. <h2>Bees on coins, today and tomorrow</h2> Public awareness of bees and environmental sustainability may well be factors in the current interest in bee coins. The diversity of countries using bees as a design feature over the entire history of coins suggests people have valued the relationship with bees as essential to our own prosperity for a long time. In Australia, the 2022 honeybee $2 coin is part of a series developed by the <a href="https://www.ramint.gov.au/about-mint">Royal Australian Mint</a>. In 2019, the Perth Mint in Western Australia also released coins and stamps celebrating native bees. <figure class="align-center "><img src="https://images.theconversation.com/files/536405/original/file-20230709-15-iditcb.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/536405/original/file-20230709-15-iditcb.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=373&fit=crop&dpr=1 600w, https://images.theconversation.com/files/536405/original/file-20230709-15-iditcb.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=373&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/536405/original/file-20230709-15-iditcb.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=373&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/536405/original/file-20230709-15-iditcb.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=469&fit=crop&dpr=1 754w, https://images.theconversation.com/files/536405/original/file-20230709-15-iditcb.jpg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=469&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/536405/original/file-20230709-15-iditcb.jpg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=469&fit=crop&dpr=3 2262w" alt="" /><figcaption>Australian native bee coin and stamps released in 2019 by the Perth Mint.</figcaption></figure> Despite the decline of cash, bee coins still appear to be going strong. The buzzing companions of human society are likely to be an important subject for coin design for as long as coins continue to be used.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/208912/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/adrian-dyer-387798">Adrian Dyer</a>, Associate Professor, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a> Image credits: Australian Royal Mint / NZ Post Collectables This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/bees-have-appeared-on-coins-for-millennia-hinting-at-an-age-old-link-between-sweetness-and-value-208912">original article</a>.

Money & Banking

Adorable reason behind Chris Hemsworth’s red carpet cheat notes

Chris Hemsworth has been married to Spanish actress Elsa Pataky for almost 13 years, and while she speaks fluent English, Hemsworth is yet to master her native tongue. On June 8, he was at the premiere of his Netflix film Extraction 2 and was caught with some Spanish words scrawled on the palm of his hand. The 39-year-old was then photographed with what appeared to be a cheat sheet, which he personally found hilarious. “After years of coming to Spain and being asked ‘has my Spanish improved’ I can safely say it’s in the palm of my hand,” he wrote on Instagram alongside the photo. <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/p/CtOqVmTJ1sx/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/p/CtOqVmTJ1sx/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Chris Hemsworth (@chrishemsworth)</a> </div> </blockquote> The actor revealed what was written on his hand during a guest appearance on local TV program El Hormiguero (Spanish for The Anthill). In the interview, Hemsworth also shared that his three children with Pataky - daughter India, 10, and twin sons Tristan and Sasha, 9 - find it funny that he doesn’t speak nor understand the language. “I try, but I can’t. My children laugh at me when I try to have a conversation with them in Spanish,” he admitted. “Sh*t, f**k, what happened? … I know that, that’s what my wife yells at me. The more she gets angry, the more she speaks Spanish.” Speaking to Today in 2017, Pataky said she had pretty much given up on teaching Hemsworth her native language, focusing on teaching their children instead. “He promised me, he said, ‘I’ll be speaking Spanish in two months.’ There we go, we have been together for six years,” Pataky – who speaks five languages: English, Spanish, Italian, French and Romanian - told Today. “That’s important, that’s what my mum did to me, talked in Romanian. I start to speak in English, I’m like, ‘I don’t express myself great.’ I got used to making an effort to speak to [the kids] in Spanish.” Image credit: Instagram

Movies

Brand new mitey $2 coin revealed

A brand new $2 coin has been revealed to celebrate 100 years of Australia’s beloved Vegemite. Woolworths and the Royal Australian Mint have partnered up for the release of the limited edition $2 coins marking 100 years of the infamous Aussie spread. Woolworths shoppers across the country will be able to collect three exclusive versions of the gold coin so long as they pay with cash. Three million limited edition coins will be available for customers, with a new design released to cash tills each week over a three-week period. The coins have been designed by Royal Australian Mint designer Aaron Baggio, with each featuring a unique illustration honouring Vegemite. Each of the three designs has a different coloured circle - yellow, red and black. The first coin shows a jar of Vegemite with the words “100 Mitey Years” at the bottom. Another features a slice of Vegemite toast with the words “Tastes Like Australia”, while the third features a child eating a slice of Vegemite toast with the words “Happy Little Vegemites”. Woolworths brand and marketing director Jane Sales said the supermarket has supported Vegemite for decades and is excited to be part of the centenary celebrations. “It’s been a staple in the shopping baskets of Australians for years; we’re delighted to be a part of the celebrations for this iconic brand’s centenary,” she said. As part of the celebrations, there will also be a $1 coin featuring the Queen Elizabeth II Memorial Obverse. The portrait of Queen Elizabeth II by British engraver Jody Clark, the Memorial Obverse also added the Queen’s years of reign – reading “Elizabeth II 1952-2022”. Assistant Minister for Competition, Charities and Treasury, Andrew Leigh, said Vegemite is one of the most loved brands across the country. “It is fitting the Royal Australian Mint has partnered with Vegemite and Woolworths to celebrate 100 Mitey years of an Australian icon,” he said. The Vegemite brand will be celebrating its 100th birthday on October 25, 2023, with Bega’s marketing manager for spreads, Jess Hoare, thanking all Aussies for loving the mitey product. Image credit: Getty/Royal Australian Mint

Money & Banking

“Lunch-box-mum queen”: Woman goes viral over 2 ingredient cake mix

One Coles shopper has taken the internet by storm, revealing a “snack hack” with just two ingredients. Aussie mum-of-three Claudia creates content on TikTok on cheap Kmart buys, a day in the life of her family, and her most popular videos, her “snack hacks”. For her most recent hack, only two ingredients are required. “Snack hack! Again!” she said in the video, which has attracted more than 70,000 views on TikTok. “So, this one, I’m looking forward to. “This is not healthy. This is two ingredients — Nutella and eggs — and it’s supposed to make the gooiest, chocolatiest, yummiest cake ever. “I have not tried this before so let’s get to it.” Claudia didn’t have Nutella in her pantry, but she bought the Coles version, which she claims tastes very similar. “I’m a big believer in using what you have, but a few people said ‘check this out, make this, it’s delicious’ so I just had to,” she continued. “I did go and buy the Coles one, and it was very cheap, and I’m sure a lot of people do have Nutella in their cupboard.” For the snack hack, Claudia used one cup of the chocolate spread and four eggs to create a gooey cake mix. “If ‘trust the process’ had a cover photo, it would be this,” she said, visibly grossed out by the gooey batter. “It is safe to say I won’t be trying any of this cake mix.” Claudia instructed viewers to put the cake in the oven for 20-25 minutes at 180 degrees Celsius. “Do you know how good this smells?” she said as she took the cake tin out of the oven. She was even more excited by the time she tried it. “That is so good. So, so good. Like no exaggeration. So freaking good,” she said. People flooded the comments, applauding Claudia for another great “snack hack”. “I’m soooo trying this,” one person wrote. “Thank you, lunch-box-mum queen,” another added. “I honestly thought it was going to come out looking like chocolate scrambled eggs,” a third said. Claudia is renowned on TikTok for making snacks that are easy on household budgets, so people were grateful she used the cheaper Coles chocolate spread. “The Coles Nutella is just as good in my opinion,” one wrote. Image credit: TikTok

Food & Wine

Autism and ADHD assessment waits are up to 2 years’ long. What can families do in the meantime?

Reports have emerged from around Australia of waitlists of <a href="https://www.smh.com.au/national/nsw/unacceptable-wait-to-screen-children-for-developmental-delays-autism-20220125-p59r1d.html">up to two years</a> to receive a diagnostic assessment for neurodevelopmental conditions, such as autism and attention deficit hyperactivity disorder (ADHD). Assessment delays can create additional stress for families who are already worrying their child may be developing differently. These waiting times are a symptom of the significant strain our health systems are under. System reform will take time, and in the meantime, there are many children who require urgent support. But supporting your child doesn’t need to be put on hold while you wait for assessment. Why are waitlists so long? <a href="https://theconversation.com/wondering-about-adhd-autism-and-your-childs-development-what-to-know-about-getting-a-neurodevelopmental-assessment-197528">Diagnostic assessments</a> are an important part of the <a href="https://www.autismcrc.com.au/access/national-guideline">clinical pathway</a> for children developing differently. Diagnoses can provide parents and carers with a deeper understanding of their child. A diagnosis allows the child, their family and the supporting health professionals to benefit from all the information we have about that diagnosis, to understand how best to <a href="https://www.autismcrc.com.au/access/supporting-children">support the child going forward</a>. One reason why our diagnostic systems are currently under so much strain is because of expanding diagnostic boundaries. The criteria for autism and ADHD have <a href="https://theconversation.com/from-deficits-to-a-spectrum-thinking-around-autism-has-changed-now-there-are-calls-for-a-profound-autism-diagnosis-194049">changed over time</a>, meaning more children meet criteria for these conditions than before. Another reason is that our health, disability and education systems often require a formal diagnosis for a child to receive support. This further increases demand for diagnostic assessments. Often, long waitlists result in children and families not getting timely access to crucial early therapy services. Delays can mean that many of the best opportunities to support children’s development early in life are missed, which can further <a href="https://www.sciencedirect.com/science/article/abs/pii/S1750946719301849">entrench developmental disability and disadvantage</a>. <blockquote class="twitter-tweet"> Parents turn to equine therapy for children with autism, ADHD as disability services wait times blow out <a href="https://t.co/JezFJ2TmuV">https://t.co/JezFJ2TmuV</a> via <a href="https://twitter.com/ABCaustralia?ref_src=twsrc%5Etfw">@ABCaustralia</a> — Robert Koenig-Luck (@koenig_luck) <a href="https://twitter.com/koenig_luck/status/1645227611193475077?ref_src=twsrc%5Etfw">April 10, 2023</a></blockquote> However, importantly, there are many beneficial things that families can do in the meantime to pave the way for the future. 3 things families can do While a diagnosis may help a child access support services, they are still able to access services without a diagnosis. If a parent is worried about their child’s development, then it is important they continue to seek out support services while the child is on a diagnostic waitlist. A GP is typically the best person to consult in the first instance. They can then refer the child and family to public or private therapy services. However, private service options may involve out-of-pocket expenses, which can create inequity in access to services. Parents can also take steps to: 1. Build connections with their child A key part of all early supports is nurturing the connection parents have with their child. All children benefit from having frequent, meaningful time set aside to <a href="https://clinikids.telethonkids.org.au/information-hub/blog/serve-and-return-interactions/">connect</a> with their primary caregivers. During this special connection time, parents might focus on slowing down, approaching their child with curiosity, being open to following their child’s special interests, and trying a variety of communication strategies (including words, gestures or using pictures) to <a href="https://clinikids.telethonkids.org.au/information-hub/blog/shared-attention/">support communication</a>. Parents needn’t feel pressure to spend all their time engaging with their child – but any time that can be dedicated to this will be time well spent. 2. Gather information to support diagnosis Diagnoses of <a href="https://www.cdc.gov/ncbddd/adhd/diagnosis.html">ADHD</a> and <a href="https://www.cdc.gov/ncbddd/autism/hcp-dsm.html">autism</a> are based on the observation of certain behaviours. A clinician will be able to observe some of these behaviours in their assessment, but they will also rely on information from parents about how their child usually behaves or interacts in different situations. Parents can support this process by noting examples of the patterns of behaviours they’ve observed. These might include special interests, repetitive activities, social interactions, emotional regulation, sensory preferences or how their child communicates. It is important parents don’t only note what a child finds difficult, but also their strengths and interests. Sometimes, the things a child is particularly good at can tell us just as much as their challenges. 3. Prioritise family wellbeing While parents are often proactive in seeking support for their child, they can sometimes neglect their own need for support. Parents are the most important person in a child’s life, and parental capacity and wellbeing can have a significant influence on their child’s outcomes. While waiting for a diagnosis, parents should start to plan how they are also going to get the support they need. This can include staying connected within the community and making time for activities that bring them and their family joy. Looking beyond diagnosis When parents seek out a diagnosis for their child, they want help to support their child’s development. But long waits for assessment and diagnosis can present barriers between Australia’s health, education and disability systems and the help families need. The long waiting lists to receive a diagnostic assessment are at odds with what we know about the importance of early intervention. Recent <a href="https://jamanetwork.com/journals/jamapediatrics/fullarticle/2784066">clinical trials</a> have shown how providing support to babies and parents at the first sign of developmental concern can lead to <a href="https://theconversation.com/therapy-for-babies-showing-early-signs-of-autism-reduces-the-chance-of-clinical-diagnosis-at-age-3-167146">positive developmental outcomes</a> for children. This approach prioritises acting quickly over diagnostic clarity, and makes it more likely children and families receive support during critical times in brain development. As Australia seeks to reform our early childhood development system, the need of families to receive prompt support should be front of mind. <blockquote class="twitter-tweet"> Can we have a twitter thread where we list all of the therapies and supports that parents -can- use to help their autistic kid? <a href="https://twitter.com/hashtag/OtherWaysThanABA?src=hash&ref_src=twsrc%5Etfw">#OtherWaysThanABA</a> <a href="https://twitter.com/hashtag/SayNotoABA?src=hash&ref_src=twsrc%5Etfw">#SayNotoABA</a> <a href="https://twitter.com/hashtag/AskingAutistics?src=hash&ref_src=twsrc%5Etfw">#AskingAutistics</a> <a href="https://twitter.com/hashtag/ActaullyAutistic?src=hash&ref_src=twsrc%5Etfw">#ActaullyAutistic</a> My go-to is ear defenders/earplugs/sunglasses/hoodies/fidget toys/punching bag — AutisticSciencePerson, MSc (@AutSciPerson) <a href="https://twitter.com/AutSciPerson/status/1116527158564986880?ref_src=twsrc%5Etfw">April 12, 2019</a></blockquote> Image credit: Shutterstock This article originally appeared on <a href="https://theconversation.com/autism-and-adhd-assessment-waits-are-up-to-2-years-long-what-can-families-do-in-the-meantime-203232" target="_blank" rel="noopener">The Conversation</a>.

Family & Pets

New $2 coin skyrockets in value to $1,200

A coin expert has shared how a brand new $2 coin has soared in value just 10 days after its release. The accredited numismatist from Perth, Joel Kandiah, revealed the Royal Australian Mint’s latest Vietnam Silver $2 is now in high demand. Kandiah, known online as @TheHistoryOf_Money has attracted a large following on his Instagram and TikTok with his insights on the value of multiple coin and bank notes. Speaking to 7News.com.au, he said the coveted coin is selling for up to $1,200 online with an increase of 1,400 per cent on the retail value. Kandiah shared that the shocking price hike, “has never been witnessed before on the numismatic market”. He added that the Royal Australian Mint released two brand new Vietnam War commemorative $2 coins on April 6, 2023. The first coin was an uncirculated C mintmark that has a retail value of $15 and a mintage value of 80,000. The second coin was a silver version of that same coin, but was proof finished, meaning it had been struck multiple times and hand polished for a shiny, spot-free finish. That coin had a retail value of $80, and a lowly mintage of just 5,000. Both coins were initially in demand, however the silver $2 commemorative coin has seen a remarkable price increase. Kandiah said there were two reasons behind the massive interest in the silver coin. “The first reason for the hype of this coin is that it is the first silver version of a coloured two coin and will most likely the only version that will feature the Queen’s effigy,” he told 7News.com.au. “It has a lower mintage than the most valuable coloured $2 coin, the Mars $2 coin from the 2018 Planetary Series collection, which is currently valued at around $4000. “The combination of these two factors have led to this price spiral which has never been witnessed before on the numismatic market.” Kandiah also revealed the release of the sought-after coin has stirred up a heated debate among coin collectors in Australia. “Within minutes of release, dealers across the country sold out of their allocations,” he explained. “Meanwhile, people were on the mint’s online store, call centre and in person in Canberra. Silver coins were completely sold out by the afternoon and the uncirculated coins were sold out by midnight. “People were waiting up to 16 hours in the Mint’s online queue system to try and get their hands on the coin. “Many collectors who were purchasing online had to compete with people from the ’sneaker bot’ community, who used sophisticated software to buy up new releases - only to release them on eBay at a significantly higher markup hours later. “By mid-afternoon, prices had gone up to $100 for the uncirculated coin and $300 for the silver coin. “Since then, the price of the uncirculated coin has fallen down to around $60 as ‘fear of missing out’ (FOMO) dies down. “However, the silver coin has continued to spike in price, which has now increased 15-fold to $1200 by April 16.” Kandiah said time will tell whether the silver coin will continue to increase in value. “Whether the value is due to FOMO, is too early to determine,” he told 7News.com.au. “Once we find more people get hands on their orders from the Mint, it is likely that they will sell their coins to cash in on the price rise but the relative rarity of the coin could be a long-lasting factor in its valuation. “The coin collector market has changed considerably over the last few years and it is in a new space where the buying and selling are done with each other through Facebook groups and eBay rather than through dealers. “The democratisation and accessibility of this process has led to improved liquidity within the market and hence we will continue to see rapid fluctuations in the Australian coin collector market in the future.” Image credit: Instagram

Money & Banking

ChatGPT, DALL-E 2 and the collapse of the creative process

In 2022, OpenAI – one of the world’s leading artificial intelligence research laboratories – released the text generator <a href="https://chat.openai.com/chat">ChatGPT</a> and the image generator <a href="https://openai.com/dall-e-2/">DALL-E 2</a>. While both programs represent monumental leaps in natural language processing and image generation, they’ve also been met with apprehension. Some critics have <a href="https://www.theatlantic.com/technology/archive/2022/12/chatgpt-ai-writing-college-student-essays/672371/">eulogized the college essay</a>, while others have even <a href="https://www.nytimes.com/2022/09/02/technology/ai-artificial-intelligence-artists.html">proclaimed the death of art</a>. But to what extent does this technology really interfere with creativity? After all, for the technology to generate an image or essay, a human still has to describe the task to be completed. The better that description – the more accurate, the more detailed – the better the results. After a result is generated, some further human tweaking and feedback may be needed – touching up the art, editing the text or asking the technology to create a new draft in response to revised specifications. Even the DALL-E 2 art piece that recently won first prize in the Colorado State Fair’s digital arts competition <a href="https://www.smithsonianmag.com/smart-news/artificial-intelligence-art-wins-colorado-state-fair-180980703/">required a great deal of human “help”</a> – approximately 80 hours’ worth of tweaking and refining the descriptive task needed to produce the desired result. <blockquote class="twitter-tweet"> Today's moody <a href="https://twitter.com/hashtag/AIart?src=hash&ref_src=twsrc%5Etfw">#AIart</a> style is... 🖤 deep blacks ↘️ angular light 🧼 clean lines 🌅 long shadows More in thread, full prompts in [ALT] text! <a href="https://t.co/tUV0ZfQyYb">pic.twitter.com/tUV0ZfQyYb</a> — Guy Parsons (@GuyP) <a href="https://twitter.com/GuyP/status/1612539185214234624?ref_src=twsrc%5Etfw">January 9, 2023</a></blockquote> It could be argued that by being freed from the tedious execution of our ideas – by focusing on just having ideas and describing them well to a machine – people can let the technology do the dirty work and can spend more time inventing. But in our work as philosophers at <a href="https://www.umb.edu/ethics">the Applied Ethics Center at University of Massachusetts Boston</a>, we have written about <a href="https://doi.org/10.1515/mopp-2021-0026">the effects of AI on our everyday decision-making</a>, <a href="https://www.taylorfrancis.com/chapters/edit/10.4324/9780429470325-28/owning-future-work-alec-stubbs">the future of work</a> and <a href="https://doi.org/10.1007/s43681-022-00245-6">worker attitudes toward automation</a>. Leaving aside the very real ramifications of <a href="https://www.latimes.com/opinion/story/2022-12-21/artificial-intelligence-artists-stability-ai-digital-images">robots displacing artists who are already underpaid</a>, we believe that AI art devalues the act of artistic creation for both the artist and the public. <h2>Skill and practice become superfluous</h2> In our view, the desire to close the gap between ideation and execution is a chimera: There’s no separating ideas and execution. It is the work of making something real and working through its details that carries value, not simply that moment of imagining it. Artistic works are lauded not merely for the finished product, but for the struggle, the playful interaction and the skillful engagement with the artistic task, all of which carry the artist from the moment of inception to the end result. The focus on the idea and the framing of the artistic task amounts to <a href="https://theconversation.com/what-paul-mccartneys-the-lyrics-can-teach-us-about-harnessing-our-creativity-170987">the fetishization of the creative moment</a>. Novelists write and rewrite the chapters of their manuscripts. Comedians “write on stage” in response to the laughs and groans of their audience. Musicians tweak their work in response to a discordant melody as they compose a piece. In fact, the process of execution is a gift, allowing artists to become fully immersed in a task and a practice. It allows them to enter <a href="https://www.harpercollins.com/products/flow-mihaly-csikszentmihalyi?variant=32118048686114">what some psychologists call the “flow” state</a>, where they are wholly attuned to something that they are doing, unaware of the passage of time and momentarily freed from the boredom or anxieties of everyday life. This playful state is something that would be a shame to miss out on. <a href="https://www.press.uillinois.edu/books/?id=p073182">Play tends to be understood as an autotelic activity</a> – a term derived from the Greek words auto, meaning “self,” and telos meaning “goal” or “end.” As an autotelic activity, play is done for itself – it is self-contained and requires no external validation. For the artist, the process of artistic creation is an integral part, maybe even the greatest part, of their vocation. But there is no flow state, no playfulness, without engaging in skill and practice. And the point of ChatGPT and DALL-E is to make this stage superfluous. <h2>A cheapened experience for the viewer</h2> But what about the perspective of those experiencing the art? Does it really matter how the art is produced if the finished product elicits delight? We think that it does matter, particularly because the process of creation adds to the value of art for the people experiencing it as much as it does for the artists themselves. Part of the experience of art is knowing that human effort and labor has gone into the work. Flow states and playfulness notwithstanding, art is the result of skillful and rigorous expression of human capabilities. Recall <a href="https://www.youtube.com/watch?v=rUOlnvGpcbs">the famous scene</a> from the 1997 film “<a href="https://www.imdb.com/title/tt0119177/">Gattaca</a>,” in which a pianist plays a haunting piece. At the conclusion of his performance, he throws his gloves into the admiring audience, which sees that the pianist has 12 fingers. They now understand that he was genetically engineered to play the transcendent piece they just heard – and that he could not play it with the 10 fingers of a mere mortal. Does that realization retroactively change the experience of listening? Does it take away any of the awe? <a href="https://www.theatlantic.com/magazine/archive/2004/04/the-case-against-perfection/302927/">As the philosopher Michael Sandel notes</a>: Part of what gives art and athletic achievement its power is the process of witnessing natural gifts playing out. People enjoy and celebrate this talent because, in a fundamental way, it represents the paragon of human achievement – the amalgam of talent and work, human gifts and human sweat. <h2>Is it all doom and gloom?</h2> Might ChatGPT and DALL-E be worth keeping around? Perhaps. These technologies could serve as catalysts for creativity. It’s possible that the link between ideation and execution can be sustained if these AI applications are simply viewed as mechanisms for creative imagining – <a href="https://openai.com/blog/dall-e-2-extending-creativity/">what OpenAI calls</a> “extending creativity.” They can generate stimuli that allow artists to engage in more imaginative thinking about their own process of conceiving an art piece. Put differently, if ChatGPT and DALL-E are the end results of the artistic process, something meaningful will be lost. But if they are merely tools for fomenting creative thinking, this might be less of a concern. For example, a game designer could ask DALL-E to provide some images about what a Renaissance town with a steampunk twist might look like. A writer might ask about descriptors that capture how a restrained, shy person expresses surprise. Both creators could then incorporate these suggestions into their work. But in order for what they are doing to still count as art – in order for it to feel like art to the artists and to those taking in what they have made – the artists would still have to do the bulk of the artistic work themselves. Art requires makers to keep making. <h2>The warped incentives of the internet</h2> Even if AI systems are used as catalysts for creative imaging, we believe that people should be skeptical of what these systems are drawing from. It’s important to pay close attention to the incentives that underpin and reward artistic creation, particularly online. Consider the generation of AI art. These works draw on images and video that <a href="https://www.theguardian.com/technology/2022/nov/12/when-ai-can-make-art-what-does-it-mean-for-creativity-dall-e-midjourney">already exist</a> online. But the AI is not sophisticated enough – nor is it incentivized – to consider whether works evoke a sense of wonder, sadness, anxiety and so on. They are not capable of factoring in aesthetic considerations of novelty and cross-cultural influence. Rather, training ChatGPT and DALL-E on preexisting measurements of artistic success online will tend to replicate the dominant incentives of the internet’s largest platforms: <a href="https://doi.org/10.1111/josp.12489">grabbing and retaining attention</a> for the sake of data collection and user engagement. The catalyst for creative imagining therefore can easily become subject to an addictiveness and attention-seeking imperative rather than more transcendent artistic values. It’s possible that artificial intelligence is at a precipice, one that evokes a sense of “<a href="https://www.theatlantic.com/magazine/archive/2004/04/the-case-against-perfection/302927/">moral vertigo</a>” – the uneasy dizziness people feel when scientific and technological developments outpace moral understanding. Such vertigo can lead to apathy and detachment from creative expression. If human labor is removed from the process, what value does creative expression hold? Or perhaps, having opened Pandora’s box, this is an indispensable opportunity for humanity to reassert the value of art – and to push back against a technology that may prevent many real human artists from thriving. Image credits: Getty Images This article originally appeared on <a href="https://theconversation.com/chatgpt-dall-e-2-and-the-collapse-of-the-creative-process-196461" target="_blank" rel="noopener">The Conversation</a>.

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